Complete description of the three pathways of the complement system in a series of 430 patients with rheumatoid arthritis

The complement (C) system is implicated in the etiopathogenesis of rheumatoid arthritis (RA). However, there is a lack of studies characterizing all three C pathways in RA patients. This study aimed to evaluate the association between an in-depth examination of the C system and RA patient characteri...

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Autores: Rodríguez-González, Dara, Carcía-González, María, Gómez-Bernal, Fuensanta, Quevedo-Abeledo, Juan C., González-Rivero, Agustín F., Fernández-Cladera, Yolanda, González López, Elena, Ocejo-Vinyals, Javier Gonzalo, Jiménez-Sosa, Alejandro, González-Toledo, Beatriz, González-Gay Mantecón, Miguel Ángel
Tipo de documento: artigo
Data de publicação:2024
País:España
Recursos:Universidad de Cantabria (UC)
Repositório:UCrea Repositorio Abierto de la Universidad de Cantabria
Idioma:inglês
OAI Identifier:oai:repositorio.unican.es:10902/34462
Acesso em linha:https://hdl.handle.net/10902/34462
Access Level:Acceso aberto
Palavra-chave:Rheumatoid arthritis
Complement system
Complement pathways
Complement activity assays
Disease activity
Rheumatoid factor
Anti-citrullinated protein autoantibodies
Inflammation
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spelling Complete description of the three pathways of the complement system in a series of 430 patients with rheumatoid arthritisRodríguez-González, DaraCarcía-González, MaríaGómez-Bernal, FuensantaQuevedo-Abeledo, Juan C.González-Rivero, Agustín F.Fernández-Cladera, YolandaGonzález López, ElenaOcejo-Vinyals, Javier GonzaloJiménez-Sosa, AlejandroGonzález-Toledo, BeatrizGonzález-Gay Mantecón, Miguel ÁngelRheumatoid arthritisComplement systemComplement pathwaysComplement activity assaysDisease activityRheumatoid factorAnti-citrullinated protein autoantibodiesInflammationThe complement (C) system is implicated in the etiopathogenesis of rheumatoid arthritis (RA). However, there is a lack of studies characterizing all three C pathways in RA patients. This study aimed to evaluate the association between an in-depth examination of the C system and RA patient characteristics, focusing on disease activity and the presence of rheumatoid factor and anti-citrullinated protein autoantibodies (ACPA). In a cohort of 430 RA patients, functional assays of the three C pathways (classical, alternative, and lectin) and serum levels of their components were assessed. Components included C1q (classical); factor D and properdin (alternative); lectin (lectin); C1-inhibitor; C2, C4, and C4b (classical and lectin); C3, C3a, and C4b (common); and C5, C5a, and C9 (terminal). A multivariable linear regression analysis showed significant positive correlations between C-reactive protein and C system proteins and functional assays, especially in the terminal and common pathways. Disease activity, measured by scores with or without acute phase reactants, positively correlated with the classical pathway functional test and terminal pathway products. Conversely, rheumatoid factor or ACPA presence was associated with lower classical pathway values and decreased C3a and C4b levels, suggesting complement depletion. In conclusion, RA disease activity increases C molecules and functional complement assays, while rheumatoid factor or ACPA positivity is linked to C consumption. Our study offers a detailed analysis of the complement system?s role in RA, potentially guiding the development of more targeted and effective treatment strategies.Funding: This study was funded by a grant to IF-A by Instituto de Salud Carlos III (ISCIII) through the project PI20/00084 and co-funded by the European Union. Acknowledgments: Iván Ferraz-Amaro would like to acknowledge that he has received grants/research supports from Abbott, MSD, Janssen, and Roche, as well as consultation fees from company-sponsored speakers’ bureaus associated with Abbott, Pfizer, Roche, Sanofi, Celgene, and M.A. González-Gay received consultation fees/participation from a company-sponsored speakers’ bureau from GSK.MDPIUniversidad de Cantabria20242024-01-01journal articlehttp://purl.org/coar/resource_type/c_6501NAhttp://purl.org/coar/version/c_be7fb7dd8ff6fe43info:eu-repo/semantics/articlehttps://hdl.handle.net/10902/34462International Journal of Molecular Sciences, 2024, 25, 8360reponame:UCrea Repositorio Abierto de la Universidad de Cantabriainstname:Universidad de Cantabria (UC)Inglésengopen accesshttp://purl.org/coar/access_right/c_abf2Attribution 4.0 Internationalhttp://creativecommons.org/licenses/by/4.0/info:eu-repo/semantics/openAccessoai:repositorio.unican.es:10902/344622026-06-02T12:39:31Z
dc.title.none.fl_str_mv Complete description of the three pathways of the complement system in a series of 430 patients with rheumatoid arthritis
title Complete description of the three pathways of the complement system in a series of 430 patients with rheumatoid arthritis
spellingShingle Complete description of the three pathways of the complement system in a series of 430 patients with rheumatoid arthritis
Rodríguez-González, Dara
Rheumatoid arthritis
Complement system
Complement pathways
Complement activity assays
Disease activity
Rheumatoid factor
Anti-citrullinated protein autoantibodies
Inflammation
title_short Complete description of the three pathways of the complement system in a series of 430 patients with rheumatoid arthritis
title_full Complete description of the three pathways of the complement system in a series of 430 patients with rheumatoid arthritis
title_fullStr Complete description of the three pathways of the complement system in a series of 430 patients with rheumatoid arthritis
title_full_unstemmed Complete description of the three pathways of the complement system in a series of 430 patients with rheumatoid arthritis
title_sort Complete description of the three pathways of the complement system in a series of 430 patients with rheumatoid arthritis
dc.creator.none.fl_str_mv Rodríguez-González, Dara
Carcía-González, María
Gómez-Bernal, Fuensanta
Quevedo-Abeledo, Juan C.
