CagL polymorphisms D58/K59 are predominant in Helicobacter pylori strains isolated from Mexican patients with chronic gastritis.

Helicobacter pylori is a Gram¿negative bacterium that colonizes the gastric mucosa in humans. One of the main virulence factors of H. pylori is the cag pathogenicity island (cagPAI), which encodes a type 4¿secretion system (T4SS) and the cytotoxin CagA. Translocation of CagA through the T4SS trigger...

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Detalhes bibliográficos
Autores: Roman Roman, Adolfo, Martinez Santos, Veronica Iranzu, Castañon Sanchez, Carlos Alberto, Albañil Muñoz, Alan J, González Mendoza, Paola, Soto¿Flores, Diana G, Martinez Carrillo, Dinorah Nashely, Fernandez Tilapa, Gloria
Formato: artículo
Estado:Versión publicada
Fecha de publicación:2019
País:México
Recursos:Universidad Autónoma de Guerrero
Repositorio:Repositorio Institucional de Ciencia Abierta de la Universidad Autónoma de Guerrero
Idioma:español
OAI Identifier:oai:ri.uagro.mx:uagro/1214
Acesso em linha:http://ri.uagro.mx/handle/uagro/1214
Access Level:acceso abierto
Palavra-chave:Chronic gastritis
polymorphisms
Polymorphic
MEDICINA Y CIENCIAS DE LA SALUD::CIENCIAS MÉDICAS::OTRAS ESPECIALIDADES MÉDICAS
Descrição
Resumo:Helicobacter pylori is a Gram¿negative bacterium that colonizes the gastric mucosa in humans. One of the main virulence factors of H. pylori is the cag pathogenicity island (cagPAI), which encodes a type 4¿secretion system (T4SS) and the cytotoxin CagA. Translocation of CagA through the T4SS triggers host¿signaling pathways. One of the T4SS proteins is CagL, which is necessary for CagA translocation. CagL is a 26¿kDa protein that contains a hypervariable motif, which spans residues 58 to 62. Several polymorphisms in this region have been associated with different disease outcomes, e.g. in Mexico, N58 is associated with a higher risk of gastric cancer. The aim of this work is to analyze the sequence of the hypervariable motif (residues 58 to 62) of clinical isolates from Mexican patients with chronic gastritis, and to correlate these polymorphisms with the vacA genotype.