Investigation of Autosomal Genetic Sex Differences in Parkinson's Disease

Objective: Parkinson's disease (PD) is a complex neurodegenerative disorder. Men are on average similar to 1.5 times more likely to develop PD compared to women with European ancestry. Over the years, genomewide association studies (GWAS) have identified numerous genetic risk factors for PD, ho...

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Autores: Blauwendraat, C, Iwaki, H, Makarious, MB, Bandres-Ciga, S, Leonard, HL, Grenn, FP, Lake, J, Krohn, L, Tan, M, Kim, JJ, Gibbs, JR, Hernandez, DG, Ruskey, JA, Pihlstrom, L, Toft, M, van Hilten, JJ, Marinus, J, Schulte, C, Brockmann, K, Sharma, M, Siitonen, A, Majamaa, K, Eerola-Rautio, J, Tienari, PJ, Grosset, DG, Lesage, S, Corvol, JC, Brice, A, Wood, N, Hardy, J, Gan-Or, Z, Heutink, P, Gasser, T, Morris, HR, Noyce, AJ, Nalls, MA, Singleton, AB, Clarimón J., Dols-Icardo, O, Kulisevsky J., Pagonabarraga, J, Int Parkinsons Dis Genomics Consor
Tipo de recurso: artículo
Estado:Versión publicada
Fecha de publicación:2021
País:España
Institución:Institut d’Investigació Biomèdica Sant Pau (IIB Sant Pau)
Repositorio:r-IIB SANT PAU. Repositorio Institucional de Producción Científica del Instituto de Investigación Biomédica Sant Pau
OAI Identifier:oai:iibsantpau.fundanetsuite.com:p7994
Acceso en línea:https://iibsantpau.fundanetsuite.com/Publicaciones/ProdCientif/PublicacionFrw.aspx?id=7994
http://hdl.handle.net/10138/332389
Access Level:acceso abierto
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spelling Investigation of Autosomal Genetic Sex Differences in Parkinson's DiseaseBlauwendraat, CIwaki, HMakarious, MBBandres-Ciga, SLeonard, HLGrenn, FPLake, JKrohn, LTan, MKim, JJGibbs, JRHernandez, DGRuskey, JAPihlstrom, LToft, Mvan Hilten, JJMarinus, JSchulte, CBrockmann, KSharma, MSiitonen, AMajamaa, KEerola-Rautio, JTienari, PJGrosset, DGLesage, SCorvol, JCBrice, AWood, NHardy, JGan-Or, ZHeutink, PGasser, TMorris, HRNoyce, AJNalls, MASingleton, ABClarimón J.Dols-Icardo, OKulisevsky J.Pagonabarraga, JInt Parkinsons Dis Genomics ConsorObjective: Parkinson's disease (PD) is a complex neurodegenerative disorder. Men are on average similar to 1.5 times more likely to develop PD compared to women with European ancestry. Over the years, genomewide association studies (GWAS) have identified numerous genetic risk factors for PD, however, it is unclear whether genetics contribute to disease etiology in a sex-specific manner. Methods: In an effort to study sex-specific genetic factors associated with PD, we explored 2 large genetic datasets from the International Parkinson's Disease Genomics Consortium and the UK Biobank consisting of 13,020 male PD cases, 7,936 paternal proxy cases, 89,660 male controls, 7,947 female PD cases, 5,473 maternal proxy cases, and 90,662 female controls. We performed GWAS meta-analyses to identify distinct patterns of genetic risk contributing to disease in male versus female PD cases. Results: In total, 19 genomewide significant regions were identified and no sex-specific effects were observed. A high genetic correlation between the male and female PD GWAS were identified (rg = 0.877) and heritability estimates were identical between male and female PD cases (similar to 20%). Interpretation: We did not detect any significant genetic differences between male or female PD cases. Our study does not support the notion that common genetic variation on the autosomes could explain the difference in prevalence of PD between males and females cases at least when considering the current sample size under study. Further studies are warranted to investigate the genetic architecture of PD explained by X and Y chromosomes and further evaluate environmental effects that could potentially contribute to PD etiology in male versus female patients.WILEY2021info:eu-repo/semantics/articleinfo:eu-repo/semantics/publishedVersionhttps://iibsantpau.fundanetsuite.com/Publicaciones/ProdCientif/PublicacionFrw.aspx?id=7994http://hdl.handle.net/10138/332389ANNALS OF NEUROLOGYISSN: 03645134ISSNe: 15318249reponame:r-IIB SANT PAU. Repositorio Institucional de Producción Científica del Instituto de Investigación Biomédica Sant Pauinstname:Institut d’Investigació Biomèdica Sant Pau (IIB Sant Pau)Inglésinfo:eu-repo/semantics/openAccessoai:iibsantpau.fundanetsuite.com:p79942026-06-14T12:41:47Z
dc.title.none.fl_str_mv Investigation of Autosomal Genetic Sex Differences in Parkinson's Disease
title Investigation of Autosomal Genetic Sex Differences in Parkinson's Disease
spellingShingle Investigation of Autosomal Genetic Sex Differences in Parkinson's Disease
Blauwendraat, C
title_short Investigation of Autosomal Genetic Sex Differences in Parkinson's Disease
title_full Investigation of Autosomal Genetic Sex Differences in Parkinson's Disease
title_fullStr Investigation of Autosomal Genetic Sex Differences in Parkinson's Disease
title_full_unstemmed Investigation of Autosomal Genetic Sex Differences in Parkinson's Disease
title_sort Investigation of Autosomal Genetic Sex Differences in Parkinson's Disease
dc.creator.none.fl_str_mv Blauwendraat, C
Iwaki, H
Makarious, MB
Bandres-Ciga, S
Leonard, HL
Grenn, FP
Lake, J
Krohn, L
Tan, M
Kim, JJ
Gibbs, JR
Hernandez, DG
Ruskey, JA
Pihlstrom, L
Toft, M
van Hilten, JJ
Marinus, J
Schulte, C
Brockmann, K
Sharma, M
Siitonen, A
Majamaa, K
Eerola-Rautio, J
Tienari, PJ
Grosset, DG
Lesage, S
Corvol, JC
Brice, A
Wood, N
Hardy, J
Gan-Or, Z
Heutink, P
Gasser, T
Morris, HR
Noyce, AJ
Nalls, MA
Singleton, AB
Clarimón J.