González-Rivero, Agustín F.
Fernández-Cladera, Yolanda
González López, Elena
Ocejo-Vinyals, Javier Gonzalo
Jiménez-Sosa, Alejandro
González-Toledo, Beatriz
González-Gay Mantecón, Miguel Ángel
author Rodríguez-González, Dara
author_facet Rodríguez-González, Dara
Carcía-González, María
Gómez-Bernal, Fuensanta
Quevedo-Abeledo, Juan C.
González-Rivero, Agustín F.
Fernández-Cladera, Yolanda
González López, Elena
Ocejo-Vinyals, Javier Gonzalo
Jiménez-Sosa, Alejandro
González-Toledo, Beatriz
González-Gay Mantecón, Miguel Ángel
author_role author
author2 Carcía-González, María
Gómez-Bernal, Fuensanta
Quevedo-Abeledo, Juan C.
González-Rivero, Agustín F.
Fernández-Cladera, Yolanda
González López, Elena
Ocejo-Vinyals, Javier Gonzalo
Jiménez-Sosa, Alejandro
González-Toledo, Beatriz
González-Gay Mantecón, Miguel Ángel
author2_role author
author
author
author
author
author
author
author
author
author
dc.contributor.none.fl_str_mv Universidad de Cantabria
dc.subject.none.fl_str_mv Rheumatoid arthritis
Complement system
Complement pathways
Complement activity assays
Disease activity
Rheumatoid factor
Anti-citrullinated protein autoantibodies
Inflammation
topic Rheumatoid arthritis
Complement system
Complement pathways
Complement activity assays
Disease activity
Rheumatoid factor
Anti-citrullinated protein autoantibodies
Inflammation
description The complement (C) system is implicated in the etiopathogenesis of rheumatoid arthritis (RA). However, there is a lack of studies characterizing all three C pathways in RA patients. This study aimed to evaluate the association between an in-depth examination of the C system and RA patient characteristics, focusing on disease activity and the presence of rheumatoid factor and anti-citrullinated protein autoantibodies (ACPA). In a cohort of 430 RA patients, functional assays of the three C pathways (classical, alternative, and lectin) and serum levels of their components were assessed. Components included C1q (classical); factor D and properdin (alternative); lectin (lectin); C1-inhibitor; C2, C4, and C4b (classical and lectin); C3, C3a, and C4b (common); and C5, C5a, and C9 (terminal). A multivariable linear regression analysis showed significant positive correlations between C-reactive protein and C system proteins and functional assays, especially in the terminal and common pathways. Disease activity, measured by scores with or without acute phase reactants, positively correlated with the classical pathway functional test and terminal pathway products. Conversely, rheumatoid factor or ACPA presence was associated with lower classical pathway values and decreased C3a and C4b levels, suggesting complement depletion. In conclusion, RA disease activity increases C molecules and functional complement assays, while rheumatoid factor or ACPA positivity is linked to C consumption. Our study offers a detailed analysis of the complement system?s role in RA, potentially guiding the development of more targeted and effective treatment strategies.
publishDate 2024
dc.date.none.fl_str_mv 2024
2024-01-01
dc.type.none.fl_str_mv journal article
http://purl.org/coar/resource_type/c_6501
NA
http://purl.org/coar/version/c_be7fb7dd8ff6fe43
dc.type.openaire.fl_str_mv info:eu-repo/semantics/article
format article
dc.identifier.none.fl_str_mv https://hdl.handle.net/10902/34462
url https://hdl.handle.net/10902/34462
dc.language.none.fl_str_mv Inglés
eng
language_invalid_str_mv Inglés
language eng
dc.rights.none.fl_str_mv open access
http://purl.org/coar/access_right/c_abf2
Attribution 4.0 International
http://creativecommons.org/licenses/by/4.0/
dc.rights.openaire.fl_str_mv info:eu-repo/semantics/openAccess
rights_invalid_str_mv open access
http://purl.org/coar/access_right/c_abf2
Attribution 4.0 International
http://creativecommons.org/licenses/by/4.0/
eu_rights_str_mv openAccess
dc.publisher.none.fl_str_mv MDPI
publisher.none.fl_str_mv MDPI
dc.source.none.fl_str_mv International Journal of Molecular Sciences, 2024, 25, 8360
reponame:UCrea Repositorio Abierto de la Universidad de Cantabria
instname:Universidad de Cantabria (UC)
instname_str Universidad de Cantabria (UC)
reponame_str UCrea Repositorio Abierto de la Universidad de Cantabria
collection UCrea Repositorio Abierto de la Universidad de Cantabria
repository.name.fl_str_mv
repository.mail.fl_str_mv
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