Distinct roles for PARP-1 and PARP-2 in c-Myc-driven B-cell lymphoma in mice

Dysregulation of the c-Myc oncogene occurs in a wide variety of hematologic malignancies, and its overexpression has been linked with aggressive tumor progression. Here, we show that poly (ADP-ribose) polymerase 1 (PARP-1) and PARP-2 exert opposing influences on progression of c-Myc-driven B-cell ly...

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Authors: Galindo-Campos, M. A., Lutfi, Nuria, Bonnin, S., Martínez, C., Velasco-Hernández, Talia, García-Hernández, V., Martín Caballero, Juan, Ampurdanés, Coral, Gimeno, R., Colomo, L., Roué, Gaël, Guilbaud, G., Dantzer, Françoise, Navarro Medrano, Pilar, Murga, Matilde, Fernández-Capetillo, Óscar, Bigas, A., Menéndez, Pablo, Sale; J. E.
Format: article
Status:Versión aceptada para publicación
Publication Date:2022
Country:España
Institution:Consejo Superior de Investigaciones Científicas (CSIC)
Repository:DIGITAL.CSIC. Repositorio Institucional del CSIC
OAI Identifier:oai:digital.csic.es:10261/278133
Online Access:http://hdl.handle.net/10261/278133
Access Level:Open access
Keyword:Animals
Carcinogenesis / genetics
DNA Damage
Gene Deletion
Gene expression regulation
Neoplastic Lymphoma
B-Cell / genetics
Mice
Mice, Knockout
Poly (ADP-Ribose) Polymerase-1 / genetics
Poly(ADP-ribose) Polymerases / genetics
Proto-Oncogene Proteins c-myc / genetics
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oai_identifier_str oai:digital.csic.es:10261/278133
network_acronym_str ES
network_name_str España
repository_id_str
dc.title.none.fl_str_mv Distinct roles for PARP-1 and PARP-2 in c-Myc-driven B-cell lymphoma in mice
title Distinct roles for PARP-1 and PARP-2 in c-Myc-driven B-cell lymphoma in mice
spellingShingle Distinct roles for PARP-1 and PARP-2 in c-Myc-driven B-cell lymphoma in mice
Galindo-Campos, M. A.
Animals
Carcinogenesis / genetics
DNA Damage
Gene Deletion
Gene expression regulation
Neoplastic Lymphoma
B-Cell / genetics
Mice
Mice, Knockout
Poly (ADP-Ribose) Polymerase-1 / genetics
Poly(ADP-ribose) Polymerases / genetics
Proto-Oncogene Proteins c-myc / genetics
title_short Distinct roles for PARP-1 and PARP-2 in c-Myc-driven B-cell lymphoma in mice
title_full Distinct roles for PARP-1 and PARP-2 in c-Myc-driven B-cell lymphoma in mice
title_fullStr Distinct roles for PARP-1 and PARP-2 in c-Myc-driven B-cell lymphoma in mice
title_full_unstemmed Distinct roles for PARP-1 and PARP-2 in c-Myc-driven B-cell lymphoma in mice
title_sort Distinct roles for PARP-1 and PARP-2 in c-Myc-driven B-cell lymphoma in mice
dc.creator.none.fl_str_mv Galindo-Campos, M. A.
Lutfi, Nuria
Bonnin, S.
Martínez, C.
Velasco-Hernández, Talia
García-Hernández, V.
Martín Caballero, Juan
Ampurdanés, Coral
Gimeno, R.
Colomo, L.
Roué, Gaël
Guilbaud, G.
Dantzer, Françoise
Navarro Medrano, Pilar
Murga, Matilde
Fernández-Capetillo, Óscar
Bigas, A.
Menéndez, Pablo
Sale; J. E.
author Galindo-Campos, M. A.
author_facet Galindo-Campos, M. A.
Lutfi, Nuria
Bonnin, S.
Martínez, C.
Velasco-Hernández, Talia
García-Hernández, V.
Martín Caballero, Juan
Ampurdanés, Coral
Gimeno, R.
Colomo, L.
Roué, Gaël
Guilbaud, G.
Dantzer, Françoise
Navarro Medrano, Pilar
Murga, Matilde
Fernández-Capetillo, Óscar
Bigas, A.
Menéndez, Pablo
Sale; J. E.
author_role author
author2 Lutfi, Nuria
Bonnin, S.
Martínez, C.
Velasco-Hernández, Talia
García-Hernández, V.
Martín Caballero, Juan
Ampurdanés, Coral
Gimeno, R.
