The role of platelet-activating factor antagonists in the prohylasis of postransplant acute tubular necrosis

Renal reperfusion injury can be defined as the renal damage suffered by a kidney because of reestablishment of blood flow prolonged ischemia. Increased permeability for calcium ions, subsequent activation of endogenous phospholipases, and damage mediated by oxygen free radicals are involved in this...

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Detalles Bibliográficos
Autores: Grinyó Boira, Josep M., Torras Ambròs, Joan, Valles, Joaquim
Tipo de recurso: artículo
Estado:Versión publicada
Fecha de publicación:1996
País:España
Institución:Universidad de Barcelona
Repositorio:Dipòsit Digital de la UB
OAI Identifier:oai:diposit.ub.edu:2445/156959
Acceso en línea:https://hdl.handle.net/2445/156959
Access Level:acceso abierto
Palabra clave:Trasplantament renal
Necrosi
Agregació plaquetària
Kidney transplantation
Necrosis
Blood platelet aggregation
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spelling The role of platelet-activating factor antagonists in the prohylasis of postransplant acute tubular necrosisGrinyó Boira, Josep M.Torras Ambròs, JoanValles, JoaquimTrasplantament renalNecrosiAgregació plaquetàriaKidney transplantationNecrosisBlood platelet aggregationRenal reperfusion injury can be defined as the renal damage suffered by a kidney because of reestablishment of blood flow prolonged ischemia. Increased permeability for calcium ions, subsequent activation of endogenous phospholipases, and damage mediated by oxygen free radicals are involved in this reperfusion injury. Inhibition of inflammatory mediators of endothelial origin, such as superoxide radicals and platelet-activating factor (PAF), is a possible way of protecting the injured endothelium following blood reperfusion.Marcel Dekker1996info:eu-repo/semantics/articleinfo:eu-repo/semantics/publishedVersionapplication/pdfhttps://hdl.handle.net/2445/156959Articles publicats en revistes (Ciències Clíniques)reponame:Dipòsit Digital de la UBinstname:Universidad de BarcelonaInglésReproducció del document publicat a: https://doi.org/10.3109/08860229609052815Renal Failure, 1996, vol. 18, num. 3, p. 445-451https://doi.org/10.3109/08860229609052815cc-by (c) Grinyó Boira, Josep M. et al., 1996http://creativecommons.org/licenses/by/3.0/esinfo:eu-repo/semantics/openAccessoai:diposit.ub.edu:2445/1569592026-05-27T06:46:51Z
dc.title.none.fl_str_mv The role of platelet-activating factor antagonists in the prohylasis of postransplant acute tubular necrosis
title The role of platelet-activating factor antagonists in the prohylasis of postransplant acute tubular necrosis
spellingShingle The role of platelet-activating factor antagonists in the prohylasis of postransplant acute tubular necrosis
Grinyó Boira, Josep M.
Trasplantament renal
Necrosi
Agregació plaquetària
Kidney transplantation
Necrosis
Blood platelet aggregation
title_short The role of platelet-activating factor antagonists in the prohylasis of postransplant acute tubular necrosis
title_full The role of platelet-activating factor antagonists in the prohylasis of postransplant acute tubular necrosis
title_fullStr The role of platelet-activating factor antagonists in the prohylasis of postransplant acute tubular necrosis
title_full_unstemmed The role of platelet-activating factor antagonists in the prohylasis of postransplant acute tubular necrosis
title_sort The role of platelet-activating factor antagonists in the prohylasis of postransplant acute tubular necrosis
dc.creator.none.fl_str_mv Grinyó Boira, Josep M.
Torras Ambròs, Joan
Valles, Joaquim
author Grinyó Boira, Josep M.
author_facet Grinyó Boira, Josep M.
Torras Ambròs, Joan
Valles, Joaquim
author_role author
author2 Torras Ambròs, Joan
Valles, Joaquim
author2_role author
author
dc.subject.none.fl_str_mv Trasplantament renal
Necrosi
Agregació plaquetària
Kidney transplantation
Necrosis
Blood platelet aggregation
topic Trasplantament renal
Necrosi
Agregació plaquetària
Kidney transplantation
Necrosis
Blood platelet aggregation
description Renal reperfusion injury can be defined as the renal damage suffered by a kidney because of reestablishment of blood flow prolonged ischemia. Increased permeability for calcium ions, subsequent activation of endogenous phospholipases, and damage mediated by oxygen free radicals are involved in this reperfusion injury. Inhibition of inflammatory mediators of endothelial origin, such as superoxide radicals and platelet-activating factor (PAF), is a possible way of protecting the injured endothelium following blood reperfusion.
publishDate 1996
dc.date.none.fl_str_mv 1996
dc.type.none.fl_str_mv info:eu-repo/semantics/article
info:eu-repo/semantics/publishedVersion
format article
status_str publishedVersion
dc.identifier.none.fl_str_mv https://hdl.handle.net/2445/156959
url https://hdl.handle.net/2445/156959
dc.language.none.fl_str_mv Inglés
language_invalid_str_mv Inglés
dc.relation.none.fl_str_mv Reproducció del document publicat a: https://doi.org/10.3109/08860229609052815
Renal Failure, 1996, vol. 18, num. 3, p. 445-451
https://doi.org/10.3109/08860229609052815
dc.rights.none.fl_str_mv cc-by (c) Grinyó Boira, Josep M. et al., 1996
http://creativecommons.org/licenses/by/3.0/es
info:eu-repo/semantics/openAccess
rights_invalid_str_mv cc-by (c) Grinyó Boira, Josep M. et al., 1996
http://creativecommons.org/licenses/by/3.0/es
eu_rights_str_mv openAccess
dc.format.none.fl_str_mv application/pdf
dc.publisher.none.fl_str_mv Marcel Dekker
publisher.none.fl_str_mv Marcel Dekker
dc.source.none.fl_str_mv Articles publicats en revistes (Ciències Clíniques)
reponame:Dipòsit Digital de la UB
instname:Universidad de Barcelona
instname_str Universidad de Barcelona
reponame_str Dipòsit Digital de la UB
collection Dipòsit Digital de la UB
repository.name.fl_str_mv
repository.mail.fl_str_mv
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score 15,300719

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