Influence of Cellular Aging on Liver Stiffness in Patients With Hepatitis C Virus Achieving Sustained Viral Response

Background. Liver stiffness (LS) is not reduced in 10%–30% of patients who achieve sustained viral response (SVR) after hepatitis C virus (HCV) elimination with direct-acting antivirals (DAA). Our aim was to analyze whether the parameters associated with cellular aging measured at the DAA initiation...

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Autores: González-Serna Martín, Manuel Alejandro, Corma Gómez, Anaïs, Cano Rodríguez, María Mercedes, Rubio Sánchez, Ricardo, Martín Sierra, Carmen, Rincón, Pilar, Martín Carmona, Jesica, Pérez, Margarita, Pineda Vergara, Juan Antonio, Real Navarrete, Luis Miguel, Macías Sánchez, Juan
Tipo de recurso: artículo
Estado:Versión publicada
Fecha de publicación:2025
País:España
Institución:Instituto Valenciano de Investigaciones Agrarias (IVIA)
Repositorio:idUS. Depósito de Investigación de la Universidad de Sevilla
OAI Identifier:oai:idus.us.es:11441/172665
Acceso en línea:https://hdl.handle.net/11441/172665
https://doi.org/10.1093/infdis/jiaf087
Access Level:acceso abierto
Palabra clave:Cellular aging
Liver stiffness
Telomere length
SVR
DAA.
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spelling Influence of Cellular Aging on Liver Stiffness in Patients With Hepatitis C Virus Achieving Sustained Viral ResponseGonzález-Serna Martín, Manuel AlejandroCorma Gómez, AnaïsCano Rodríguez, María MercedesRubio Sánchez, RicardoMartín Sierra, CarmenRincón, PilarMartín Carmona, JesicaPérez, MargaritaPineda Vergara, Juan AntonioReal Navarrete, Luis MiguelMacías Sánchez, JuanCellular agingLiver stiffnessTelomere lengthSVRDAA.Background. Liver stiffness (LS) is not reduced in 10%–30% of patients who achieve sustained viral response (SVR) after hepatitis C virus (HCV) elimination with direct-acting antivirals (DAA). Our aim was to analyze whether the parameters associated with cellular aging measured at the DAA initiation date are related to LS reduction upon achieving SVR. Methods. In a prospective cohort study (GEHEP-011) we measured several parameters associated with cellular aging, such as telomere attrition, mitochondrial alterations, and soluble biomarkers associated with senescence-associated secretory phenotype at the DAA initiation date, and examined their associations with a significant (≥20%) LS decrease at the SVR time point. Results. In total, 175 individuals were included in this study. In 101 (57.7%) patients, the LS reduction was ≥20% at SVR. In the multivariate analysis adjusted for sex, age, CXCL10, hsPCR, and CCL11 levels, greater relative telomere length (RTL) emerged as the sole variable independently associated with a significant LS decrease in SVR (1.102; 95% confidence interval, 1.001–1.1214; P = .047). Furthermore, changes in LS, including significant decrease, decrease <20%, or increase, were congruently associated with RTL (P = .011). Conclusions. Greater RTL was independently associated with a significant LS reduction in SVR. Thus, increased cellular aging may be responsible for the absence of liver regeneration after HCV eradication. Further studies are required to assess the long-term effects of cellular aging after SVR.OxfordFisiologíaMedicinaInstituto de Salud Carlos III2025info:eu-repo/semantics/articleinfo:eu-repo/semantics/publishedVersionapplication/pdfapplication/pdfhttps://hdl.handle.net/11441/172665https://doi.org/10.1093/infdis/jiaf087reponame:idUS. Depósito de Investigación de la Universidad de Sevillainstname:Instituto Valenciano de Investigaciones Agrarias (IVIA)InglésThe Journal of Infectious Diseases, jiaf087.PI20/01457https://doi.org/10.1093/infdis/jiaf087info:eu-repo/semantics/openAccessoai:idus.us.es:11441/1726652026-06-17T12:51:07Z
dc.title.none.fl_str_mv Influence of Cellular Aging on Liver Stiffness in Patients With Hepatitis C Virus Achieving Sustained Viral Response
title Influence of Cellular Aging on Liver Stiffness in Patients With Hepatitis C Virus Achieving Sustained Viral Response
spellingShingle Influence of Cellular Aging on Liver Stiffness in Patients With Hepatitis C Virus Achieving Sustained Viral Response
González-Serna Martín, Manuel Alejandro
Cellular aging
Liver stiffness
Telomere length
SVR
DAA.
