Investigation in the CB1 and CB2 receptor binding profile and intrinsic activity of ( ) and (+)-enantiomers of some naturally occurring phytocannabinoids or synthetic derivatives

Cannabidiol (CBD) and cannabidivarin (CBDV) have shown promising clinical efficacy for the management of epilepsy, and beneficial effects have been demonstrated for CBD in a diversity of other pathologies (pain, schizophrenia, Tourette syndrome, anxiety). However, the mechanism(s) involved are still...

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Autores: Rodríguez-Carreiro, Santiago, Gómez-Cañasa, María, Lubrinia, Francesca, Gonzalo-Consuegra, Claudia, Winklerd, Matthias, Caprioglio, Diego, Appendino, Giovanni, García, Concepción, Morales, Paula, Jagerovic, Nadine, Fischer, Joerg T., Fiebich, Bernd L., Goetz, Marcus R., Muñoz, Eduardo, Fernández-Ruiz, Javier
Tipo de recurso: artículo
Estado:Versión publicada
Fecha de publicación:2025
País:España
Institución:Consejo Superior de Investigaciones Científicas (CSIC)
Repositorio:DIGITAL.CSIC. Repositorio Institucional del CSIC
OAI Identifier:oai:digital.csic.es:10261/388571
Acceso en línea:http://hdl.handle.net/10261/388571
Access Level:acceso abierto
Palabra clave:CBD
CBDV
CBDA-Derivatives
( )- and (+)-enantiomers
CB1 receptor
CB2 receptor
Binding
Agonist
Inverse agonist
Antagonist
Anti-inflammatory and neuroprotective effects
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spelling Investigation in the CB1 and CB2 receptor binding profile and intrinsic activity of ( ) and (+)-enantiomers of some naturally occurring phytocannabinoids or synthetic derivativesRodríguez-Carreiro, SantiagoGómez-Cañasa, MaríaLubrinia, FrancescaGonzalo-Consuegra, ClaudiaWinklerd, MatthiasCaprioglio, DiegoAppendino, GiovanniGarcía, ConcepciónMorales, PaulaJagerovic, NadineFischer, Joerg T.Fiebich, Bernd L.Goetz, Marcus R.Muñoz, EduardoFernández-Ruiz, JavierCBDCBDVCBDA-Derivatives( )- and (+)-enantiomersCB1 receptorCB2 receptorBindingAgonistInverse agonistAntagonistAnti-inflammatory and neuroprotective effectsCannabidiol (CBD) and cannabidivarin (CBDV) have shown promising clinical efficacy for the management of epilepsy, and beneficial effects have been demonstrated for CBD in a diversity of other pathologies (pain, schizophrenia, Tourette syndrome, anxiety). However, the mechanism(s) involved are still largely elusive, as are the molecular target(s) involved. CBD and CBDV do not orthosterically bind the cannabinoid type-1 (CB1) and type-2 (CB2) receptors, showing only modest allosteric modulation of both end-points. CBD and CBDV are biosynthesized as optically highly pure ( )-enantiomers, and most bioactivity data refer to these forms. (+)-CBD and related analogues [(+)-cannabidiolic acid (CBDA), its esters, and (+)-CBDV] can be obtained by chemical synthesis, and we present evidence that the (+)- and ( )-enantiomers of CBD, CBDV and of a selection of derivatives of CBDA have distinct binding profiles and functional activity at the CB1/CB2 receptors. Thus, with the single exception of the methyl ester of CBDA, all the (+)-enantiomers showed higher affinities than the ( )-isomers for both receptors, in particular for the CB2 receptors. The affinity of the (+)-enantiomers for both CB1 and CB2 receptors showed a marked dependence on the nature of the alkyl residue on the aromatic ring and the esterification pattern of CBDA. Potency was rarely in the low nM value for CB1, but generally so for CB2. Enantiomers showing low nM activity were further investigated for their intrinsic activity using GTPγS binding assays. This proved that (+)-CBD, (+)-CBDV and the methyl ester of (+)-CBDA are agonists at the CB2 receptor, with the β-hydroxyethyl ester of (+)-CBDA being an inverse agonist, and its β-hydroxypentyl ester behaving as an agonist at CB1 and an inverse agonist at CB2. Finally, we assayed in vitro the