Long noncoding RNAs with enhancer-like function in human cells

While the long noncoding RNAs (ncRNAs) constitute a large portion of the mammalian transcriptome, their biological functions has remained elusive. A few long ncRNAs that have been studied in any detail silence gene expression in processes such as X-inactivation and imprinting. We used a GENCODE anno...

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Detalles Bibliográficos
Autores: Orom, Ulf Andersson, Derrien, Thomas, Beringer, Malte, Gumireddy, Kiranmai, Gardini, Alessandro, Bussotti, Giovanni, 1983-, Lai, Fan, Zytnicki, Matthias, Notredame, Cedric, Huang, Qihong, Guigó Serra, Roderic, Shiekhattar, Ramin
Tipo de recurso: artículo
Estado:Versión publicada
Fecha de publicación:2010
País:España
Institución:Varias* (Consorci de Biblioteques Universitáries de Catalunya, Centre de Serveis Científics i Acadèmics de Catalunya)
Repositorio:Recercat. Dipósit de la Recerca de Catalunya
OAI Identifier:oai:recercat.cat:10230/25539
Acceso en línea:http://hdl.handle.net/10230/25539
http://dx.doi.org/10.1016/j.cell.2010.09.001
Access Level:acceso abierto
Palabra clave:RNA missatger
Factors de transcripció
Descripción
Sumario:While the long noncoding RNAs (ncRNAs) constitute a large portion of the mammalian transcriptome, their biological functions has remained elusive. A few long ncRNAs that have been studied in any detail silence gene expression in processes such as X-inactivation and imprinting. We used a GENCODE annotation of the human genome to characterize over a thousand long ncRNAs that are expressed in multiple cell lines. Unexpectedly, we found an enhancer-like function for a set of these long ncRNAs in human cell lines. Depletion of a number of ncRNAs led to decreased expression of their neighboring protein-coding genes, including the master regulator of hematopoiesis, SCL (also called TAL1), Snai1 and Snai2. Using heterologous transcription assays we demonstrated a requirement for the ncRNAs in activation of gene expression. These results reveal an unanticipated role for a class of long ncRNAs in activation of critical regulators of development and differentiation.