Colomo, L.
Roué, Gaël
Guilbaud, G.
Dantzer, Françoise
Navarro Medrano, Pilar
Murga, Matilde
Fernández-Capetillo, Óscar
Bigas, A.
Menéndez, Pablo
Sale; J. E.
author2_role author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
dc.contributor.none.fl_str_mv Ministerio de Industria y Competitividad (España)
Ministerio de Ciencia e Innovación (España)
Fundación Científica Asociación Española Contra el Cáncer
Medical Research Council (UK)
Laboratory of Excellence of Biology for Psychiatry (France)
Université de Strasbourg
Centre National de la Recherche Scientifique (France)
Instituto de Salud Carlos III
European Commission
Generalitat de Catalunya
Centres de Recerca de Catalunya
Josep Carreras Leukemia Foundation
Fundación Unoentrecienmil
Marie Curie International Incoming Fellowship
Consejo Superior de Investigaciones Científicas [https://ror.org/02gfc7t72]
dc.subject.none.fl_str_mv Animals
Carcinogenesis / genetics
DNA Damage
Gene Deletion
Gene expression regulation
Neoplastic Lymphoma
B-Cell / genetics
Mice
Mice, Knockout
Poly (ADP-Ribose) Polymerase-1 / genetics
Poly(ADP-ribose) Polymerases / genetics
Proto-Oncogene Proteins c-myc / genetics
topic Animals
Carcinogenesis / genetics
DNA Damage
Gene Deletion
Gene expression regulation
Neoplastic Lymphoma
B-Cell / genetics
Mice
Mice, Knockout
Poly (ADP-Ribose) Polymerase-1 / genetics
Poly(ADP-ribose) Polymerases / genetics
Proto-Oncogene Proteins c-myc / genetics
description Dysregulation of the c-Myc oncogene occurs in a wide variety of hematologic malignancies, and its overexpression has been linked with aggressive tumor progression. Here, we show that poly (ADP-ribose) polymerase 1 (PARP-1) and PARP-2 exert opposing influences on progression of c-Myc-driven B-cell lymphoma. PARP-1 and PARP-2 catalyze the synthesis and transfer of ADP-ribose units onto amino acid residues of acceptor proteins in response to DNA strand breaks, playing a central role in the response to DNA damage. Accordingly, PARP inhibitors have emerged as promising new cancer therapeutics. However, the inhibitors currently available for clinical use are not able to discriminate between individual PARP proteins. We found that genetic deletion of PARP-2 prevents c-Myc-driven B-cell lymphoma, whereas PARP-1 deficiency accelerates lymphomagenesis in the E¿-Myc mouse model of aggressive B-cell lymphoma. Loss of PARP-2 aggravates replication stress in preleukemic E¿-Myc B cells, resulting in accumulation of DNA damage and concomitant cell death that restricts the c-Myc-driven expansion of B cells, thereby providing protection against B-cell lymphoma. In contrast, PARP-1 deficiency induces a proinflammatory response and an increase in regulatory T cells, likely contributing to immune escape of B-cell lymphoma, resulting in an acceleration of lymphomagenesis. These findings pinpoint specific functions for PARP-1 and PARP-2 in c-Myc-driven lymphomagenesis with antagonistic consequences that may help inform the design of new PARP-centered therapeutic strategies, with selective PARP-2 inhibition potentially representing a new therapeutic approach for the treatment of c-Myc-driven tumors.
