Reactive oxygen species generators affect quality parameters and apoptosis markers differently in red deer spermatozoa.

Fe2+/ascorbate, hydrogen peroxide (H2O2) and hypoxanthine/xanthine oxidase (XOD) are commonly used for inducing oxidative stress on spermatozoa. A comparative study of these agents was carried out on thawed spermatozoa from red deer. First, we tested a high, medium and low concentration of each agen...

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Detalles Bibliográficos
Autores: Martínez Pastor, Felipe, Aisen, Eduardo, Fernández Santos, María del Rocío, Esteso, Milagros C., Maroto Morales, Alejandro, García Álvarez, Olga, Garde López-Brea, José Julián
Tipo de recurso: artículo
Fecha de publicación:2009
País:España
Institución:Universidad de Castilla-La Mancha
Repositorio:RUIdeRA. Repositorio Institucional de la UCLM
OAI Identifier:oai:ruidera.uclm.es:10578/7462
Acceso en línea:http://hdl.handle.net/10578/7462
Access Level:acceso abierto
Palabra clave:Apoptosis
Espermatozoides
Mitocondrias
Toxicología
Ciervos
Descripción
Sumario:Fe2+/ascorbate, hydrogen peroxide (H2O2) and hypoxanthine/xanthine oxidase (XOD) are commonly used for inducing oxidative stress on spermatozoa. A comparative study of these agents was carried out on thawed spermatozoa from red deer. First, we tested a high, medium and low concentration of each agent: 100, 10 and 1 μM Fe2+ (hydroxil radical generator); 1 mM, 100 and 10 μM H2O2; and 100, 10 and 1 mU/mL XOD (superoxide and H2O2 generator), incubating at 37 C for 180 min. Intracellular ROS (H2DCFDA) increased with dose and time, and similarly for the three systems at each concentration level. Motility and mitocondrial membrane potential ( m) were considerably decreased by H2O2 (1 mM and 100 μM) and XOD (100 and 10 mU/mL). Only H2O2 1 mM reduced viability. The antioxidant Trolox (10 μM) reduced intracellular ROS, but could not prevent H2O2 or XOD effects. In a second experiment, we used YO-PRO-1 and M540 as apoptotic and membrane-stability markers, respectively. Only H2O2 increased the proportion of apoptotic and membrane-destabilized spermatozoa. Catalase added to XOD prevented m loss, confirming that H2O2 was the causative agent, not superoxide. In a third experiment, caspase activation was tested using the FLICA probe. Only H2O2 increased the proportion of viable spermatozoa with activated caspases. There were important differences between ROS generators, being H2O2 the most cytotoxic. Although H2O2 and XOD caused m dissipation, it did not reflect on increasing apoptotic markers. We hypothesize that a subpopulation of spermatozoa may lack apoptotic markers.