The E1a Adenoviral Gene Upregulates the Yamanaka Factors to Induce Partial Cellular Reprogramming

The induction of pluripotency by enforced expression of different sets of genes in somatic cells has been achieved with reprogramming technologies first described by Yamanaka’s group. Methodologies for generating induced pluripotent stem cells are as varied as the combinations of genes used. It has...

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Detalles Bibliográficos
Autores: Mendoza, Gracia, González-Pastor, Rebeca, Sánchez, Juan Miguel, Arce-Cerezo, Altamira, Quintanilla, Miguel, Moreno-Bueno, Gema, Pujol, Anna, Belmar-Lopez, Carolina, Martino, Alba de, Riu, Efrén, Rodríguez, Tristán A., Martín-Duque, Pilar
Tipo de recurso: artículo
Estado:Versión publicada
Fecha de publicación:2023
País:España
Institución:Consejo Superior de Investigaciones Científicas (CSIC)
Repositorio:DIGITAL.CSIC. Repositorio Institucional del CSIC
OAI Identifier:oai:digital.csic.es:10261/350320
Acceso en línea:http://hdl.handle.net/10261/350320
Access Level:acceso abierto
Palabra clave:Reprogramming
Induced pluripotent stem cells
Embryonic stem cells
Yamanaka factors
Adenovirus
E1a gene
Descripción
Sumario:The induction of pluripotency by enforced expression of different sets of genes in somatic cells has been achieved with reprogramming technologies first described by Yamanaka’s group. Methodologies for generating induced pluripotent stem cells are as varied as the combinations of genes used. It has previously been reported that the adenoviral E1a gene can induce the expression of two of the Yamanaka factors (c-Myc and Oct-4) and epigenetic changes. Here, we demonstrate that the E1a-12S over-expression is sufficient to induce pluripotent-like characteristics closely to epiblast stem cells in mouse embryonic fibroblasts through the activation of the pluripotency gene regulatory network. These findings provide not only empirical evidence that the expression of one single factor is sufficient for partial reprogramming but also a potential mechanistic explanation for how viral infection could lead to neoplasia if they are surrounded by the appropriate environment or the right medium, as happens with the tumorogenic niche.