Gut microbiota DPP4-like enzymes are increased in type-2 diabetes and contribute to incretin inactivation.

The gut microbiota controls broad aspects of human metabolism and feeding behavior, but the basis for this control remains largely unclear. Given the key role of human dipeptidyl peptidase 4 (DPP4) in host metabolism, we investigate whether microbiota DPP4-like counterparts perform the same function...

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Autores: Olivares M, Hernández-Calderón P, Cárdenas-Brito S, Liébana-García R, Sanz Y, Benítez-Páez A
Tipo de recurso: artículo
Estado:Versión publicada
Fecha de publicación:2024
País:España
Institución:Centro de Investigación Principe Felipe (CIPF)
Repositorio:r-CIPF. Repositorio Institucional Producción Científica del Centro de Investigación Principe Felipe (CIPF)
OAI Identifier:oai:cipf.fundanetsuite.com:p4377
Acceso en línea:https://cipf.fundanetsuite.com/Publicaciones/ProdCientif/PublicacionFrw.aspx?id=4377
Access Level:acceso abierto
Palabra clave:DPP4 enzyme, GLP-1, Gliptins, Glucose metabolism, Gut microbiome, Incretins, Obesity, Type 2 diabetes
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spelling Gut microbiota DPP4-like enzymes are increased in type-2 diabetes and contribute to incretin inactivation.Olivares MHernández-Calderón PCárdenas-Brito SLiébana-García RSanz YBenítez-Páez ADPP4 enzyme, GLP-1, Gliptins, Glucose metabolism, Gut microbiome, Incretins, Obesity, Type 2 diabetesThe gut microbiota controls broad aspects of human metabolism and feeding behavior, but the basis for this control remains largely unclear. Given the key role of human dipeptidyl peptidase 4 (DPP4) in host metabolism, we investigate whether microbiota DPP4-like counterparts perform the same function.BMC2024info:eu-repo/semantics/articleinfo:eu-repo/semantics/publishedVersionhttps://cipf.fundanetsuite.com/Publicaciones/ProdCientif/PublicacionFrw.aspx?id=4377GENOME BIOLOGYISSN: 1474760XISSNl: 14747596reponame:r-CIPF. Repositorio Institucional Producción Científica del Centro de Investigación Principe Felipe (CIPF)instname:Centro de Investigación Principe Felipe (CIPF)Inglésinfo:eu-repo/semantics/openAccessoai:cipf.fundanetsuite.com:p43772026-06-17T11:19:47Z
dc.title.none.fl_str_mv Gut microbiota DPP4-like enzymes are increased in type-2 diabetes and contribute to incretin inactivation.
title Gut microbiota DPP4-like enzymes are increased in type-2 diabetes and contribute to incretin inactivation.
spellingShingle Gut microbiota DPP4-like enzymes are increased in type-2 diabetes and contribute to incretin inactivation.
Olivares M
DPP4 enzyme, GLP-1, Gliptins, Glucose metabolism, Gut microbiome, Incretins, Obesity, Type 2 diabetes
title_short Gut microbiota DPP4-like enzymes are increased in type-2 diabetes and contribute to incretin inactivation.
title_full Gut microbiota DPP4-like enzymes are increased in type-2 diabetes and contribute to incretin inactivation.
title_fullStr Gut microbiota DPP4-like enzymes are increased in type-2 diabetes and contribute to incretin inactivation.
title_full_unstemmed Gut microbiota DPP4-like enzymes are increased in type-2 diabetes and contribute to incretin inactivation.
title_sort Gut microbiota DPP4-like enzymes are increased in type-2 diabetes and contribute to incretin inactivation.
dc.creator.none.fl_str_mv Olivares M
Hernández-Calderón P
Cárdenas-Brito S
Liébana-García R
Sanz Y
Benítez-Páez A
author Olivares M
author_facet Olivares M
Hernández-Calderón P
Cárdenas-Brito S
Liébana-García R
Sanz Y
Benítez-Páez A
author_role author
author2 Hernández-Calderón P
Cárdenas-Brito S
Liébana-García R
Sanz Y
Benítez-Páez A
author2_role author
author
author
author
author
dc.subject.none.fl_str_mv DPP4 enzyme, GLP-1, Gliptins, Glucose metabolism, Gut microbiome, Incretins, Obesity, Type 2 diabetes
topic DPP4 enzyme, GLP-1, Gliptins, Glucose metabolism, Gut microbiome, Incretins, Obesity, Type 2 diabetes
description The gut microbiota controls broad aspects of human metabolism and feeding behavior, but the basis for this control remains largely unclear. Given the key role of human dipeptidyl peptidase 4 (DPP4) in host metabolism, we investigate whether microbiota DPP4-like counterparts perform the same function.
publishDate 2024
dc.date.none.fl_str_mv 2024
dc.type.none.fl_str_mv info:eu-repo/semantics/article
info:eu-repo/semantics/publishedVersion
format article
status_str publishedVersion
dc.identifier.none.fl_str_mv https://cipf.fundanetsuite.com/Publicaciones/ProdCientif/PublicacionFrw.aspx?id=4377
url https://cipf.fundanetsuite.com/Publicaciones/ProdCientif/PublicacionFrw.aspx?id=4377
dc.language.none.fl_str_mv Inglés
language_invalid_str_mv Inglés
dc.rights.none.fl_str_mv info:eu-repo/semantics/openAccess
eu_rights_str_mv openAccess
dc.publisher.none.fl_str_mv BMC
publisher.none.fl_str_mv BMC
dc.source.none.fl_str_mv GENOME BIOLOGY
ISSN: 1474760X
ISSNl: 14747596
reponame:r-CIPF. Repositorio Institucional Producción Científica del Centro de Investigación Principe Felipe (CIPF)
instname:Centro de Investigación Principe Felipe (CIPF)
instname_str Centro de Investigación Principe Felipe (CIPF)
reponame_str r-CIPF. Repositorio Institucional Producción Científica del Centro de Investigación Principe Felipe (CIPF)
collection r-CIPF. Repositorio Institucional Producción Científica del Centro de Investigación Principe Felipe (CIPF)
repository.name.fl_str_mv
repository.mail.fl_str_mv
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score 15,812429

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