Dols-Icardo, O
Kulisevsky J.
Pagonabarraga, J
Int Parkinsons Dis Genomics Consor
author Blauwendraat, C
author_facet Blauwendraat, C
Iwaki, H
Makarious, MB
Bandres-Ciga, S
Leonard, HL
Grenn, FP
Lake, J
Krohn, L
Tan, M
Kim, JJ
Gibbs, JR
Hernandez, DG
Ruskey, JA
Pihlstrom, L
Toft, M
van Hilten, JJ
Marinus, J
Schulte, C
Brockmann, K
Sharma, M
Siitonen, A
Majamaa, K
Eerola-Rautio, J
Tienari, PJ
Grosset, DG
Lesage, S
Corvol, JC
Brice, A
Wood, N
Hardy, J
Gan-Or, Z
Heutink, P
Gasser, T
Morris, HR
Noyce, AJ
Nalls, MA
Singleton, AB
Clarimón J.
Dols-Icardo, O
Kulisevsky J.
Pagonabarraga, J
Int Parkinsons Dis Genomics Consor
author_role author
author2 Iwaki, H
Makarious, MB
Bandres-Ciga, S
Leonard, HL
Grenn, FP
Lake, J
Krohn, L
Tan, M
Kim, JJ
Gibbs, JR
Hernandez, DG
Ruskey, JA
Pihlstrom, L
Toft, M
van Hilten, JJ
Marinus, J
Schulte, C
Brockmann, K
Sharma, M
Siitonen, A
Majamaa, K
Eerola-Rautio, J
Tienari, PJ
Grosset, DG
Lesage, S
Corvol, JC
Brice, A
Wood, N
Hardy, J
Gan-Or, Z
Heutink, P
Gasser, T
Morris, HR
Noyce, AJ
Nalls, MA
Singleton, AB
Clarimón J.
Dols-Icardo, O
Kulisevsky J.
Pagonabarraga, J
Int Parkinsons Dis Genomics Consor
author2_role author
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author
author
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author
author
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author
author
author
author
author
author
author
author
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description Objective: Parkinson's disease (PD) is a complex neurodegenerative disorder. Men are on average similar to 1.5 times more likely to develop PD compared to women with European ancestry. Over the years, genomewide association studies (GWAS) have identified numerous genetic risk factors for PD, however, it is unclear whether genetics contribute to disease etiology in a sex-specific manner. Methods: In an effort to study sex-specific genetic factors associated with PD, we explored 2 large genetic datasets from the International Parkinson's Disease Genomics Consortium and the UK Biobank consisting of 13,020 male PD cases, 7,936 paternal proxy cases, 89,660 male controls, 7,947 female PD cases, 5,473 maternal proxy cases, and 90,662 female controls. We performed GWAS meta-analyses to identify distinct patterns of genetic risk contributing to disease in male versus female PD cases. Results: In total, 19 genomewide significant regions were identified and no sex-specific effects were observed. A high genetic correlation between the male and female PD GWAS were identified (rg = 0.877) and heritability estimates were identical between male and female PD cases (similar to 20%). Interpretation: We did not detect any significant genetic differences between male or female PD cases. Our study does not support the notion that common genetic variation on the autosomes could explain the difference in prevalence of PD between males and females cases at least when considering the current sample size under study. Further studies are warranted to investigate the genetic architecture of PD explained by X and Y chromosomes and further evaluate environmental effects that could potentially contribute to PD etiology in male versus female patients.
publishDate 2021
dc.date.none.fl_str_mv 2021
dc.type.none.fl_str_mv info:eu-repo/semantics/article
info:eu-repo/semantics/publishedVersion
format article
status_str publishedVersion
dc.identifier.none.fl_str_mv https://iibsantpau.fundanetsuite.com/Publicaciones/ProdCientif/PublicacionFrw.aspx?id=7994
http://hdl.handle.net/10138/332389
url https://iibsantpau.fundanetsuite.com/Publicaciones/ProdCientif/PublicacionFrw.aspx?id=7994
http://hdl.handle.net/10138/332389
dc.language.none.fl_str_mv Inglés
language_invalid_str_mv Inglés
dc.rights.none.fl_str_mv info:eu-repo/semantics/openAccess
eu_rights_str_mv openAccess
dc.publisher.none.fl_str_mv WILEY
publisher.none.fl_str_mv WILEY
dc.source.none.fl_str_mv ANNALS OF NEUROLOGY
ISSN: 03645134
ISSNe: 15318249
reponame:r-IIB SANT PAU. Repositorio Institucional de Producción Científica del Instituto de Investigación Biomédica Sant Pau
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