publishDate 2022
dc.date.none.fl_str_mv 2022
2022
2022
2022
dc.type.none.fl_str_mv info:eu-repo/semantics/article
http://purl.org/coar/resource_type/c_6501
Postprint
info:eu-repo/semantics/acceptedVersion
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status_str acceptedVersion
dc.identifier.none.fl_str_mv http://hdl.handle.net/10261/278133
url http://hdl.handle.net/10261/278133
dc.language.none.fl_str_mv Inglés
language_invalid_str_mv Inglés
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info:eu-repo/grantAgreement/AEI/Plan Estatal de Investigación Científica y Técnica y de Innovación 2013-2016/SAF2017-83565-R
info:eu-repo/grantAgreement/AEI/Plan Estatal de Investigación Científica y Técnica y de Innovación 2017-2020/PID2020-112526RB-I00
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info:eu-repo/grantAgreement/AEI/Plan Estatal de Investigación Científica y Técnica y de Innovación 2017-2020/PID2019-104695RB-I00
Galindo-Campos, M. A.; Lutfi, Nuria; Bonnin, Sarah; Martínez, Carlos; Velasco-Hernández, Talia; García-Hernández, V.; Martín Caballero, Juan; Ampurdanés, Coral; Gimeno, Ramón; Colomo, Lluis; Roué, Gaël; Guilbaud, G.; Dantzer, Françoise; Navarro Medrano, Pilar; Murga, Matilde; Fernández-Capetillo, Óscar; Bigas, Anna; Menéndez, Pablo; Sale; J. E.; 2022; Supplementary Data Distinct roles for PARP-1 and PARP-2 in c-Myc-driven B-cell lymphoma in mice [Dataset]; American Society of Hematology; http://dx.doi.org/10.1182/blood.2021012805
http://dx.doi.org/10.1182/blood.2021012805

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dc.publisher.none.fl_str_mv American Society of Hematology
publisher.none.fl_str_mv American Society of Hematology
dc.source.none.fl_str_mv reponame:DIGITAL.CSIC. Repositorio Institucional del CSIC
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spelling Distinct roles for PARP-1 and PARP-2 in c-Myc-driven B-cell lymphoma in miceGalindo-Campos, M. A.Lutfi, NuriaBonnin, S.Martínez, C.Velasco-Hernández, TaliaGarcía-Hernández, V.Martín Caballero, JuanAmpurdanés, CoralGimeno, R.Colomo, L.Roué, GaëlGuilbaud, G.Dantzer, FrançoiseNavarro Medrano, PilarMurga, MatildeFernández-Capetillo, ÓscarBigas, A.Menéndez, PabloSale; J. E.AnimalsCarcinogenesis / geneticsDNA DamageGene DeletionGene expression regulationNeoplastic LymphomaB-Cell / geneticsMiceMice, KnockoutPoly (ADP-Ribose) Polymerase-1 / geneticsPoly(ADP-ribose) Polymerases / geneticsProto-Oncogene Proteins c-myc / geneticsDysregulation of the c-Myc oncogene occurs in a wide variety of hematologic malignancies, and its overexpression has been linked with aggressive tumor progression. Here, we show that poly (ADP-ribose) polymerase 1 (PARP-1) and PARP-2 exert opposing influences on progression of c-Myc-driven B-cell lymphoma. PARP-1 and PARP-2 catalyze the synthesis and transfer of ADP-ribose units onto amino acid residues of acceptor proteins in response to DNA strand breaks, playing a central role in the response to DNA damage. Accordingly, PARP inhibitors have emerged as promising new cancer therapeutics. However, the inhibitors currently available for clinical use are not able to discriminate between individual PARP proteins. We found that genetic deletion of PARP-2 prevents c-Myc-driven B-cell lymphoma, whereas PARP-1 deficiency accelerates lymphomagenesis in the E¿-Myc mouse model of aggressive B-cell lymphoma. Loss of PARP-2 aggravates replication stress in preleukemic E¿-Myc B cells, resulting in accumulation of DNA damage and concomitant cell death that restricts the c-Myc-driven expansion of B cells, thereby providing protection against B-cell lymphoma. In contrast, PARP-1 deficiency induces a proinflammatory response and an increase in regulatory T cells, likely contributing to immune escape of B-cell lymphoma, resulting in an acceleration of lymphomagenesis. These findings pinpoint specific functions for PARP-1 and PARP-2 in c-Myc-driven lymphomagenesis with antagonistic consequences that may help inform the design of new PARP-centered therapeutic strategies, with selective PARP-2 inhibition potentially representing a new therapeutic approach for the treatment of c-Myc-driven tumors.The J.Y. laboratory is funded by the Spanish Ministerio de Economía, Industria y Competitividad (grant SAF2017-83565-R), Spanish Ministerio de Ciencia e Innovación (grant PID2020-112526RB-I00), and Fundación Científica de la Asociación Española