Targeting β2-adrenergic receptors shows therapeutical benefits in clear cell renal cell carcinoma from Von Hippel–Lindau disease

Von Hippel–Lindau (VHL), is a rare autosomal dominant inherited cancer in which the lack of VHL protein triggers the development of multisystemic tumors such us retinal hemangioblastomas (HB), CNS-HB, and clear cell renal cell carcinoma (ccRCC). ccRCC ranks third in terms of incidence and first in c...

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Autores: Albiñana, Virginia, Gallardo-Vara, Eunate, Rojas-P, Isabel de, Recio-Poveda, Lucía, Aguado, Tania, Canto-Cano, Ana, Aguirre, Daniel T., Serra, Marcelo M., González-Peramato, Pilar, Martínez-Piñeiro, Luis, Cuesta, Ángel M., Botella, Luisa María
Tipo de recurso: artículo
Estado:Versión publicada
Fecha de publicación:2020
País:España
Institución:Consejo Superior de Investigaciones Científicas (CSIC)
Repositorio:DIGITAL.CSIC. Repositorio Institucional del CSIC
OAI Identifier:oai:digital.csic.es:10261/220318
Acceso en línea:http://hdl.handle.net/10261/220318
Access Level:acceso abierto
Palabra clave:β-adrenergic receptor antagonist
ICI-118,551
propranolol
HIF
ccRCC
VHL
Anticarcinogenic
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spelling Targeting β2-adrenergic receptors shows therapeutical benefits in clear cell renal cell carcinoma from Von Hippel–Lindau diseaseAlbiñana, VirginiaGallardo-Vara, EunateRojas-P, Isabel deRecio-Poveda, LucíaAguado, TaniaCanto-Cano, AnaAguirre, Daniel T.Serra, Marcelo M.González-Peramato, PilarMartínez-Piñeiro, LuisCuesta, Ángel M.Botella, Luisa Maríaβ-adrenergic receptor antagonistICI-118,551propranololHIFccRCCVHLAnticarcinogenicVon Hippel–Lindau (VHL), is a rare autosomal dominant inherited cancer in which the lack of VHL protein triggers the development of multisystemic tumors such us retinal hemangioblastomas (HB), CNS-HB, and clear cell renal cell carcinoma (ccRCC). ccRCC ranks third in terms of incidence and first in cause of death. Standard systemic therapies for VHL-ccRCC have shown limited response, with recurrent surgeries being the only effective treatment. Targeting of β2-adrenergic receptor (ADRB) has shown therapeutic antitumor benefits on VHL-retinal HB (clinical trial) and VHL-CNS HB (in vitro). Therefore, the in vitro and in vivo antitumor benefits of propranolol (ADRB-1,2 antagonist) and ICI-118,551 (ADRB-2 antagonist) on VHL−/− ccRCC primary cultures and 786-O tumor cell lines have been addressed. Propranolol and ICI-118,551 activated apoptosis inhibited gene and protein expression of HIF-2α, CAIX, and VEGF, and impaired partially the nuclear internalization of HIF-2α and NFĸB/p65. Moreover, propranolol and ICI-118,551 reduced tumor growth on two in vivo xenografts. Finally, ccRCC patients receiving propranolol as off-label treatment have shown a positive therapeutic response for two years on average. In summary, propranolol and ICI-118,551 have shown antitumor benefits in VHL-derived ccRCC, and since ccRCCs comprise 63% of the total RCCs, targeting ADRB2 becomes a promising drug for VHL and other non-VHL tumors.This research was funded by Ministry of Economy and Competitivity, grant number SAF2017-83351-R. L.M-P. was supported by grants from Madrid Regional Government “IMMUNOTHERCAN” [B2017/BMD-3733-2]. A.M.C. was funded by the Spanish Alliance of VHL patients and V.A. was funded by CIBERER.Peer reviewedMultidisciplinary Digital Publishing InstituteMinisterio de Ciencia, Innovación y Universidades (España)Agencia Estatal de Investigación (España)Consejo Superior de Investigaciones Científicas [https://ror.org/02gfc7t72]2020202020202020info:eu-repo/semantics/articlehttp://purl.org/coar/resource_type/c_6501Publisher's versioninfo:eu-repo/semantics/publishedVersionhttp://hdl.handle.net/10261/220318reponame:DIGITAL.CSIC. Repositorio Institucional del CSICinstname:Consejo Superior de Investigaciones Científicas (CSIC)Inglés#PLACEHOLDER_PARENT_METADATA_VALUE##PLACEHOLDER_PARENT_METADATA_VALUE##PLACEHOLDER_PARENT_METADATA_VALUE#info:eu-repo/grantAgreement/AEI/Plan Estatal de Investigación Científica y Técnica y de Innovación 2017-2020/SAF2017-83351-RSAF2017-83351-R/AEI/10.13039/501100011033B2017/BMD-3733-2/IMMUNOTHERCANhttps://doi.org/10.3390/jcm9092740Síinfo:eu-repo/semantics/openAccessoai:digital.csic.es:10261/2203182026-05-22T06:33:51Z
dc.title.none.fl_str_mv Targeting β2-adrenergic receptors shows therapeutical benefits in clear cell renal cell carcinoma from Von Hippel–Lindau disease
title Targeting β2-adrenergic receptors shows therapeutical benefits in clear cell renal cell carcinoma from Von Hippel–Lindau disease
spellingShingle Targeting β2-adrenergic receptors shows therapeutical benefits in clear cell renal cell carcinoma from Von Hippel–Lindau disease
Albiñana, Virginia
β-adrenergic receptor antagonist
ICI-118,551
propranolol
HIF
ccRCC
VHL
Anticarcinogenic
