State of the art of the molecular hyperselection to guide treatment with anti-EGFR antibodies in RAS WT mCRC: implications for clinical practice and future perspectives

[Introduction] Adding monoclonal antibodies to chemotherapy drastically changed the landscape of advanced colorectal cancer. The prediction of benefit from anti-EGFR therapies is mainly based on the absence of mutations in RAS and BRAF genes, the primary tumor sidedness and microsatellite MSS/MSI st...

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Detalles Bibliográficos
Autores: García-Alfonso, Pilar, Valladares-Ayerbes, Manuel, Muñoz Martín, Andrés J., Morales Herrero, Rocío, Galvez Muñoz, Elisa, Prat-Llorens, Gerard
Tipo de recurso: artículo
Estado:Versión publicada
Fecha de publicación:2025
País:España
Institución:Consejo Superior de Investigaciones Científicas (CSIC)
Repositorio:DIGITAL.CSIC. Repositorio Institucional del CSIC
OAI Identifier:oai:digital.csic.es:10261/391487
Acceso en línea:http://hdl.handle.net/10261/391487
https://api.elsevier.com/content/abstract/scopus_id/105000126996
Access Level:acceso abierto
Palabra clave:Colorectal cancer
Anti-EGFR therapies
CtDNA
Molecular profile
Negative hyperselection
Descripción
Sumario:[Introduction] Adding monoclonal antibodies to chemotherapy drastically changed the landscape of advanced colorectal cancer. The prediction of benefit from anti-EGFR therapies is mainly based on the absence of mutations in RAS and BRAF genes, the primary tumor sidedness and microsatellite MSS/MSI status. Molecular hyperselection may optimize the outcome of patients receiving anti-EGFR while detecting additional resistance alterations, both in chemo-naïve and in chemo-refractory settings.