Recognition of fibrotic infarct density by the pattern of local systolic-diastolic myocardial electrical impedance

Myocardial electrical impedance is a biophysical property of the heart that is influenced by the intrinsic structural characteristics of the tissue. Therefore, the structural derangements elicited in a chronic myocardial infarction should cause specific changes in the local systolic-diastolic myocar...

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Detalles Bibliográficos
Autores: Amoros Figueras, Gerard, Jorge, Esther, García Sánchez, Tomás, Bragós Bardia, Ramon|||0000-0002-1373-1588, Rosell Ferrer, Francisco Javier|||0000-0002-9691-328X, Cinca Cuscullola, Juan Maria
Tipo de recurso: artículo
Fecha de publicación:2016
País:España
Institución:Universitat Politècnica de Catalunya (UPC)
Repositorio:UPCommons. Portal del coneixement obert de la UPC
Idioma:inglés
OAI Identifier:oai:upcommons.upc.edu:2117/99738
Acceso en línea:https://hdl.handle.net/2117/99738
https://dx.doi.org/10.3389/fphys.2016.00389
Access Level:acceso abierto
Palabra clave:Impedance (Electricity)
Cardiology -- Research
Healed myocardial infarction
Myocardial electrical impedance
Bioimpedance
Biomedical engineering
Impedància (Electricitat)
Cardiologia -- Investigació
Àrees temàtiques de la UPC::Enginyeria biomèdica::Aparells mèdics::Aparells cardiovasculars
Àrees temàtiques de la UPC::Enginyeria electrònica
Descripción
Sumario:Myocardial electrical impedance is a biophysical property of the heart that is influenced by the intrinsic structural characteristics of the tissue. Therefore, the structural derangements elicited in a chronic myocardial infarction should cause specific changes in the local systolic-diastolic myocardial impedance, but this is not known. This study aimed to characterize the local changes of systolic-diastolic myocardial impedance in a healed myocardial infarction model. Six pigs were successfully submitted to 150 min of left anterior descending (LAD) coronary artery occlusion followed by reperfusion. 4 weeks later, myocardial impedance spectroscopy (1–1000 kHz) was measured at different infarction sites. The electrocardiogram, left ventricular (LV) pressure, LV dP/dt, and aortic blood flow (ABF) were also recorded. A total of 59 LV tissue samples were obtained and histopathological studies were performed to quantify the percentage of fibrosis. Samples were categorized as normal myocardium (<10% fibrosis), heterogeneous scar (10–50%) and dense scar (>50%). Resistivity of normal myocardium depicted phasic changes during the cardiac cycle and its amplitude markedly decreased in dense scar (18 ± 2 ·cm vs. 10 ± 1 ·cm, at 41 kHz; P < 0.001, respectively). The mean phasic resistivity decreased progressively from normal to heterogeneous and dense scar regions (285 ± 10 ·cm, 225 ± 25 ·cm, and 162 ± 6 ·cm, at 41 kHz; P < 0.001 respectively). Moreover, myocardial resistivity and phase angle correlated significantly with the degree of local fibrosis (resistivity: r = 0.86 at 1 kHz, P < 0.001; phase angle: r = 0.84 at 41 kHz, P < 0.001). Myocardial infarcted regions with greater fibrotic content show lower mean impedance values and more depressed systolic-diastolic dynamic impedance changes. In conclusion, this study reveals that differences in the degree of yocardial fibrosis can be detected in vivo by local measurement of phasic systolic-diastolic bioimpedance spectrum. Once this new bioimpedance method could be used via a catheter-based device, it would be of potential clinical applicability for the recognition of fibrotic tissue to guide the ablation of atrial or ventricular arrhythmias.