The ventromedial hypothalamic nucleus responds differentially to genistein exposure during development in male and female rats in the long term

Genistein may act as an endocrine disruptor due to its structural similarity to estradiol. Exposure to genistein during development has been shown to alter neuromorphology and G protein-coupled estrogen receptor (GPER) expression in the arcuate nucleus in adult rats. In this study, we investigated t...

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Detalles Bibliográficos
Autores: Primo Chulvi, Ulises, Fernández García, José Manuel, Collado Guirao, Paloma, Pinos Sánchez, María Elena, Carrillo Urbano, Beatriz
Tipo de recurso: artículo
Fecha de publicación:2026
País:España
Institución:Universidad Nacional de Educación a Distancia
Repositorio:e-spacio. Repositorio Institucional de la UNED
Idioma:inglés
OAI Identifier:oai:dnet:espacio_____::51f91b134f955e80cf7007a663842458
Acceso en línea:https://hdl.handle.net/20.500.14468/32524
Access Level:acceso abierto
Palabra clave:2410 Biología humana
6106 Psicología experimental
Genistein
Ventromedial hypothalamic nucleus
Number of neurons
GPER
Sex differences
Descripción
Sumario:Genistein may act as an endocrine disruptor due to its structural similarity to estradiol. Exposure to genistein during development has been shown to alter neuromorphology and G protein-coupled estrogen receptor (GPER) expression in the arcuate nucleus in adult rats. In this study, we investigated the long-term effects of genistein exposure in the ventromedial nucleus of the hypothalamus (VMH) during the second week of life. Rats of both sexes were injected daily (s.c.) from postnatal day 6–13, with the following treatments: I) corn oil vehicle (control group); II) 10 μg/g of genistein (G10); and III) 50 μg/g of genistein (G50). Morphometric analyses of the VMH in Nissl-stained and GPER-immunostained brain sections were performed at P90 using ImageJ software. In males, the low dose of genistein provoked a significant increase of GPER-ir neurons in the dorsomedial, central and ventrolateral subdivisions and in the medial division, as well as in the total VMH, while in females no effects in GPER-ir neurons were observed. In females, the high dose of genistein produced a significant decrease of Nissl-stained neurons in the ventrolateral subdivision and medial division, but no effect was detected in males. Early exposure to genistein may differentially alter the VMH in the long-term depending on the sex, dose, and subdivision studied.