Genome-wide transcriptional silencing and mRNA stabilization allow the coordinated expression of the meiotic program in mice

[EN]The transcriptional dynamic of mammalian cells when these transit from the ubiquitous mitotic to a meiotic-specific program is key to understand this switch central to sexual reproduction. By quantifying active RNA polymerase II and nascent transcripts using single cell dataset and ethynyl-uridi...

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Detalles Bibliográficos
Autores: Bellutti, Laura, Chan Sock Peng, Edith, Cluzet, Victoria, Guerquin, Marie-Justine, Rolland, Antoine, Messiaen, Sébastien, Llano Cuadra, María Elena, Dereli, Ihsan, Martini, Emmanuelle, Tóth, Attila, Martín Pendás, José Alberto, Chalmel, Frederic, Livera, Gabriel
Tipo de recurso: artículo
Estado:Versión publicada
Fecha de publicación:2025
País:España
Institución:Universidad de Salamanca (USAL)
Repositorio:GREDOS. Repositorio Institucional de la Universidad de Salamanca
OAI Identifier:oai:gredos.usal.es:10366/169242
Acceso en línea:http://hdl.handle.net/10366/169242
Access Level:acceso abierto
Palabra clave:Meiotic entry
spermatogenesis
transcription
Spermatogenesis
Meiosis
RNA
RNA Polymerase II
Spermatogonia
Gene Silencing
Testis
Animals
Histones
Genome
RNA Stability
Mice
2407 Biología Celular
silenciamiento génico
meiosis
estabilidad del ARN
ARN polimerasa II
histonas
animales
espermatogénesis
ratones
ARN
testículo
espermatogonias
genoma
Descripción
Sumario:[EN]The transcriptional dynamic of mammalian cells when these transit from the ubiquitous mitotic to a meiotic-specific program is key to understand this switch central to sexual reproduction. By quantifying active RNA polymerase II and nascent transcripts using single cell dataset and ethynyl-uridine pool-down with sorted cells from synchronized testes, we detailed the transcriptional activity of murine male germ cells. When spermatogonia differentiate, transcription slows down, reaching minimal activity at meiotic entry and resumes during pachytene stage. This event, we termed EMLT (for early meiotic low transcription), is distinct from the silencing of sex chromosomes as it is independent of Setdb1, though it is accompanied by the same chromatin mark, H3K9me3. EMLT is delayed in Stra8KO but occurs in mutants altering meiotic chromosome structure or double-strand break formation or repair. By comparing transcript abundance and nascent transcription we unveil a massive event of messenger RNA stabilization that parallels EMLT. Altogether our data indicate that meiosis is initiated with a nearly silent genome, and we propose that the stabilization of transcripts at that time facilitates the meiotic entry by synchronizing the expression of several meiotic subprograms.