Metabolismo energético, estado redox y parámetros de las células MCF10A inducidas a senescencia por estrés oxidante

Cellular senescence (SC) is a biological phenomenon that occurs in response to damage and has been linked to different biological and pathological processes. Much of the knowledge of cellular senescence has been described in fibroblasts; however, this is not the only cell type in which senescence oc...

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Detalles Bibliográficos
Autor: ANGELICA ALEJANDRA AQUINO CRUZ
Tipo de recurso: tesis de maestría
Estado:Versión publicada
Fecha de publicación:2018
País:México
Institución:Universidad Autónoma Metropolitana
Repositorio:Repositorio Institucional de la UAM Iztapalapa
Idioma:español
OAI Identifier:oai:bindani.izt.uam.mx:k0698751r
Acceso en línea:https://doi.org/10.24275/uami.k0698751r
Access Level:acceso abierto
Palabra clave:info:eu-repo/classification/LEM/Cells -- Aging
info:eu-repo/classification/LEM/Oxidation-reduction reaction
info:eu-repo/classification/LEM/Oxidative stress
info:eu-repo/classification/LEM/Estrés oxidativo
info:eu-repo/classification/LEM/Reacción de oxidación-reducción
info:eu-repo/classification/LEM/Células -- Envejecimiento
info:eu-repo/classification/cti/2
Descripción
Sumario:Cellular senescence (SC) is a biological phenomenon that occurs in response to damage and has been linked to different biological and pathological processes. Much of the knowledge of cellular senescence has been described in fibroblasts; however, this is not the only cell type in which senescence occurs. The establishment of senescence can occur due to oxidative stress that induces DNA damage. Aspects such as energy metabolism and the redox state of senescent cells are little studied. In this work, senescence was induced in the MCF10A cell line by oxidative stress. The energy metabolism of senescent cells was determined by measuring the rate of oxygen consumption (OCR) and the acidification rate of the extracellular medium (ECAR), as well as, the redox state evaluating the presence of reactive oxygen species (ROS) and the oxidized and reduced glutathione coefficient (GSH/GSSG).Our results determined that senescent MCF10A cells induced to senescence by oxidative stress exhibit an energy metabolism preferentially directed towards glycolysis, and a redox state prooxidant, due to an elevation in GSSG content that coincides with an increase in ROS.