IL-2 and Anti-TGF-β Promote NK Cell Reconstitution and Anti-tumor Effects after Syngeneic Hematopoietic Stem Cell Transplantation

The failure of autologous hematopoietic stem cell transplantation (HSCT) has been associated with a profound immunodeficiency that follows shortly after treatment, which renders patients susceptible to opportunistic infections and/or cancer relapse. Thus, given the additional immunosuppressive pathw...

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Autores: Álvarez, M. (Maite)|||/items/869c2b2f-e806-47ac-8d76-303de908d205, Dunai, C. (Cordelia)|||/items/0e85a6b8-62fb-46d9-8450-dd528b66d1d7, Khuat, L.T. (Lam T.)|||/items/dc0231cf-8f40-4dae-8186-bc8cc8d43a17, Aguilar, E.G. (Ethan G.)|||/items/55b6679c-87a8-45d9-8159-c9fe3df147a5, Barao, I. (Isabel)|||/items/01c9f74e-ac99-4327-a77c-3c168b2010d1, Murphy, W.J. (William J.)|||/items/a1db583e-c3c0-4814-9585-f5b7ef75e50e
Tipo de recurso: artículo
Fecha de publicación:2020
País:España
Institución:Universidad de Navarra
Repositorio:Dadun. Depósito Académico Digital de la Universidad de Navarra
Idioma:inglés
OAI Identifier:oai:dadun.unav.edu:10171/66341
Acceso en línea:https://hdl.handle.net/10171/66341
Access Level:acceso abierto
Palabra clave:HSCT
IL-2
TGF-β
NK cells
Myeloid cells
Immune reconstitution
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spelling IL-2 and Anti-TGF-β Promote NK Cell Reconstitution and Anti-tumor Effects after Syngeneic Hematopoietic Stem Cell TransplantationÁlvarez, M. (Maite)|||/items/869c2b2f-e806-47ac-8d76-303de908d205Dunai, C. (Cordelia)|||/items/0e85a6b8-62fb-46d9-8450-dd528b66d1d7Khuat, L.T. (Lam T.)|||/items/dc0231cf-8f40-4dae-8186-bc8cc8d43a17Aguilar, E.G. (Ethan G.)|||/items/55b6679c-87a8-45d9-8159-c9fe3df147a5Barao, I. (Isabel)|||/items/01c9f74e-ac99-4327-a77c-3c168b2010d1Murphy, W.J. (William J.)|||/items/a1db583e-c3c0-4814-9585-f5b7ef75e50eHSCTIL-2TGF-βNK cellsMyeloid cellsImmune reconstitutionThe failure of autologous hematopoietic stem cell transplantation (HSCT) has been associated with a profound immunodeficiency that follows shortly after treatment, which renders patients susceptible to opportunistic infections and/or cancer relapse. Thus, given the additional immunosuppressive pathways involved in immune evasion in cancer, strategies that induce a faster reconstitution of key immune effector cells are needed. Natural killer (NK) cells mediate potent anti-tumor effector functions and are the first immune cells to repopulate after HSCT. TGF-β is a potent immunosuppressive cytokine that can impede both the development and function of immune cells. Here, we evaluated the use of an immunotherapeutic regimen that combines low dose of IL-2, an NK cell stimulatory signal, with TGF-β neutralization, in order to accelerate NK cell reconstitution following congenic HSCT in mice by providing stimulatory signals yet also abrogating inhibitory ones. This therapy led to a marked expansion of NK cells and accelerated NK cell maturation. Following HSCT, mature NK cells from the treated recipients displayed an activated phenotype and enhanced anti-tumor responses both in vitro and in vivo. No overt toxicities or adverse effects were observed in the treated recipients. However, these stimulatory effects on NK cell recovery were predicated upon continuous treatment