Multiple immunofluorescence assay identifies upregulation of Active β-catenin in prostate cancer

Objectives: To apply a systems pathology-based approach to the quantification of nuclear Active β-catenin and human leukocyte antigen class I, and assess the biomarker involvement in a cohort of prostate tumor patients. Results: The systems pathology approach applied allows a precise quantification...

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Detalles Bibliográficos
Autores: Puig, Pere, Erill, Nadina, Terricabras, Marta, Subirana, Isaac, González-García, Judit, Asensi-Puig, Adrià, Donovan, Michael J., Mengual Brichs, Lourdes, Agulló-Ortuño, M. Teresa, Olivan Riera, Mireia, Alcaraz Asensio, Antonio, López-Martín, José A., Torres, Inés de, Rodríguez Peralto, José Luis, Rodríguez-Antolín, Alfredo, Morote, Juan, González-Rumayor, Víctor
Tipo de recurso: artículo
Estado:Versión publicada
Fecha de publicación:2019
País:España
Institución:Varias* (Consorci de Biblioteques Universitáries de Catalunya, Centre de Serveis Científics i Acadèmics de Catalunya)
Repositorio:Recercat. Dipósit de la Recerca de Catalunya
OAI Identifier:oai:recercat.cat:2445/164367
Acceso en línea:https://hdl.handle.net/2445/164367
Access Level:acceso abierto
Palabra clave:Marcadors bioquímics
Càncer de pròstata
Proteïnes
Biochemical markers
Prostate cancer
Proteins
Descripción
Sumario:Objectives: To apply a systems pathology-based approach to the quantification of nuclear Active β-catenin and human leukocyte antigen class I, and assess the biomarker involvement in a cohort of prostate tumor patients. Results: The systems pathology approach applied allows a precise quantification of the marker expression in the different cell compartments as well as the determination of the areas that coexpress two markers. Our data shows that the accumulation of β-catenin in the nuclear compartment is significantly decreased in the adjacent normal areas when compared to tumor of the same patients (p < 0.001). In conclusion, the application of this novel multiple immunofluorescence assay demonstrates that the upregulation of Active β-catenin is a relatively common feature of prostate tumor development, and further supports the activation of the Wnt/β-catenin pathway in prostate cancer progression.