Endogenous retinoid X receptor ligands in mouse hematopoietic cells.

The retinoid X receptor α (RXRA) has been implicated in diverse hematological processes. To identify natural ligands of RXRA that are present in hematopoietic cells, we adapted an upstream activation sequence-green fluorescent protein (UAS-GFP) reporter mouse to detect natural RXRA ligands in vivo....

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Detalles Bibliográficos
Autores: Niu, Haixia, Fujiwara, Hideji, di Martino, Orsola, Hadwiger, Gayla, Frederick, Thomas E, Menéndez-Gutiérrez, María P, Ricote, Mercedes, Bowman, Gregory R, Welch, John S
Tipo de recurso: artículo
Fecha de publicación:2017
País:España
Institución:Instituto de Salud Carlos III (ISCIII)
Repositorio:Repisalud
Idioma:inglés
OAI Identifier:oai:repisalud.isciii.es:20.500.12105/15140
Acceso en línea:http://hdl.handle.net/20.500.12105/15140
Access Level:acceso abierto
Palabra clave:Animals
Fatty Acids
Granulocyte Colony-Stimulating Factor
Granulocytes
Green Fluorescent Proteins
HEK293 Cells
Hematopoietic Stem Cells
Humans
Leukopoiesis
Ligands
Mice
Mice, Knockout
Mice, Mutant Strains
Myeloid Cells
Retinoid X Receptor alpha
Vitamin A
Descripción
Sumario:The retinoid X receptor α (RXRA) has been implicated in diverse hematological processes. To identify natural ligands of RXRA that are present in hematopoietic cells, we adapted an upstream activation sequence-green fluorescent protein (UAS-GFP) reporter mouse to detect natural RXRA ligands in vivo. We observed reporter activity in diverse types of hematopoietic cells in vivo. Reporter activity increased during granulocyte colony-stimulating factor (G-CSF)-induced granulopoiesis and after phenylhydrazine (PHZ)-induced anemia, suggesting the presence of dynamically regulated natural RXRA ligands in hematopoietic cells. Mouse plasma activated Gal4-UAS reporter cells in vitro, and plasma from mice treated with G-CSF or PHZ recapitulated the patterns of reporter activation that we observed in vivo. Plasma from mice with dietary vitamin A deficiency only mildly reduced RXRA reporter activity, whereas plasma from mice on a fatty acid restriction diet reduced reporter activity, implicating fatty acids as plasma RXRA ligands. Through differential extraction coupled with mass spectrometry, we identified the long-chain fatty acid C24:5 as a natural RXRA ligand that was greatly increased in abundance in response to hematopoietic stress. Together, these data suggest that natural RXRA ligands are present and dynamically increased in abundance in mouse hematopoietic cells in vivo.