as cessation of treatment led to return to baseline levels and to no improvement of overall immune recovery when assessed at later time-points, indicating strict regulatory control of the NK cell compartment. Overall, this study still demonstrates that therapies that combine positive and negative signals can be plausible strategies to accelerate NK cell reconstitution following HSCT and augment anti-tumor efficacy.Dadun. Depósito Académico Digital Universidad de Navarra20232023-05-2320202020-01-0120202020-01-01journal articlehttp://purl.org/coar/resource_type/c_6501info:eu-repo/semantics/articleapplication/pdfhttps://hdl.handle.net/10171/66341reponame:Dadun. Depósito Académico Digital de la Universidad de Navarrainstname:Universidad de NavarraInglésengopen accesshttp://purl.org/coar/access_right/c_abf2info:eu-repo/semantics/openAccessoai:dadun.unav.edu:10171/663412026-06-21T12:47:57Z
dc.title.none.fl_str_mv IL-2 and Anti-TGF-β Promote NK Cell Reconstitution and Anti-tumor Effects after Syngeneic Hematopoietic Stem Cell Transplantation
title IL-2 and Anti-TGF-β Promote NK Cell Reconstitution and Anti-tumor Effects after Syngeneic Hematopoietic Stem Cell Transplantation
spellingShingle IL-2 and Anti-TGF-β Promote NK Cell Reconstitution and Anti-tumor Effects after Syngeneic Hematopoietic Stem Cell Transplantation
Álvarez, M. (Maite)|||/items/869c2b2f-e806-47ac-8d76-303de908d205
HSCT
IL-2
TGF-β
NK cells
Myeloid cells
Immune reconstitution
title_short IL-2 and Anti-TGF-β Promote NK Cell Reconstitution and Anti-tumor Effects after Syngeneic Hematopoietic Stem Cell Transplantation
title_full IL-2 and Anti-TGF-β Promote NK Cell Reconstitution and Anti-tumor Effects after Syngeneic Hematopoietic Stem Cell Transplantation
title_fullStr IL-2 and Anti-TGF-β Promote NK Cell Reconstitution and Anti-tumor Effects after Syngeneic Hematopoietic Stem Cell Transplantation
title_full_unstemmed IL-2 and Anti-TGF-β Promote NK Cell Reconstitution and Anti-tumor Effects after Syngeneic Hematopoietic Stem Cell Transplantation
title_sort IL-2 and Anti-TGF-β Promote NK Cell Reconstitution and Anti-tumor Effects after Syngeneic Hematopoietic Stem Cell Transplantation
dc.creator.none.fl_str_mv Álvarez, M. (Maite)|||/items/869c2b2f-e806-47ac-8d76-303de908d205
Dunai, C. (Cordelia)|||/items/0e85a6b8-62fb-46d9-8450-dd528b66d1d7
Khuat, L.T. (Lam T.)|||/items/dc0231cf-8f40-4dae-8186-bc8cc8d43a17
Aguilar, E.G. (Ethan G.)|||/items/55b6679c-87a8-45d9-8159-c9fe3df147a5
Barao, I. (Isabel)|||/items/01c9f74e-ac99-4327-a77c-3c168b2010d1
Murphy, W.J. (William J.)|||/items/a1db583e-c3c0-4814-9585-f5b7ef75e50e
author Álvarez, M. (Maite)|||/items/869c2b2f-e806-47ac-8d76-303de908d205
author_facet Álvarez, M. (Maite)|||/items/869c2b2f-e806-47ac-8d76-303de908d205
Dunai, C. (Cordelia)|||/items/0e85a6b8-62fb-46d9-8450-dd528b66d1d7
Khuat, L.T. (Lam T.)|||/items/dc0231cf-8f40-4dae-8186-bc8cc8d43a17
Aguilar, E.G. (Ethan G.)|||/items/55b6679c-87a8-45d9-8159-c9fe3df147a5
Barao, I. (Isabel)|||/items/01c9f74e-ac99-4327-a77c-3c168b2010d1
Murphy, W.J. (William J.)|||/items/a1db583e-c3c0-4814-9585-f5b7ef75e50e
author_role author
author2 Dunai, C. (Cordelia)|||/items/0e85a6b8-62fb-46d9-8450-dd528b66d1d7
Khuat, L.T. (Lam T.)|||/items/dc0231cf-8f40-4dae-8186-bc8cc8d43a17
Aguilar, E.G. (Ethan G.)|||/items/55b6679c-87a8-45d9-8159-c9fe3df147a5