anti-inflammatory and neuroprotective properties of three compounds [(+)-CBD, (+)-CBDV and (+)-CBDA methyl ester] strongly activating CB2, showing their ability to reduce the production of proinflammatory factors and protecting neurons against their toxicity. Remarkably, these benefits were eliminated by the selective blockade of the CB2 receptor,Authors want to thank the support by grants from MCINN “Programa Nacional de Biomedicina” (PID2021-128906OB-I00 to JF-R) with co- funding from the European Regional Development Funds (FEDER), “Instituto de Salud Carlos III” (CIBERNED, CB06/05/0089), Symrise AG, Holzminden, Germany, and VivaCell Biotechnology-Spain. These agencies had no further role in study design, the collection, analysis and interpretation of data, in the writing of the report, or in the decision to submit the paper for publication.Peer reviewedElsevierMinisterio de Ciencia e Innovación (España)European CommissionInstituto de Salud Carlos IIIConsejo Superior de Investigaciones Científicas [https://ror.org/02gfc7t72]202520252025info:eu-repo/semantics/articlehttp://purl.org/coar/resource_type/c_6501Publisher's versioninfo:eu-repo/semantics/publishedVersionhttp://hdl.handle.net/10261/388571reponame:DIGITAL.CSIC. Repositorio Institucional del CSICinstname:Consejo Superior de Investigaciones Científicas (CSIC)Inglés#PLACEHOLDER_PARENT_METADATA_VALUE#PID2021-128906OB-I00https://doi.org/10.1016/j.ejmcr.2025.100262Síinfo:eu-repo/semantics/openAccessoai:digital.csic.es:10261/3885712026-05-22T06:33:51Z
dc.title.none.fl_str_mv Investigation in the CB1 and CB2 receptor binding profile and intrinsic activity of ( ) and (+)-enantiomers of some naturally occurring phytocannabinoids or synthetic derivatives
title Investigation in the CB1 and CB2 receptor binding profile and intrinsic activity of ( ) and (+)-enantiomers of some naturally occurring phytocannabinoids or synthetic derivatives
spellingShingle Investigation in the CB1 and CB2 receptor binding profile and intrinsic activity of ( ) and (+)-enantiomers of some naturally occurring phytocannabinoids or synthetic derivatives
Rodríguez-Carreiro, Santiago
CBD
CBDV
CBDA-Derivatives
( )- and (+)-enantiomers
CB1 receptor
CB2 receptor
Binding
Agonist
Inverse agonist
Antagonist
Anti-inflammatory and neuroprotective effects
title_short Investigation in the CB1 and CB2 receptor binding profile and intrinsic activity of ( ) and (+)-enantiomers of some naturally occurring phytocannabinoids or synthetic derivatives
title_full Investigation in the CB1 and CB2 receptor binding profile and intrinsic activity of ( ) and (+)-enantiomers of some naturally occurring phytocannabinoids or synthetic derivatives
title_fullStr Investigation in the CB1 and CB2 receptor binding profile and intrinsic activity of ( ) and (+)-enantiomers of some naturally occurring phytocannabinoids or synthetic derivatives
title_full_unstemmed Investigation in the CB1 and CB2 receptor binding profile and intrinsic activity of ( ) and (+)-enantiomers of some naturally occurring phytocannabinoids or synthetic derivatives
title_sort Investigation in the CB1 and CB2 receptor binding profile and intrinsic activity of ( ) and (+)-enantiomers of some naturally occurring phytocannabinoids or synthetic derivatives
dc.creator.none.fl_str_mv Rodríguez-Carreiro, Santiago
Gómez-Cañasa, María
Lubrinia, Francesca
Gonzalo-Consuegra, Claudia
Winklerd, Matthias
Caprioglio, Diego
Appendino, Giovanni
García, Concepción
Morales, Paula
Jagerovic, Nadine
Fischer, Joerg T.
Fiebich, Bernd L.
Goetz, Marcus R.
Muñoz, Eduardo
Fernández-Ruiz, Javier
author Rodríguez-Carreiro, Santiago
author_facet Rodríguez-Carreiro, Santiago
Gómez-Cañasa, María
Lubrinia, Francesca
Gonzalo-Consuegra, Claudia
Winklerd, Matthias
Caprioglio, Diego
Appendino, Giovanni
García, Concepción
Morales, Paula
Jagerovic, Nadine
Fischer, Joerg T.
Fiebich, Bernd L.
Goetz, Marcus R.
