A phase I, first-in-human clinical trial of the GDF-15 neutralizing antibody CTL-002 in subjects with advanced stage solid tumors (Acronym: GDFATHER)

Background Growth and differentiation factor 15 (GDF-15) is a TGF-β superfamily member physiologically expressed mainly in placenta and linked to feto-maternal tolerance. Under pathophysiologic conditions, prevention of excessive immune cell infiltration during tissue damage and cachexia induction h...

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Autores: Melero, I. (Ignacio)|||/items/82113ea8-7ce1-49d5-9ee3-42cf20db1c4e, Calvo, E. (Emiliano)|||/items/52242792-ef7e-42a3-80b0-88885264e12c, Goebeler, M.E. (Maria Elisabeth)|||/items/6c63f93c-674c-454f-846c-130cf720626e, Garralda, E. (Elena)|||/items/86a6b282-a0e6-4c91-9ad6-2689352ec877, Dummer, R. (Reinhard)|||/items/79077894-aa41-4ade-9c45-7e122d95a314, Ruiz-Rodríguez, M.J. (María J.)|||/items/6ff22309-2863-4f8b-936b-699f14ccd00f, Miguel, M.J. (María Jesús) de|||/items/4968c547-5897-4fb3-86c1-d860c9ba4861, Sayehli, C.M. (Cyrus Michael)|||/items/e96b3ee4-0ae7-44cd-a313-4effc23a1535, Alonso-Casal, G. (Guzman)|||/items/ec11ebc1-8035-48b8-8026-b0817da655f3, Ramelyte, E. (Egle)|||/items/69b839d1-edf8-437b-8358-3a4deaffa947, Schuler, M. (Martin)|||/items/3d702da5-b545-47d4-b1ba-4d0635d9a93a, Gromke, T. (Tanja)|||/items/14163abf-3f51-466b-9bfd-0f2e387b79c8, Sanmamed, M. (Miguel)|||/items/8e36070e-f502-4e21-8464-ca36ecc259fd, Moreno, I. (Irene)|||/items/f63e4b5a-e0ad-41fa-94a2-6573a6a81765, Bargou, R. (Ralf)|||/items/7e1d8c50-3069-49dc-aa21-dbf95256429f, Lostes, M. (Maria)|||/items/98a22e01-d5da-49dd-8632-9e058aff1838, Maul, J.T. (Julia Tatjana)|||/items/82d62090-3ece-4ff0-9e64-ed1a0bbc8c77, Eggenschwiler, C. (Corinne)|||/items/aceb4ddb-edd2-4e75-bf1d-ee6d510b838e, Richly, H. (Heike)|||/items/faa55b31-490a-4d95-9268-d78f97d4e54e, Fettes, P. (Petra)|||/items/5b4fe8fb-ac46-43eb-86e4-f286a9827f88, Klar, K. (Kathrin)|||/items/ede51ddd-226f-4265-a8bb-c3bb8f393429, Schuberth-Wagner, C. (Christine)|||/items/bdd07732-979b-4c3a-8a0e-84e19c4a7deb, Haake, M. (Markus)|||/items/3439beb0-f1ec-4239-bb1b-0e374dc2bf94, Wischhusen, J. (Jörg)|||/items/d20d722d-90a8-44a2-b5e9-00367027c812, Leo, E. (Eugen)|||/items/db3a1386-f2ba-4f3f-b710-c211a9856eb4
Tipo de recurso: artículo
Fecha de publicación:2021
País:España
Institución:Universidad de Navarra
Repositorio:Dadun. Depósito Académico Digital de la Universidad de Navarra
Idioma:inglés
OAI Identifier:oai:dadun.unav.edu:10171/116010
Acceso en línea:https://hdl.handle.net/10171/116010
Access Level:acceso abierto
Palabra clave:GDF-15
CTL-002
Tumor
Descripción
Sumario:Background Growth and differentiation factor 15 (GDF-15) is a TGF-β superfamily member physiologically expressed mainly in placenta and linked to feto-maternal tolerance. Under pathophysiologic conditions, prevention of excessive immune cell infiltration during tissue damage and cachexia induction have been ascribed to GDF-15. Recent research has though indicated a prominant role in modulation of the tumor microenvironment and the immune synapse, too1 2 indicating that GDF-15 may be a major tumor-derived immunosuppressant. Importantly, several cancer entities secrete high levels of GDF-15, correlating with poor prognosis and reduced overall survival [Front Immunol 2020 May 19;11:951]. To block this effect the GDF-15 neutralizing antibody CTL-002 was generated. In preclinical models CTL-002 demonstrated potent effector T cell shifting into tumor tissue by neutralizing GDF-15. Methods This is a phase 1, first-in-human (FIH), two-part, open-label clinical trial of intravenous (IV) administration of CTL-002 given as monotherapy and in combination with an anti-PD-1 antibody in subjects with advanced-stage, relapsed/refractory solid tumors who relapsed or were refractory to a prior anti-PD-1/PD-L1 therapy. Eligible subjects have exhausted all available approved standard treatments, including prior anti-PD1/-PD-L1 treatment, and present with a biopsy-accessible tumor for serial biopsy taking. The trial is termed GDFATHER, for ”GDF-15 Antibody-mediaTed Effector cell Relocation”.Main endpoints are safety of CTL-002 monotherapy and CTL-002 combination with an anti-PD-1 antibody, pharmacokinetics, pharmacodynamics (e.g. degree of GDF-15 neutralization achieved and change in immune-cell number and composition in the tumor tissue) as well as preliminary clinical efficacy (tumor mass reduction; anticachexia effect)In part A of the trial (dose escalation) up to 24 subjects will receive escalating doses of CTL-002 IV (0.3 – 20 mg/kg) in a ”mono-followed-by-combination”-design with CTL-002 given as monotherapy and followed by combination with an anti-PD-1 checkpoint inhibitor. In part B (expansion) up to 5 cohorts with up to 25 subjects per cohort with defined tumor entities expected to be GDF-15 dependent will be treated to determine the recommended phase 2 dose (RP2D) and further evaluate safety and preliminary efficacy of CTL-002 monotherapy and the combination.The study was initiated in December 2020 and enrolled the first patient on Dec 09, 2020. Cohort 4 is ongoing at time of submission (07/2021) and so far no DLT has occurred. Updated safety, biomarker and response assessments will be reported at the meeting. The ClinicalTrials.gov Identifier is NCT04725474. For more information please contact info@catalym.com. Trial Registration NCT04725474