title_short Targeting β2-adrenergic receptors shows therapeutical benefits in clear cell renal cell carcinoma from Von Hippel–Lindau disease
title_full Targeting β2-adrenergic receptors shows therapeutical benefits in clear cell renal cell carcinoma from Von Hippel–Lindau disease
title_fullStr Targeting β2-adrenergic receptors shows therapeutical benefits in clear cell renal cell carcinoma from Von Hippel–Lindau disease
title_full_unstemmed Targeting β2-adrenergic receptors shows therapeutical benefits in clear cell renal cell carcinoma from Von Hippel–Lindau disease
title_sort Targeting β2-adrenergic receptors shows therapeutical benefits in clear cell renal cell carcinoma from Von Hippel–Lindau disease
dc.creator.none.fl_str_mv Albiñana, Virginia
Gallardo-Vara, Eunate
Rojas-P, Isabel de
Recio-Poveda, Lucía
Aguado, Tania
Canto-Cano, Ana
Aguirre, Daniel T.
Serra, Marcelo M.
González-Peramato, Pilar
Martínez-Piñeiro, Luis
Cuesta, Ángel M.
Botella, Luisa María
author Albiñana, Virginia
author_facet Albiñana, Virginia
Gallardo-Vara, Eunate
Rojas-P, Isabel de
Recio-Poveda, Lucía
Aguado, Tania
Canto-Cano, Ana
Aguirre, Daniel T.
Serra, Marcelo M.
González-Peramato, Pilar
Martínez-Piñeiro, Luis
Cuesta, Ángel M.
Botella, Luisa María
author_role author
author2 Gallardo-Vara, Eunate
Rojas-P, Isabel de
Recio-Poveda, Lucía
Aguado, Tania
Canto-Cano, Ana
Aguirre, Daniel T.
Serra, Marcelo M.
González-Peramato, Pilar
Martínez-Piñeiro, Luis
Cuesta, Ángel M.
Botella, Luisa María
author2_role author
author
author
author
author
author
author
author
author
author
author
dc.contributor.none.fl_str_mv Ministerio de Ciencia, Innovación y Universidades (España)
Agencia Estatal de Investigación (España)
Consejo Superior de Investigaciones Científicas [https://ror.org/02gfc7t72]
dc.subject.none.fl_str_mv β-adrenergic receptor antagonist
ICI-118,551
propranolol
HIF
ccRCC
VHL
Anticarcinogenic
topic β-adrenergic receptor antagonist
ICI-118,551
propranolol
HIF
ccRCC
VHL
Anticarcinogenic
description Von Hippel–Lindau (VHL), is a rare autosomal dominant inherited cancer in which the lack of VHL protein triggers the development of multisystemic tumors such us retinal hemangioblastomas (HB), CNS-HB, and clear cell renal cell carcinoma (ccRCC). ccRCC ranks third in terms of incidence and first in cause of death. Standard systemic therapies for VHL-ccRCC have shown limited response, with recurrent surgeries being the only effective treatment. Targeting of β2-adrenergic receptor (ADRB) has shown therapeutic antitumor benefits on VHL-retinal HB (clinical trial) and VHL-CNS HB (in vitro). Therefore, the in vitro and in vivo antitumor benefits of propranolol (ADRB-1,2 antagonist) and ICI-118,551 (ADRB-2 antagonist) on VHL−/− ccRCC primary cultures and 786-O tumor cell lines have been addressed. Propranolol and ICI-118,551 activated apoptosis inhibited gene and protein expression of HIF-2α, CAIX, and VEGF, and impaired partially the nuclear internalization of HIF-2α and NFĸB/p65. Moreover, propranolol and ICI-118,551 reduced tumor growth on two in vivo xenografts. Finally, ccRCC patients receiving propranolol as off-label treatment have shown a positive therapeutic response for two years on average. In summary, propranolol and ICI-118,551 have shown antitumor benefits in VHL-derived ccRCC, and since ccRCCs comprise 63% of the total RCCs, targeting ADRB2 becomes a promising drug for VHL and other non-VHL tumors.
publishDate 2020
dc.date.none.fl_str_mv 2020
2020
2020
2020
dc.type.none.fl_str_mv info:eu-repo/semantics/article
http://purl.org/coar/resource_type/c_6501
Publisher's version
info:eu-repo/semantics/publishedVersion
format article
status_str publishedVersion
dc.identifier.none.fl_str_mv http://hdl.handle.net/10261/220318
url http://hdl.handle.net/10261/220318
dc.language.none.fl_str_mv Inglés
language_invalid_str_mv Inglés
dc.relation.none.fl_str_mv #PLACEHOLDER_PARENT_METADATA_VALUE#
#PLACEHOLDER_PARENT_METADATA_VALUE#
#PLACEHOLDER_PARENT_METADATA_VALUE#
info:eu-repo/grantAgreement/AEI/Plan Estatal de Investigación Científica y Técnica y de Innovación 2017-2020/SAF2017-83351-R
SAF2017-83351-R/AEI/10.13039/501100011033
B2017/BMD-3733-2/IMMUNOTHERCAN
https://doi.org/10.3390/jcm9092740

dc.rights.none.fl_str_mv info:eu-repo/semantics/openAccess
eu_rights_str_mv openAccess
dc.publisher.none.fl_str_mv Multidisciplinary Digital Publishing Institute
publisher.none.fl_str_mv Multidisciplinary Digital Publishing Institute
dc.source.none.fl_str_mv reponame:DIGITAL.CSIC. Repositorio Institucional del CSIC
instname:Consejo Superior de Investigaciones Científicas (CSIC)
instname_str Consejo Superior de Investigaciones Científicas (CSIC)
reponame_str DIGITAL.CSIC. Repositorio Institucional del CSIC
collection DIGITAL.CSIC. Repositorio Institucional del CSIC
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repository.mail.fl_str_mv
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