Genome-wide expression profiling of B lymphocytes reveals IL4R increase in allergic asthma

[EN] Allergic asthma is a multifactorial disorder in which activation and differentiation of B lymphocytes toward the production of IgE play an essential role. In these B cells, molecular mechanisms to generate IgE antibodies from IgM depend on a complex network of molecules that trigger class-switc...

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Detalles Bibliográficos
Autores: Pascual, Marien, Roa, Sergio, García Sánchez, María Asunción, Sanz Lozano, Catalina Sofía, Hernández Hernández, Laura, Greally, John M., Lorente Toledano, Félix, Dávila González, Ignacio Jesús, Isidoro García, María
Tipo de recurso: artículo
Estado:Versión publicada
Fecha de publicación:2014
País:España
Institución:Universidad de Salamanca (USAL)
Repositorio:GREDOS. Repositorio Institucional de la Universidad de Salamanca
OAI Identifier:oai:gredos.usal.es:10366/158887
Acceso en línea:https://doi.org/10.1016/j.jaci.2014.05.015
http://hdl.handle.net/10366/158887
Access Level:acceso abierto
Palabra clave:asthma
Allergic asthma
IgE
genome-wide
B lymphocytes
polymorphisms
Genetics
Allergy and Immunology
3207.01 Alergias
2409 Genética
alergia e inmunología
genética
Descripción
Sumario:[EN] Allergic asthma is a multifactorial disorder in which activation and differentiation of B lymphocytes toward the production of IgE play an essential role. In these B cells, molecular mechanisms to generate IgE antibodies from IgM depend on a complex network of molecules that trigger class-switch recombination specifically at the Sε region of the IgH locus. Several trans-membrane receptors and activating signaling cascades are known to physiologically participate in these processes, including the IL4R signaling pathway. In this regard, our group has studied in the past genetic polymorphisms in the IL4/IL4R pathway that might hold clinical correlations with atopy and asthma.