The impact of mutational clonality in predicting the response to immune checkpoint inhibitors in advanced urothelial cancer

Immune checkpoint inhibitors (ICI) have revolutionized cancer treatment and can result in complete remissions even at advanced stages of the disease. However, only a small fraction of patients respond to the treatment. To better understand which factors drive clinical benefit, we have generated whol...

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Autores: Boll, Lilian Marie, Perera Bel, Júlia, Rodriguez-Vida, Alejo, Arpí Llucià, Oriol, Rovira, Ana, Juanpere, Nuria, Vázquez Montes de Oca, Sergio, Hernández Llodrà, Silvia, Lloreta, Josep, 1958-, Albà Soler, Mar, Bellmunt Molins, Joaquim, 1959-
Tipo de recurso: artículo
Estado:Versión publicada
Fecha de publicación:2023
País:España
Institución:Universitat Pompeu Fabra
Repositorio:Repositorio Digital de la UPF
OAI Identifier:oai:repositori.upf.edu:10230/59600
Acceso en línea:http://hdl.handle.net/10230/59600
http://dx.doi.org/10.1038/s41598-023-42495-2
Access Level:acceso abierto
Palabra clave:Cancer genomics
Cancer immunotherapy
Immunotherapy
Urological cancer
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spelling The impact of mutational clonality in predicting the response to immune checkpoint inhibitors in advanced urothelial cancerBoll, Lilian MariePerera Bel, JúliaRodriguez-Vida, AlejoArpí Llucià, OriolRovira, AnaJuanpere, NuriaVázquez Montes de Oca, SergioHernández Llodrà, SilviaLloreta, Josep, 1958-Albà Soler, MarBellmunt Molins, Joaquim, 1959-Cancer genomicsCancer immunotherapyImmunotherapyUrological cancerImmune checkpoint inhibitors (ICI) have revolutionized cancer treatment and can result in complete remissions even at advanced stages of the disease. However, only a small fraction of patients respond to the treatment. To better understand which factors drive clinical benefit, we have generated whole exome and RNA sequencing data from 27 advanced urothelial carcinoma patients treated with anti-PD-(L)1 monoclonal antibodies. We assessed the influence on the response of non-synonymous mutations (tumor mutational burden or TMB), clonal and subclonal mutations, neoantigen load and various gene expression markers. We found that although TMB is significantly associated with response, this effect can be mostly explained by clonal mutations, present in all cancer cells. This trend was validated in an additional cohort. Additionally, we found that responders with few clonal mutations had abnormally high levels of T and B cell immune markers, suggesting that a high immune cell infiltration signature could be a better predictive biomarker for this subset of patients. Our results support the idea that highly clonal cancers are more likely to respond to ICI and suggest that non-additive effects of different signatures should be considered for predictive models.Nature Research202420242023info:eu-repo/semantics/articleinfo:eu-repo/semantics/publishedVersionapplication/pdfapplication/pdfhttp://hdl.handle.net/10230/59600http://dx.doi.org/10.1038/s41598-023-42495-2reponame:Repositorio Digital de la UPFinstname:Universitat Pompeu FabraInglésSci Rep. 2023 Sep 15;13(1):15287© The Author(s) 2023. Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.http://creativecommons.org/licenses/by/4.0/info:eu-repo/semantics/openAccessoai:repositori.upf.edu:10230/596002026-06-12T07:21:37Z
dc.title.none.fl_str_mv The impact of mutational clonality in predicting the response to immune checkpoint inhibitors in advanced urothelial cancer
title The impact of mutational clonality in predicting the response to immune checkpoint inhibitors in advanced urothelial cancer
spellingShingle The impact of mutational clonality in predicting the response to immune checkpoint inhibitors in advanced urothelial cancer
Boll, Lilian Marie
Cancer genomics
Cancer immunotherapy
Immunotherapy
Urological cancer
title_short The impact of mutational clonality in predicting the response to immune checkpoint inhibitors in advanced urothelial cancer
