Helicobacter pylori gamma-glutamyl transpeptidase inhibits autophagy in gastric cancer cells favouring survival of internalised bacteria

Helicobacter pylori (H. pylori) is the major risk factor associated with the development of gastric cancer. Multiple virulence factors contribute to its pathogenesis, and recently, the secreted gamma­glutamyl transpeptidase (GGT) has become of increasing

Detalles Bibliográficos
Autores: Bravo, J, Corvalan, A H, Quest, A F G
Tipo de recurso: artículo
Estado:Versión publicada
Fecha de publicación:2015
País:Chile
Idioma:inglés
OAI Identifier:oai:repositorio.anid.cl:10533/232701
Acceso en línea:https://hdl.handle.net/10533/232701
Access Level:acceso abierto
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spelling Quest, A F GCorvalan, A HBravo, J2018https://hdl.handle.net/10533/232701http://purl.org/coar/access_right/c_abf2Helicobacter pylori gamma-glutamyl transpeptidase inhibits autophagy in gastric cancer cells favouring survival of internalised bacteriaBravo, JCorvalan, A HQuest, A F G2018-12-07T13:33:44Z2022-07-07T21:40:14Z20152018-12-07T13:33:44Z2022-07-07T21:40:14Z2018Helicobacter pylori (H. pylori) is the major risk factor associated with the development of gastric cancer. Multiple virulence factors contribute to its pathogenesis, and recently, the secreted gamma­glutamyl transpeptidase (GGT) has become of increasingRegularFONDECYTFONDECYT11514111151411virtual::37969-1WOS:000437674104015https://hdl.handle.net/10533/232701enghandle/10533/111557handle/10533/111541handle/10533/108045https://2018.febscongress.org/abstract_preview.aspx?idAbstractEnc=4424170094091092097099424170Atribución-NoComercial-SinDerivadas 3.0 Chilehttp://creativecommons.org/licenses/by-nc-nd/3.0/cl/info:eu-repo/semantics/openAccessHelicobacter pylori gamma-glutamyl transpeptidase inhibits autophagy in gastric cancer cells favouring survival of internalised bacteriaFebs Open BioArticuloinfo:eu-repo/semantics/articleinfo:eu-repo/semantics/publishedVersionArticulohttps://hdl.handle.net/10533/232701FONDECYThttp://purl.org/coar/resource_type/c_2df8fbb12134cfd6-1ec3-4390-843f-86682007cf1cvirtual::37969-12134cfd6-1ec3-4390-843f-86682007cf1cvirtual::37969-110533/232701oai:repositorio.anid.cl:10533/2327012023-07-24 16:12:06.75https://repositorio.anid.clRepositorio ANIDaletelier@anid.cl
dc.title.none.fl_str_mv Helicobacter pylori gamma-glutamyl transpeptidase inhibits autophagy in gastric cancer cells favouring survival of internalised bacteria
dc.title.journal.none.fl_str_mv Febs Open Bio
title Helicobacter pylori gamma-glutamyl transpeptidase inhibits autophagy in gastric cancer cells favouring survival of internalised bacteria
spellingShingle Helicobacter pylori gamma-glutamyl transpeptidase inhibits autophagy in gastric cancer cells favouring survival of internalised bacteria
Bravo, J
title_short Helicobacter pylori gamma-glutamyl transpeptidase inhibits autophagy in gastric cancer cells favouring survival of internalised bacteria
title_full Helicobacter pylori gamma-glutamyl transpeptidase inhibits autophagy in gastric cancer cells favouring survival of internalised bacteria
title_fullStr Helicobacter pylori gamma-glutamyl transpeptidase inhibits autophagy in gastric cancer cells favouring survival of internalised bacteria
title_full_unstemmed Helicobacter pylori gamma-glutamyl transpeptidase inhibits autophagy in gastric cancer cells favouring survival of internalised bacteria
title_sort Helicobacter pylori gamma-glutamyl transpeptidase inhibits autophagy in gastric cancer cells favouring survival of internalised bacteria
dc.creator.none.fl_str_mv Bravo, J
Corvalan, A H
Quest, A F G
author Bravo, J
author_facet Bravo, J
Corvalan, A H
Quest, A F G
author_role author
author2 Corvalan, A H
Quest, A F G
author2_role author
author
description Helicobacter pylori (H. pylori) is the major risk factor associated with the development of gastric cancer. Multiple virulence factors contribute to its pathogenesis, and recently, the secreted gamma­glutamyl transpeptidase (GGT) has become of increasing
publishDate 2015
dc.date.annoconcurso.none.fl_str_mv 2015
dc.date.accessioned.none.fl_str_mv 2018-12-07T13:33:44Z
2022-07-07T21:40:14Z
dc.date.available.none.fl_str_mv 2018-12-07T13:33:44Z
2022-07-07T21:40:14Z
dc.date.issued.none.fl_str_mv 2018
dc.type.none.fl_str_mv Articulo
dc.type.driver.none.fl_str_mv info:eu-repo/semantics/article
dc.type.openaire.none.fl_str_mv info:eu-repo/semantics/publishedVersion
format article
status_str publishedVersion
dc.identifier.folio.none.fl_str_mv 1151411
1151411
dc.identifier.idwos.none.fl_str_mv WOS:000437674104015
dc.identifier.uri.none.fl_str_mv https://hdl.handle.net/10533/232701
identifier_str_mv 1151411
WOS:000437674104015
url https://hdl.handle.net/10533/232701
dc.language.iso.none.fl_str_mv eng
language eng
dc.relation.contest.none.fl_str_mv handle/10533/111557
dc.relation.instrument.none.fl_str_mv handle/10533/111541
dc.relation.program.none.fl_str_mv handle/10533/108045
dc.relation.uri.none.fl_str_mv https://2018.febscongress.org/abstract_preview.aspx?idAbstractEnc=4424170094091092097099424170
dc.rights.none.fl_str_mv Atribución-NoComercial-SinDerivadas 3.0 Chile
http://creativecommons.org/licenses/by-nc-nd/3.0/cl/
dc.rights.driver.none.fl_str_mv info:eu-repo/semantics/openAccess
rights_invalid_str_mv Atribución-NoComercial-SinDerivadas 3.0 Chile
http://creativecommons.org/licenses/by-nc-nd/3.0/cl/
eu_rights_str_mv openAccess
repository.name.fl_str_mv Repositorio ANID
repository.mail.fl_str_mv aletelier@anid.cl
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