Numb-like (NumbL) downregulation increases tumorigenicity, cancer stem cell-like properties and resistance to chemotherapy

NumbL, or Numb-like, is a close homologue of Numb, and is part of an evolutionary conserved protein family implicated in some important cellular processes. Numb is a protein involved in cell development, in cell adhesion and migration, in asymmetric cell division, and in targeting proteins for endoc...

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Detalles Bibliográficos
Autores: García-Heredia, José M, Verdugo Sivianes, Eva M, Lucena-Cacace, Antonio, Molina-Pinelo, Sonia, Carnero, Amancio
Tipo de recurso: artículo
Fecha de publicación:2016
País:España
Institución:Instituto de Salud Carlos III (ISCIII)
Repositorio:Repisalud
Idioma:inglés
OAI Identifier:oai:repisalud.isciii.es:20.500.12105/25214
Acceso en línea:https://hdl.handle.net/20.500.12105/25214
Access Level:acceso abierto
Palabra clave:Notch
NumbL
Cancer stem cells
Tumor suppressor
Tumorigenicity
Active Transport, Cell Nucleus
Animals
Antineoplastic Agents
Breast Neoplasms
Carcinogenesis
Cell Adhesion
Cell Line, Tumor
Cell Movement
Colorectal Neoplasms
Dose-Response Relationship, Drug
Down-Regulation
Drug Resistance, Neoplasm
Endocytosis
Epithelial-Mesenchymal Transition
Female
Gene Expression Regulation, Neoplastic
HeLa Cells
Humans
Intracellular Signaling Peptides and Proteins
Lung Neoplasms
Mice
Neoplastic Stem Cells
Receptors, Notch
Sarcoma
Descripción
Sumario:NumbL, or Numb-like, is a close homologue of Numb, and is part of an evolutionary conserved protein family implicated in some important cellular processes. Numb is a protein involved in cell development, in cell adhesion and migration, in asymmetric cell division, and in targeting proteins for endocytosis and ubiquitination. NumbL exhibits some overlapping functions with Numb, but its role in tumorigenesis is not fully known. Here we showed that the downregulation of NumbL alone is sufficient to increase NICD nuclear translocation and induce Notch pathway activation. Furthermore, NumbL downregulation increases epithelial-mesenchymal transition (EMT) and cancer stem cell (CSC)-related gene transcripts and CSC-like phenotypes, including an increase in the CSC-like pool. These data suggest that NumbL can act independently as a tumor suppressor gene. Furthermore, an absence of NumbL induces chemoresistance in tumor cells. An analysis of human tumors indicates that NumbL is downregulated in a variable percentage of human tumors, with lower levels of this gene correlated with worse prognosis in colon, breast and lung tumors. Therefore, NumbL can act as an independent tumor suppressor inhibiting the Notch pathway and regulating the cancer stem cell pool.