Purinergic receptors in ocular inflammation

Inflammation is a complex process that implies the interaction between cells and molecular mediators, which, when not properly 'tuned,' can lead to disease. When inflammation affects the eye, it can produce severe disorders affecting the superficial and internal parts of the visual organ....

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Detalhes bibliográficos
Autores: Guzman-Aranguez, A., Gasull Casanova, Xavier, Diebold, Y., Pintor, Jesús J.
Formato: artículo
Estado:Versión publicada
Fecha de publicación:2014
País:España
Recursos:Universidad de Barcelona
Repositorio:Dipòsit Digital de la UB
OAI Identifier:oai:diposit.ub.edu:2445/60525
Acesso em linha:https://hdl.handle.net/2445/60525
Access Level:acceso abierto
Palavra-chave:Inflamació
Adenosina
Nucleòtids
Oftalmologia
Inflammation
Adenosine
Nucleotides
Ophthalmology
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spelling Purinergic receptors in ocular inflammationGuzman-Aranguez, A.Gasull Casanova, XavierDiebold, Y.Pintor, Jesús J.InflamacióAdenosinaNucleòtidsOftalmologiaInflammationAdenosineNucleotidesOphthalmologyInflammation is a complex process that implies the interaction between cells and molecular mediators, which, when not properly 'tuned,' can lead to disease. When inflammation affects the eye, it can produce severe disorders affecting the superficial and internal parts of the visual organ. The nucleoside adenosine and nucleotides including adenine mononucleotides like ADP and ATP and dinucleotides such as P(1),P(4)-diadenosine tetraphosphate (Ap4A), and P(1),P(5)-diadenosine pentaphosphate (Ap5A) are present in different ocular locations and therefore they may contribute/modulate inflammatory processes. Adenosine receptors, in particular A2A adenosine receptors, present anti-inflammatory action in acute and chronic retinal inflammation. Regarding the A3 receptor, selective agonists like N(6)-(3-iodobenzyl)-5'-N-methylcarboxamidoadenosine (CF101) have been used for the treatment of inflammatory ophthalmic diseases such as dry eye and uveoretinitis. Sideways, diverse stimuli (sensory stimulation, large intraocular pressure increases) can produce a release of ATP from ocular sensory innervation or after injury to ocular tissues. Then, ATP will activate purinergic P2 receptors present in sensory nerve endings, the iris, the ciliary body, or other tissues surrounding the anterior chamber of the eye to produce uveitis/endophthalmitis. In summary, adenosine and nucleotides can activate receptors in ocular structures susceptible to suffer from inflammatory processes. This involvement suggests the possible use of purinergic agonists and antagonists as therapeutic targets for ocular inflammation.Hindawi Publishing Corporation2014info:eu-repo/semantics/articleinfo:eu-repo/semantics/publishedVersionapplication/pdfhttps://hdl.handle.net/2445/60525Articles publicats en revistes (Ciències Fisiològiques)reponame:Dipòsit Digital de la UBinstname:Universidad de BarcelonaInglésReproducció del document publicat a: http://dx.doi.org/10.1155/2014/320906Mediators of Inflammation, 2014, vol. 2014, p. 1-11http://dx.doi.org/10.1155/2014/320906cc-by (c) Guzman-Aranguez, A. et al., 2014http://creativecommons.org/licenses/by/3.0/esinfo:eu-repo/semantics/openAccessoai:diposit.ub.edu:2445/605252026-05-27T06:46:51Z
dc.title.none.fl_str_mv Purinergic receptors in ocular inflammation
title Purinergic receptors in ocular inflammation
spellingShingle Purinergic receptors in ocular inflammation
Guzman-Aranguez, A.
Inflamació
Adenosina
Nucleòtids
Oftalmologia
Inflammation
Adenosine
Nucleotides
Ophthalmology
title_short Purinergic receptors in ocular inflammation
title_full Purinergic receptors in ocular inflammation
title_fullStr Purinergic receptors in ocular inflammation
title_full_unstemmed Purinergic receptors in ocular inflammation
title_sort Purinergic receptors in ocular inflammation
dc.creator.none.fl_str_mv Guzman-Aranguez, A.