Barao, I. (Isabel)|||/items/01c9f74e-ac99-4327-a77c-3c168b2010d1
Murphy, W.J. (William J.)|||/items/a1db583e-c3c0-4814-9585-f5b7ef75e50e
author2_role author
author
author
author
author
dc.contributor.none.fl_str_mv Dadun. Depósito Académico Digital Universidad de Navarra
dc.subject.none.fl_str_mv HSCT
IL-2
TGF-β
NK cells
Myeloid cells
Immune reconstitution
topic HSCT
IL-2
TGF-β
NK cells
Myeloid cells
Immune reconstitution
description The failure of autologous hematopoietic stem cell transplantation (HSCT) has been associated with a profound immunodeficiency that follows shortly after treatment, which renders patients susceptible to opportunistic infections and/or cancer relapse. Thus, given the additional immunosuppressive pathways involved in immune evasion in cancer, strategies that induce a faster reconstitution of key immune effector cells are needed. Natural killer (NK) cells mediate potent anti-tumor effector functions and are the first immune cells to repopulate after HSCT. TGF-β is a potent immunosuppressive cytokine that can impede both the development and function of immune cells. Here, we evaluated the use of an immunotherapeutic regimen that combines low dose of IL-2, an NK cell stimulatory signal, with TGF-β neutralization, in order to accelerate NK cell reconstitution following congenic HSCT in mice by providing stimulatory signals yet also abrogating inhibitory ones. This therapy led to a marked expansion of NK cells and accelerated NK cell maturation. Following HSCT, mature NK cells from the treated recipients displayed an activated phenotype and enhanced anti-tumor responses both in vitro and in vivo. No overt toxicities or adverse effects were observed in the treated recipients. However, these stimulatory effects on NK cell recovery were predicated upon continuous treatment as cessation of treatment led to return to baseline levels and to no improvement of overall immune recovery when assessed at later time-points, indicating strict regulatory control of the NK cell compartment. Overall, this study still demonstrates that therapies that combine positive and negative signals can be plausible strategies to accelerate NK cell reconstitution following HSCT and augment anti-tumor efficacy.
publishDate 2020
dc.date.none.fl_str_mv 2020
2020-01-01
2020
2020-01-01
2023
2023-05-23
dc.type.none.fl_str_mv journal article
http://purl.org/coar/resource_type/c_6501
dc.type.openaire.fl_str_mv info:eu-repo/semantics/article
format article
dc.identifier.none.fl_str_mv https://hdl.handle.net/10171/66341
url https://hdl.handle.net/10171/66341
dc.language.none.fl_str_mv Inglés
eng
language_invalid_str_mv Inglés
language eng
dc.rights.none.fl_str_mv open access
http://purl.org/coar/access_right/c_abf2
dc.rights.openaire.fl_str_mv info:eu-repo/semantics/openAccess
rights_invalid_str_mv open access
http://purl.org/coar/access_right/c_abf2
eu_rights_str_mv openAccess
dc.format.none.fl_str_mv application/pdf
dc.source.none.fl_str_mv reponame:Dadun. Depósito Académico Digital de la Universidad de Navarra
instname:Universidad de Navarra
instname_str Universidad de Navarra
reponame_str Dadun. Depósito Académico Digital de la Universidad de Navarra
collection Dadun. Depósito Académico Digital de la Universidad de Navarra
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