DNA methylome biomarkers of rheumatoid arthritis-associated interstitial lung disease reflecting lung fibrosis pathways, an exploratory case–control study

Rheumatoid Arthritis-associated Interstitial Lung Disease (RA-ILD) significantly reduces life quality and survival, necessitating improvements in its understanding and clinical management. We addressed these goals using DNA methylation analysis, which has not been done in RA-ILD samples, by comparin...

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Autores: Kaczmarczyk, Bartosz, De la Calle Fabregat, Carlos, Conde, Adrian, Duarte, Ana Catarina, Mena Vazquez, Natalia, Fernandez Nebro, Antonio, Triguero Martinez, Ana, Castañeda, Santos, Dos Santos Sobrin, Raquel, Mera Varela, Antonio, Lopez Pedrera, Chary, Escudero Contreras, Alejandro, Vela Casasempere, Paloma, Molina, Maria, Narvaez, Javier, Retuerto Guerrero, Miriam, Pablos, Jose L., Sarmiento Monroy, Juan C., Sanmarti, Raimon, González-Gay Mantecón, Miguel Ángel
Tipo de recurso: artículo
Fecha de publicación:2025
País:España
Institución:Universidad de Cantabria (UC)
Repositorio:UCrea Repositorio Abierto de la Universidad de Cantabria
Idioma:inglés
OAI Identifier:oai:repositorio.unican.es:10902/37457
Acceso en línea:https://hdl.handle.net/10902/37457
Access Level:acceso abierto
Palabra clave:Rheumatoid arthritis
Interstitial lung disease
Biomarkers
Lung fibrosis
DNA methylation
Epigenetics
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spelling DNA methylome biomarkers of rheumatoid arthritis-associated interstitial lung disease reflecting lung fibrosis pathways, an exploratory case–control studyKaczmarczyk, BartoszDe la Calle Fabregat, CarlosConde, AdrianDuarte, Ana CatarinaMena Vazquez, NataliaFernandez Nebro, AntonioTriguero Martinez, AnaCastañeda, SantosDos Santos Sobrin, RaquelMera Varela, AntonioLopez Pedrera, CharyEscudero Contreras, AlejandroVela Casasempere, PalomaMolina, MariaNarvaez, JavierRetuerto Guerrero, MiriamPablos, Jose L.Sarmiento Monroy, Juan C.Sanmarti, RaimonGonzález-Gay Mantecón, Miguel ÁngelRheumatoid arthritisInterstitial lung diseaseBiomarkersLung fibrosisDNA methylationEpigeneticsRheumatoid Arthritis-associated Interstitial Lung Disease (RA-ILD) significantly reduces life quality and survival, necessitating improvements in its understanding and clinical management. We addressed these goals using DNA methylation analysis, which has not been done in RA-ILD samples, by comparing 32 RA patients with ILD diagnosed less than one year before (cases) and 32 matched RA patients without ILD (controls). This analysis identified 6679 differentially methylated positions (DMPs) with ?? ? 2% and FDR < 0.05, and 576 differentially methylated regions in RA-ILD. Some DMPs were near mucin, collagen, and telomere maintenance genes. Also, the most notably enriched gene set (up to padj = 1.9 × 10?38) included genes overexpressed in fibrosis by monocytes and alveolar macrophages. Other significantly enriched gene sets, known to be dysregulated in fibrosis, included the mitotic spindle and the Rho GTPases. Additionally, analysis of transcription factor binding sites around DMPs showed unique enrichment near the liver X receptor element (LXRE), which is associated with fibrosis in multiple tissues. These results were consistent and unaffected by stricter significance thresholds. They indicated that differential DNA methylation may serve as blood biomarkers for RA-ILD including some related to lung fibrosis pathways.Funding for this study was provided by the Instituto de Salud Carlos III (ISCIII, Spain) through grants PI23/00841, PI20/01268 and RD21/0002/0003, co-funded by the European Union by the ERDF “A way fo making Europe” and Next Generation EU/PRTR, respectively.Universidad de Cantabria20252025-01-01journal articlehttp://purl.org/coar/resource_type/c_6501NAhttp://purl.org/coar/version/c_be7fb7dd8ff6fe43info:eu-repo/semantics/articlehttps://hdl.handle.net/10902/37457Scientific Reports, 2025, 15, 15123reponame:UCrea Repositorio Abierto de la Universidad de Cantabriainstname:Universidad de Cantabria (UC)Inglésengopen accesshttp://purl.org/coar/access_right/c_abf2Attribution-NonCommercial-NoDerivatives 4.0 Internationalhttp://creativecommons.org/licenses/by-nc-nd/4.0/info:eu-repo/semantics/openAccessoai:repositorio.unican.es:10902/374572026-06-02T12:39:31Z
dc.title.none.fl_str_mv DNA methylome biomarkers of rheumatoid arthritis-associated interstitial lung disease reflecting lung fibrosis pathways, an exploratory case–control study
title DNA methylome biomarkers of rheumatoid arthritis-associated interstitial lung disease reflecting lung fibrosis pathways, an exploratory case–control study
spellingShingle DNA methylome biomarkers of rheumatoid arthritis-associated interstitial lung disease reflecting lung fibrosis pathways, an exploratory case–control study
Kaczmarczyk, Bartosz
Rheumatoid arthritis
Interstitial lung disease
Biomarkers
Lung fibrosis
DNA methylation
Epigenetics
title_short DNA methylome biomarkers of rheumatoid arthritis-associated interstitial lung disease reflecting lung fibrosis pathways, an exploratory case–control study
title_full DNA methylome biomarkers of rheumatoid arthritis-associated interstitial lung disease reflecting lung fibrosis pathways, an exploratory case–control study
title_fullStr DNA methylome biomarkers of rheumatoid arthritis-associated interstitial lung disease reflecting lung fibrosis pathways, an exploratory case–control study
title_full_unstemmed DNA methylome biomarkers of rheumatoid arthritis-associated interstitial lung disease reflecting lung fibrosis pathways, an exploratory case–control study
title_sort DNA methylome biomarkers of rheumatoid arthritis-associated interstitial lung disease reflecting lung fibrosis pathways, an exploratory case–control study
dc.creator.none.fl_str_mv Kaczmarczyk, Bartosz
De la Calle Fabregat, Carlos
Conde, Adrian
Duarte, Ana Catarina
Mena Vazquez, Natalia
Fernandez Nebro, Antonio
Triguero Martinez, Ana
Castañeda, Santos
Dos Santos Sobrin, Raquel
Mera Varela, Antonio
Lopez Pedrera, Chary
Escudero Contreras, Alejandro
Vela Casasempere, Paloma
Molina, Maria
Narvaez, Javier
Retuerto Guerrero, Miriam
Pablos, Jose L.
