Metabolic Effects of Oral Phenelzine Treatment on High-Sucrose-Drinking Mice

Phenelzine has been suggested to have an antiobesity effect by inhibiting de novo lipogenesis, which led us to investigate the metabolic effects of oral chronic phenelzine treatment in high-sucrose-drinking mice. Sucrose-drinking mice presented higher body weight gain and adiposity versus controls....

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Detalles Bibliográficos
Autores: Carpene, Christian, Gomez-Zorita, Saioa, Chaplin, Alice, Mercader, Josep
Tipo de recurso: artículo
Fecha de publicación:2018
País:España
Institución:Instituto de Salud Carlos III (ISCIII)
Repositorio:Repisalud
Idioma:inglés
OAI Identifier:oai:repisalud.isciii.es:20.500.12105/22584
Acceso en línea:https://hdl.handle.net/20.500.12105/22584
Access Level:acceso abierto
Palabra clave:Phenelzine
Obesity
Adipocyte
Lipogenesis
Amine oxidases
Hydrogen peroxide
Hyperglycemia
Oxidative stress
Sacarosa
Aumento de Peso
Animales
Ácidos Grasos no Esterificados
Masculino
Administración Oral
Fenelzina
Tejido Adiposo Blanco
Inhibidores de la Monoaminooxidasa
Obesidad
Ratones Endogámicos C57BL
Estrés Oxidativo
Ratones
Lipogénesis
Adiposidad
Oxidative Stress
Administration, Oral
Mice, Inbred C57BL
Monoamine Oxidase Inhibitors
Male
Fatty Acids, Nonesterified
Adiposity
Animals
Weight Gain
Sucrose
Adipose Tissue, White
Mice
Descripción
Sumario:Phenelzine has been suggested to have an antiobesity effect by inhibiting de novo lipogenesis, which led us to investigate the metabolic effects of oral chronic phenelzine treatment in high-sucrose-drinking mice. Sucrose-drinking mice presented higher body weight gain and adiposity versus controls. Phenelzine addition did not decrease such parameters, even though fat pad lipid content and weights were not different from controls. In visceral adipocytes, phenelzine did not impair insulin-stimulated de novo lipogenesis and had no effect on lipolysis. However, phenelzine reduced the mRNA levels of glucose transporters 1 and 4 and phosphoenolpyruvate carboxykinase in inguinal white adipose tissue (iWAT), and altered circulating levels of free fatty acids (FFA) and glycerol. Interestingly, glycemia was restored in phenelzine-treated mice, which also had higher insulinaemia. Phenelzine-treated mice presented higher rectal temperature, which was associated to reduced mRNA levels of uncoupling protein 1 in brown adipose tissue. Furthermore, unlike sucrose-drinking mice, hepatic malondialdehyde levels were not altered. In conclusion, although de novo lipogenesis was not inhibited by phenelzine, the data suggest that the ability to re-esterify FFA is impaired in iWAT. Moreover, the effects on glucose homeostasis and oxidative stress suggest that phenelzine could alleviate obesity-related alterations and deserves further investigation in obesity models.