title_short Influence of Cellular Aging on Liver Stiffness in Patients With Hepatitis C Virus Achieving Sustained Viral Response
title_full Influence of Cellular Aging on Liver Stiffness in Patients With Hepatitis C Virus Achieving Sustained Viral Response
title_fullStr Influence of Cellular Aging on Liver Stiffness in Patients With Hepatitis C Virus Achieving Sustained Viral Response
title_full_unstemmed Influence of Cellular Aging on Liver Stiffness in Patients With Hepatitis C Virus Achieving Sustained Viral Response
title_sort Influence of Cellular Aging on Liver Stiffness in Patients With Hepatitis C Virus Achieving Sustained Viral Response
dc.creator.none.fl_str_mv González-Serna Martín, Manuel Alejandro
Corma Gómez, Anaïs
Cano Rodríguez, María Mercedes
Rubio Sánchez, Ricardo
Martín Sierra, Carmen
Rincón, Pilar
Martín Carmona, Jesica
Pérez, Margarita
Pineda Vergara, Juan Antonio
Real Navarrete, Luis Miguel
Macías Sánchez, Juan
author González-Serna Martín, Manuel Alejandro
author_facet González-Serna Martín, Manuel Alejandro
Corma Gómez, Anaïs
Cano Rodríguez, María Mercedes
Rubio Sánchez, Ricardo
Martín Sierra, Carmen
Rincón, Pilar
Martín Carmona, Jesica
Pérez, Margarita
Pineda Vergara, Juan Antonio
Real Navarrete, Luis Miguel
Macías Sánchez, Juan
author_role author
author2 Corma Gómez, Anaïs
Cano Rodríguez, María Mercedes
Rubio Sánchez, Ricardo
Martín Sierra, Carmen
Rincón, Pilar
Martín Carmona, Jesica
Pérez, Margarita
Pineda Vergara, Juan Antonio
Real Navarrete, Luis Miguel
Macías Sánchez, Juan
author2_role author
author
author
author
author
author
author
author
author
author
dc.contributor.none.fl_str_mv Fisiología
Medicina
Instituto de Salud Carlos III
dc.subject.none.fl_str_mv Cellular aging
Liver stiffness
Telomere length
SVR
DAA.
topic Cellular aging
Liver stiffness
Telomere length
SVR
DAA.
description Background. Liver stiffness (LS) is not reduced in 10%–30% of patients who achieve sustained viral response (SVR) after hepatitis C virus (HCV) elimination with direct-acting antivirals (DAA). Our aim was to analyze whether the parameters associated with cellular aging measured at the DAA initiation date are related to LS reduction upon achieving SVR. Methods. In a prospective cohort study (GEHEP-011) we measured several parameters associated with cellular aging, such as telomere attrition, mitochondrial alterations, and soluble biomarkers associated with senescence-associated secretory phenotype at the DAA initiation date, and examined their associations with a significant (≥20%) LS decrease at the SVR time point. Results. In total, 175 individuals were included in this study. In 101 (57.7%) patients, the LS reduction was ≥20% at SVR. In the multivariate analysis adjusted for sex, age, CXCL10, hsPCR, and CCL11 levels, greater relative telomere length (RTL) emerged as the sole variable independently associated with a significant LS decrease in SVR (1.102; 95% confidence interval, 1.001–1.1214; P = .047). Furthermore, changes in LS, including significant decrease, decrease <20%, or increase, were congruently associated with RTL (P = .011). Conclusions. Greater RTL was independently associated with a significant LS reduction in SVR. Thus, increased cellular aging may be responsible for the absence of liver regeneration after HCV eradication. Further studies are required to assess the long-term effects of cellular aging after SVR.
publishDate 2025
dc.date.none.fl_str_mv 2025
dc.type.none.fl_str_mv info:eu-repo/semantics/article
info:eu-repo/semantics/publishedVersion
format article
status_str publishedVersion
dc.identifier.none.fl_str_mv https://hdl.handle.net/11441/172665
https://doi.org/10.1093/infdis/jiaf087
url https://hdl.handle.net/11441/172665
https://doi.org/10.1093/infdis/jiaf087
dc.language.none.fl_str_mv Inglés
language_invalid_str_mv Inglés
dc.relation.none.fl_str_mv The Journal of Infectious Diseases, jiaf087.
PI20/01457
https://doi.org/10.1093/infdis/jiaf087
dc.rights.none.fl_str_mv info:eu-repo/semantics/openAccess
eu_rights_str_mv openAccess
dc.format.none.fl_str_mv application/pdf
application/pdf
dc.publisher.none.fl_str_mv Oxford
publisher.none.fl_str_mv Oxford
dc.source.none.fl_str_mv reponame:idUS. Depósito de Investigación de la Universidad de Sevilla
instname:Instituto Valenciano de Investigaciones Agrarias (IVIA)
instname_str Instituto Valenciano de Investigaciones Agrarias (IVIA)
reponame_str idUS. Depósito de Investigación de la Universidad de Sevilla
collection idUS. Depósito de Investigación de la Universidad de Sevilla
repository.name.fl_str_mv
repository.mail.fl_str_mv
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