Unique functionality of 22-nt miRNAs in triggering RDR6-dependent siRNA biogenesis from target transcripts in Arabidopsis

[EN] NA interference pathways can involve amplification of secondary siRNAs by RNA-dependent RNA polymerases. In plants, RDR6-dependent secondary siRNAs arise from transcripts targeted by some microRNAs (miRNAs). Here, Arabidopsis thaliana secondary siRNAs from mRNA as well as trans-acting siRNAs ar...

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Detalles Bibliográficos
Autores: Cuperus, Josh T., Fahlgren, Noah, Garcia-Ruiz, Hernan, Burke, Russell T., Takeda, Atsushi, Sullivan, Christopher M., Gilbert, Sunny D., Montgomery, Taiowa A., Carrington, James C., CARBONELL, ALBERTO|||0000-0001-5628-6632
Tipo de recurso: artículo
Fecha de publicación:2010
País:España
Institución:Universitat Politècnica de València (UPV)
Repositorio:RiuNet. Repositorio Institucional de la Universitat Politécnica de Valéncia
Idioma:inglés
OAI Identifier:oai:riunet.upv.es:10251/162370
Acceso en línea:https://riunet.upv.es/handle/10251/162370
Access Level:acceso abierto
Palabra clave:MicroRNA
RNA silencing
AGO
SiRNA
Descripción
Sumario:[EN] NA interference pathways can involve amplification of secondary siRNAs by RNA-dependent RNA polymerases. In plants, RDR6-dependent secondary siRNAs arise from transcripts targeted by some microRNAs (miRNAs). Here, Arabidopsis thaliana secondary siRNAs from mRNA as well as trans-acting siRNAs are shown to be triggered through initial targeting by a 22-nucleotide (nt) miRNA that associates with AGO1. In contrast to canonical 21-nt miRNAs, 22-nt miRNAs primarily arise from foldback precursors containing asymmetric bulges. Using artificial miRNA constructs, conversion of asymmetric foldbacks to symmetric foldbacks resulted in the production of 21-nt forms of miR173, miR472 and miR828. Both 21- and 22-nt forms associated with AGO 1 and guided accurate slicer activity, but only 22-nt forms were competent to trigger RDR6-dependent siRNA production from target RNA. These data suggest that AGO 1 functions differentially with 21- and 22-nt miRNAs to engage the RDR6-associated amplification apparatus.