Vanadate increases L-type pyruvate kinase mRNAs level in adult rat hepatocytes in primary culture

In primary culture of adult rat hepatocytes, vanadate in the presence of glucose stimulates the expression of the liver (L-type) pyruvate kinase gene. Glucose by itself was inactive, and vanadate, like insulin, was also inefficient in the absence of glucose. Similar results were obtained on glucokin...

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Detalhes bibliográficos
Autores: Miralpeix, Montserrat, Decaux, Jean-François, Kahn, Axel, Bartrons Bach, Ramon
Formato: artículo
Estado:Versión publicada
Fecha de publicación:1991
País:España
Recursos:Universidad de Barcelona
Repositorio:Dipòsit Digital de la UB
OAI Identifier:oai:diposit.ub.edu:2445/134640
Acesso em linha:https://hdl.handle.net/2445/134640
Access Level:acceso abierto
Palavra-chave:Micro RNAs
Isoenzims
Genètica
Fetge
Enzimologia
Biosíntesi
Farmacologia
MicroRNAs
Isoenzymes
Genetics
Liver
Enzymology
Biosynthesis
Pharmacology
Descrição
Resumo:In primary culture of adult rat hepatocytes, vanadate in the presence of glucose stimulates the expression of the liver (L-type) pyruvate kinase gene. Glucose by itself was inactive, and vanadate, like insulin, was also inefficient in the absence of glucose. Similar results were obtained on glucokinase gene expression. An analogue of cAMP, 8-(4-chlorophenylthio)-cAMP, inhibited the production of L-type pyruvate kinase and glucokinase mRNAs in the presence of glucose plus vanadate.