Sarmiento Monroy, Juan C.
Sanmarti, Raimon
González-Gay Mantecón, Miguel Ángel
author Kaczmarczyk, Bartosz
author_facet Kaczmarczyk, Bartosz
De la Calle Fabregat, Carlos
Conde, Adrian
Duarte, Ana Catarina
Mena Vazquez, Natalia
Fernandez Nebro, Antonio
Triguero Martinez, Ana
Castañeda, Santos
Dos Santos Sobrin, Raquel
Mera Varela, Antonio
Lopez Pedrera, Chary
Escudero Contreras, Alejandro
Vela Casasempere, Paloma
Molina, Maria
Narvaez, Javier
Retuerto Guerrero, Miriam
Pablos, Jose L.
Sarmiento Monroy, Juan C.
Sanmarti, Raimon
González-Gay Mantecón, Miguel Ángel
author_role author
author2 De la Calle Fabregat, Carlos
Conde, Adrian
Duarte, Ana Catarina
Mena Vazquez, Natalia
Fernandez Nebro, Antonio
Triguero Martinez, Ana
Castañeda, Santos
Dos Santos Sobrin, Raquel
Mera Varela, Antonio
Lopez Pedrera, Chary
Escudero Contreras, Alejandro
Vela Casasempere, Paloma
Molina, Maria
Narvaez, Javier
Retuerto Guerrero, Miriam
Pablos, Jose L.
Sarmiento Monroy, Juan C.
Sanmarti, Raimon
González-Gay Mantecón, Miguel Ángel
author2_role author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
dc.contributor.none.fl_str_mv Universidad de Cantabria
dc.subject.none.fl_str_mv Rheumatoid arthritis
Interstitial lung disease
Biomarkers
Lung fibrosis
DNA methylation
Epigenetics
topic Rheumatoid arthritis
Interstitial lung disease
Biomarkers
Lung fibrosis
DNA methylation
Epigenetics
description Rheumatoid Arthritis-associated Interstitial Lung Disease (RA-ILD) significantly reduces life quality and survival, necessitating improvements in its understanding and clinical management. We addressed these goals using DNA methylation analysis, which has not been done in RA-ILD samples, by comparing 32 RA patients with ILD diagnosed less than one year before (cases) and 32 matched RA patients without ILD (controls). This analysis identified 6679 differentially methylated positions (DMPs) with ?? ? 2% and FDR < 0.05, and 576 differentially methylated regions in RA-ILD. Some DMPs were near mucin, collagen, and telomere maintenance genes. Also, the most notably enriched gene set (up to padj = 1.9 × 10?38) included genes overexpressed in fibrosis by monocytes and alveolar macrophages. Other significantly enriched gene sets, known to be dysregulated in fibrosis, included the mitotic spindle and the Rho GTPases. Additionally, analysis of transcription factor binding sites around DMPs showed unique enrichment near the liver X receptor element (LXRE), which is associated with fibrosis in multiple tissues. These results were consistent and unaffected by stricter significance thresholds. They indicated that differential DNA methylation may serve as blood biomarkers for RA-ILD including some related to lung fibrosis pathways.
publishDate 2025
dc.date.none.fl_str_mv 2025
2025-01-01
dc.type.none.fl_str_mv journal article
http://purl.org/coar/resource_type/c_6501
NA
http://purl.org/coar/version/c_be7fb7dd8ff6fe43
dc.type.openaire.fl_str_mv info:eu-repo/semantics/article
format article
dc.identifier.none.fl_str_mv https://hdl.handle.net/10902/37457
url https://hdl.handle.net/10902/37457
dc.language.none.fl_str_mv Inglés
eng
language_invalid_str_mv Inglés
language eng
dc.rights.none.fl_str_mv open access
http://purl.org/coar/access_right/c_abf2
Attribution-NonCommercial-NoDerivatives 4.0 International
http://creativecommons.org/licenses/by-nc-nd/4.0/
dc.rights.openaire.fl_str_mv info:eu-repo/semantics/openAccess
rights_invalid_str_mv open access
http://purl.org/coar/access_right/c_abf2
Attribution-NonCommercial-NoDerivatives 4.0 International
http://creativecommons.org/licenses/by-nc-nd/4.0/
eu_rights_str_mv openAccess
dc.source.none.fl_str_mv Scientific Reports, 2025, 15, 15123
reponame:UCrea Repositorio Abierto de la Universidad de Cantabria
instname:Universidad de Cantabria (UC)
instname_str Universidad de Cantabria (UC)
reponame_str UCrea Repositorio Abierto de la Universidad de Cantabria
collection UCrea Repositorio Abierto de la Universidad de Cantabria
repository.name.fl_str_mv
repository.mail.fl_str_mv
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