Gasull Casanova, Xavier
Diebold, Y.
Pintor, Jesús J.
author Guzman-Aranguez, A.
author_facet Guzman-Aranguez, A.
Gasull Casanova, Xavier
Diebold, Y.
Pintor, Jesús J.
author_role author
author2 Gasull Casanova, Xavier
Diebold, Y.
Pintor, Jesús J.
author2_role author
author
author
dc.subject.none.fl_str_mv Inflamació
Adenosina
Nucleòtids
Oftalmologia
Inflammation
Adenosine
Nucleotides
Ophthalmology
topic Inflamació
Adenosina
Nucleòtids
Oftalmologia
Inflammation
Adenosine
Nucleotides
Ophthalmology
description Inflammation is a complex process that implies the interaction between cells and molecular mediators, which, when not properly 'tuned,' can lead to disease. When inflammation affects the eye, it can produce severe disorders affecting the superficial and internal parts of the visual organ. The nucleoside adenosine and nucleotides including adenine mononucleotides like ADP and ATP and dinucleotides such as P(1),P(4)-diadenosine tetraphosphate (Ap4A), and P(1),P(5)-diadenosine pentaphosphate (Ap5A) are present in different ocular locations and therefore they may contribute/modulate inflammatory processes. Adenosine receptors, in particular A2A adenosine receptors, present anti-inflammatory action in acute and chronic retinal inflammation. Regarding the A3 receptor, selective agonists like N(6)-(3-iodobenzyl)-5'-N-methylcarboxamidoadenosine (CF101) have been used for the treatment of inflammatory ophthalmic diseases such as dry eye and uveoretinitis. Sideways, diverse stimuli (sensory stimulation, large intraocular pressure increases) can produce a release of ATP from ocular sensory innervation or after injury to ocular tissues. Then, ATP will activate purinergic P2 receptors present in sensory nerve endings, the iris, the ciliary body, or other tissues surrounding the anterior chamber of the eye to produce uveitis/endophthalmitis. In summary, adenosine and nucleotides can activate receptors in ocular structures susceptible to suffer from inflammatory processes. This involvement suggests the possible use of purinergic agonists and antagonists as therapeutic targets for ocular inflammation.
publishDate 2014
dc.date.none.fl_str_mv 2014
dc.type.none.fl_str_mv info:eu-repo/semantics/article
info:eu-repo/semantics/publishedVersion
format article
status_str publishedVersion
dc.identifier.none.fl_str_mv https://hdl.handle.net/2445/60525
url https://hdl.handle.net/2445/60525
dc.language.none.fl_str_mv Inglés
language_invalid_str_mv Inglés
dc.relation.none.fl_str_mv Reproducció del document publicat a: http://dx.doi.org/10.1155/2014/320906
Mediators of Inflammation, 2014, vol. 2014, p. 1-11
http://dx.doi.org/10.1155/2014/320906
dc.rights.none.fl_str_mv cc-by (c) Guzman-Aranguez, A. et al., 2014
http://creativecommons.org/licenses/by/3.0/es
info:eu-repo/semantics/openAccess
rights_invalid_str_mv cc-by (c) Guzman-Aranguez, A. et al., 2014
http://creativecommons.org/licenses/by/3.0/es
eu_rights_str_mv openAccess
dc.format.none.fl_str_mv application/pdf
dc.publisher.none.fl_str_mv Hindawi Publishing Corporation
publisher.none.fl_str_mv Hindawi Publishing Corporation
dc.source.none.fl_str_mv Articles publicats en revistes (Ciències Fisiològiques)
reponame:Dipòsit Digital de la UB
instname:Universidad de Barcelona
instname_str Universidad de Barcelona
reponame_str Dipòsit Digital de la UB
collection Dipòsit Digital de la UB
repository.name.fl_str_mv
repository.mail.fl_str_mv
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