title_full The impact of mutational clonality in predicting the response to immune checkpoint inhibitors in advanced urothelial cancer
title_fullStr The impact of mutational clonality in predicting the response to immune checkpoint inhibitors in advanced urothelial cancer
title_full_unstemmed The impact of mutational clonality in predicting the response to immune checkpoint inhibitors in advanced urothelial cancer
title_sort The impact of mutational clonality in predicting the response to immune checkpoint inhibitors in advanced urothelial cancer
dc.creator.none.fl_str_mv Boll, Lilian Marie
Perera Bel, Júlia
Rodriguez-Vida, Alejo
Arpí Llucià, Oriol
Rovira, Ana
Juanpere, Nuria
Vázquez Montes de Oca, Sergio
Hernández Llodrà, Silvia
Lloreta, Josep, 1958-
Albà Soler, Mar
Bellmunt Molins, Joaquim, 1959-
author Boll, Lilian Marie
author_facet Boll, Lilian Marie
Perera Bel, Júlia
Rodriguez-Vida, Alejo
Arpí Llucià, Oriol
Rovira, Ana
Juanpere, Nuria
Vázquez Montes de Oca, Sergio
Hernández Llodrà, Silvia
Lloreta, Josep, 1958-
Albà Soler, Mar
Bellmunt Molins, Joaquim, 1959-
author_role author
author2 Perera Bel, Júlia
Rodriguez-Vida, Alejo
Arpí Llucià, Oriol
Rovira, Ana
Juanpere, Nuria
Vázquez Montes de Oca, Sergio
Hernández Llodrà, Silvia
Lloreta, Josep, 1958-
Albà Soler, Mar
Bellmunt Molins, Joaquim, 1959-
author2_role author
author
author
author
author
author
author
author
author
author
dc.subject.none.fl_str_mv Cancer genomics
Cancer immunotherapy
Immunotherapy
Urological cancer
topic Cancer genomics
Cancer immunotherapy
Immunotherapy
Urological cancer
description Immune checkpoint inhibitors (ICI) have revolutionized cancer treatment and can result in complete remissions even at advanced stages of the disease. However, only a small fraction of patients respond to the treatment. To better understand which factors drive clinical benefit, we have generated whole exome and RNA sequencing data from 27 advanced urothelial carcinoma patients treated with anti-PD-(L)1 monoclonal antibodies. We assessed the influence on the response of non-synonymous mutations (tumor mutational burden or TMB), clonal and subclonal mutations, neoantigen load and various gene expression markers. We found that although TMB is significantly associated with response, this effect can be mostly explained by clonal mutations, present in all cancer cells. This trend was validated in an additional cohort. Additionally, we found that responders with few clonal mutations had abnormally high levels of T and B cell immune markers, suggesting that a high immune cell infiltration signature could be a better predictive biomarker for this subset of patients. Our results support the idea that highly clonal cancers are more likely to respond to ICI and suggest that non-additive effects of different signatures should be considered for predictive models.
publishDate 2023
dc.date.none.fl_str_mv 2023
2024
2024
dc.type.none.fl_str_mv info:eu-repo/semantics/article
info:eu-repo/semantics/publishedVersion
format article
status_str publishedVersion
dc.identifier.none.fl_str_mv http://hdl.handle.net/10230/59600
http://dx.doi.org/10.1038/s41598-023-42495-2
url http://hdl.handle.net/10230/59600
http://dx.doi.org/10.1038/s41598-023-42495-2
dc.language.none.fl_str_mv Inglés
language_invalid_str_mv Inglés
dc.relation.none.fl_str_mv Sci Rep. 2023 Sep 15;13(1):15287
dc.rights.none.fl_str_mv http://creativecommons.org/licenses/by/4.0/
info:eu-repo/semantics/openAccess
rights_invalid_str_mv http://creativecommons.org/licenses/by/4.0/
eu_rights_str_mv openAccess
dc.format.none.fl_str_mv application/pdf
application/pdf
dc.publisher.none.fl_str_mv Nature Research
publisher.none.fl_str_mv Nature Research
dc.source.none.fl_str_mv reponame:Repositorio Digital de la UPF
instname:Universitat Pompeu Fabra
instname_str Universitat Pompeu Fabra
reponame_str Repositorio Digital de la UPF
collection Repositorio Digital de la UPF
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repository.mail.fl_str_mv
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