Contra el Cáncer (grant PROYEI6018YÉLA). Work in the J.E.S. laboratory is supported by a core grant to the Laboratory of Molecular Biology from the Medical Research Council (U105178808). The F.D. laboratory is supported by a Laboratory of Excellence grant (ANR-10-LABX-0034_Medalis) to Strasbourg University, Centre National de la Recherche Scientifique. The P.N. laboratory is supported by grants from the Spanish Ministry of Economy and Competitiveness/Instituto de Salud Carlos III–Fondo Europeo de Desarrollo Regional (FEDER; PI17/00199 and PI20/00625) and the Generalitat de Catalunya (2017-SGR-225). The P.M. laboratory acknowledges support from Centres de Recerca de Catalunya/Generalitat de Catalunya and Fundació Josep Carreras-Obra Social la Caixa for core support, the Spanish Ministry of Economy and Competitiveness (grant SAF-2019-108160-R), the Fundación Uno entre Cienmil, the Obra Social La Caixa (grant LCF/PR/HR19/52160011), and the German Josep Carreras Leukamie Stiftung. Work at the G.R. and P.M. laboratories are cofinanced by the European Regional Development Fund through the Interreg V-A Spain-France-Andorra Program (project PROTEOblood; grant EFA360/19). The O.F.-C. laboratory is funded by grants from the Spanish Ministry of Science, Innovation and Universities (RTI2018-102204-B-I00; cofinanced with European FEDER funds) and the European Research Council (ERC-617840). T.V.-H. was supported by a Marie Sklodowska Curie fellowship (GA792923). The A.B. laboratory is supported by the Spanish Ministry of Economy and Competitiveness (grant PID2019-104695RB-I00).Peer reviewedAmerican Society of HematologyMinisterio de Industria y Competitividad (España)Ministerio de Ciencia e Innovación (España)Fundación Científica Asociación Española Contra el CáncerMedical Research Council (UK)Laboratory of Excellence of Biology for Psychiatry (France)Université de StrasbourgCentre National de la Recherche Scientifique (France)Instituto de Salud Carlos IIIEuropean CommissionGeneralitat de CatalunyaCentres de Recerca de CatalunyaJosep Carreras Leukemia FoundationFundación UnoentrecienmilMarie Curie International Incoming FellowshipConsejo Superior de Investigaciones Científicas [https://ror.org/02gfc7t72]2022202220222022info:eu-repo/semantics/articlehttp://purl.org/coar/resource_type/c_6501Postprintinfo:eu-repo/semantics/acceptedVersionapplication/pdfhttp://hdl.handle.net/10261/278133reponame:DIGITAL.CSIC. Repositorio Institucional del CSICinstname:Consejo Superior de Investigaciones Científicas (CSIC)Inglés#PLACEHOLDER_PARENT_METADATA_VALUE##PLACEHOLDER_PARENT_METADATA_VALUE##PLACEHOLDER_PARENT_METADATA_VALUE##PLACEHOLDER_PARENT_METADATA_VALUE##PLACEHOLDER_PARENT_METADATA_VALUE##PLACEHOLDER_PARENT_METADATA_VALUE##PLACEHOLDER_PARENT_METADATA_VALUE#info:eu-repo/grantAgreement/AEI/Plan Estatal de Investigación Científica y Técnica y de Innovación 2013-2016/SAF2017-83565-Rinfo:eu-repo/grantAgreement/AEI/Plan Estatal de Investigación Científica y Técnica y de Innovación 2017-2020/PID2020-112526RB-I00info:eu-repo/grantAgreement/ISCIII/Plan Estatal de Investigación Científica y Técnica y de Innovación 2013-2016 (ISCIII)/PI17%2F00199info:eu-repo/grantAgreement/ISCIII/Plan Estatal de Investigación Científica y Técnica y de Innovación 2017-2020 (ISCIII)/PI20%2F00625info:eu-repo/grantAgreement/AEI/Plan Estatal de Investigación Científica y Técnica y de Innovación 2017-2020/PID2019-108160RB-I00info:eu-repo/grantAgreement/AEI/Plan Estatal de Investigación Científica y Técnica y de Innovación 2017-2020/RTI2018-102204-B-I00info:eu-repo/grantAgreement/AEI/Plan Estatal de Investigación Científica y Técnica y de Innovación 2017-2020/PID2019-104695RB-I00Galindo-Campos, M. A.; Lutfi, Nuria; Bonnin, Sarah; Martínez, Carlos; Velasco-Hernández, Talia; García-Hernández, V.; Martín Caballero, Juan; Ampurdanés, Coral; Gimeno, Ramón; Colomo, Lluis; Roué, Gaël; Guilbaud, G.; Dantzer, Françoise; Navarro Medrano, Pilar; Murga, Matilde; Fernández-Capetillo, Óscar; Bigas, Anna; Menéndez, Pablo; Sale; J. E.; 2022; Supplementary Data Distinct roles for PARP-1 and PARP-2 in c-Myc-driven B-cell lymphoma in mice [Dataset]; American Society of Hematology; http://dx.doi.org/10.1182/blood.2021012805http://dx.doi.org/10.1182/blood.2021012805Síinfo:eu-repo/semantics/openAccessoai:digital.csic.es:10261/2781332026-05-22T06:33:51Z
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