Muñoz, Eduardo
Fernández-Ruiz, Javier
author_role author
author2 Gómez-Cañasa, María
Lubrinia, Francesca
Gonzalo-Consuegra, Claudia
Winklerd, Matthias
Caprioglio, Diego
Appendino, Giovanni
García, Concepción
Morales, Paula
Jagerovic, Nadine
Fischer, Joerg T.
Fiebich, Bernd L.
Goetz, Marcus R.
Muñoz, Eduardo
Fernández-Ruiz, Javier
author2_role author
author
author
author
author
author
author
author
author
author
author
author
author
author
dc.contributor.none.fl_str_mv Ministerio de Ciencia e Innovación (España)
European Commission
Instituto de Salud Carlos III
Consejo Superior de Investigaciones Científicas [https://ror.org/02gfc7t72]
dc.subject.none.fl_str_mv CBD
CBDV
CBDA-Derivatives
( )- and (+)-enantiomers
CB1 receptor
CB2 receptor
Binding
Agonist
Inverse agonist
Antagonist
Anti-inflammatory and neuroprotective effects
topic CBD
CBDV
CBDA-Derivatives
( )- and (+)-enantiomers
CB1 receptor
CB2 receptor
Binding
Agonist
Inverse agonist
Antagonist
Anti-inflammatory and neuroprotective effects
description Cannabidiol (CBD) and cannabidivarin (CBDV) have shown promising clinical efficacy for the management of epilepsy, and beneficial effects have been demonstrated for CBD in a diversity of other pathologies (pain, schizophrenia, Tourette syndrome, anxiety). However, the mechanism(s) involved are still largely elusive, as are the molecular target(s) involved. CBD and CBDV do not orthosterically bind the cannabinoid type-1 (CB1) and type-2 (CB2) receptors, showing only modest allosteric modulation of both end-points. CBD and CBDV are biosynthesized as optically highly pure ( )-enantiomers, and most bioactivity data refer to these forms. (+)-CBD and related analogues [(+)-cannabidiolic acid (CBDA), its esters, and (+)-CBDV] can be obtained by chemical synthesis, and we present evidence that the (+)- and ( )-enantiomers of CBD, CBDV and of a selection of derivatives of CBDA have distinct binding profiles and functional activity at the CB1/CB2 receptors. Thus, with the single exception of the methyl ester of CBDA, all the (+)-enantiomers showed higher affinities than the ( )-isomers for both receptors, in particular for the CB2 receptors. The affinity of the (+)-enantiomers for both CB1 and CB2 receptors showed a marked dependence on the nature of the alkyl residue on the aromatic ring and the esterification pattern of CBDA. Potency was rarely in the low nM value for CB1, but generally so for CB2. Enantiomers showing low nM activity were further investigated for their intrinsic activity using GTPγS binding assays. This proved that (+)-CBD, (+)-CBDV and the methyl ester of (+)-CBDA are agonists at the CB2 receptor, with the β-hydroxyethyl ester of (+)-CBDA being an inverse agonist, and its β-hydroxypentyl ester behaving as an agonist at CB1 and an inverse agonist at CB2. Finally, we assayed in vitro the anti-inflammatory and neuroprotective properties of three compounds [(+)-CBD, (+)-CBDV and (+)-CBDA methyl ester] strongly activating CB2, showing their ability to reduce the production of proinflammatory factors and protecting neurons against their toxicity. Remarkably, these benefits were eliminated by the selective blockade of the CB2 receptor,
publishDate 2025
dc.date.none.fl_str_mv 2025
2025
2025
dc.type.none.fl_str_mv info:eu-repo/semantics/article
http://purl.org/coar/resource_type/c_6501
Publisher's version
info:eu-repo/semantics/publishedVersion
format article
status_str publishedVersion
dc.identifier.none.fl_str_mv http://hdl.handle.net/10261/388571
url http://hdl.handle.net/10261/388571
dc.language.none.fl_str_mv Inglés
language_invalid_str_mv Inglés
dc.relation.none.fl_str_mv #PLACEHOLDER_PARENT_METADATA_VALUE#
PID2021-128906OB-I00
https://doi.org/10.1016/j.ejmcr.2025.100262

dc.rights.none.fl_str_mv info:eu-repo/semantics/openAccess
eu_rights_str_mv openAccess
dc.publisher.none.fl_str_mv Elsevier
publisher.none.fl_str_mv Elsevier
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instname:Consejo Superior de Investigaciones Científicas (CSIC)
instname_str Consejo Superior de Investigaciones Científicas (CSIC)
reponame_str DIGITAL.CSIC. Repositorio Institucional del CSIC
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