Immunization of Balb/c mice with modified auto-antigens for induction of autoimmune sialoadenitis

Sjögren's syndrome is an autoimmune disease characterized by sialoadenitis and elevated titers of autoantibodies. To assess whether it is possible to induce inflammatory changes in salivary gland tissues, a series of immunizations in Balb/c mice have been undertaken, using salivary gland extrac...

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Detalles Bibliográficos
Autores: Unterkircher, Carmelinda Schmidt [UNESP], Leão, Mariella Vieira Pereira, Carvalho, Yasmin Rodarte [UNESP]
Tipo de recurso: artículo
Estado:Versión publicada
Fecha de publicación:2007
País:Brasil
Institución:Universidade Estadual Paulista (UNESP)
Repositorio:Repositório Institucional da UNESP
Idioma:inglés
OAI Identifier:oai:repositorio.unesp.br:11449/70065
Acceso en línea:http://dx.doi.org/10.1590/S0103-64402007000100009
http://hdl.handle.net/11449/70065
Access Level:acceso abierto
Palabra clave:Autoimmune disease
Carbonic anhydrase
Salivary gland
Sialoadenitis
autoantigen
mitogenic agent
periodate
periodic acid
animal
Bagg albino mouse
cattle
female
hybridoma
immunology
mouse
pathology
salivary gland
sialoadenitis
Sjoegren syndrome
vaccination
Animals
Autoantigens
Cattle
Female
Hybridomas
Mice
Mice, Inbred BALB C
Mitogens
Periodic Acid
Salivary Glands
Sialadenitis
Sjogren's Syndrome
Vaccination
Descripción
Sumario:Sjögren's syndrome is an autoimmune disease characterized by sialoadenitis and elevated titers of autoantibodies. To assess whether it is possible to induce inflammatory changes in salivary gland tissues, a series of immunizations in Balb/c mice have been undertaken, using salivary gland extract, modified or not, added to several adjuvants. Mice's humoral immune response to salivary gland antigens was monitored by ELISA. Inflammatory cells infiltrating gland tissue were seen 3 months after immunization with salivary gland extract modified with pepsin (AgGp) and metaperiodate (AgGMp). Although pathological progression was not observed, the histopathological picture was similar to the initial phase of Sjögren's syndrome. In addition, a monoclonal antibody reactive with 3 gland polypeptides and anhydrase carbonic II was rescued among B cells from immunized mice. Thus, immunizations with modified autoantigens were able to initiate pathological damage to glandular tissue and stimulate the proliferation of auto-reactive B cells.