A bicyclic α-iminophosphonate improves cognitive decline in 5xFAD murine model of neurodegeneration

I2 receptors (I2-IR) are widely distributed in the central nervous system. I2-IR ligands are associated with a neuroprotective effect but, as I2-IR structure remains unknown, the discovery of better and more selective ligands is necessary to understand the pharmacological and molecular implications...

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Autores: Escolano Mirón, Carmen, Abás Prades, Sònia, Rodríguez-Arévalo, Sergio, Bagan Polonio, Andrea, Griñán Ferré, Christian, Vasilopoulou, Foteini, Brocos-Mosquera, Iria, Muguruza, Carolina, Callado, Luis F., Pérez, Belén, Pérez Lozano, Pilar, Brea, José, Loza, María Isabel, Hernández-Hernández, Elena, García-Sevilla, Jesús A, García-Fuster, M. Julia, Radan, Milica, Djikic, Teodora, Nikolic, Katarina, Díaz, C., Pérez, J., Ramos Guerra, Cristian, Vicente, Filipa, Molins i Grau, Elies, Pallàs i Llibería, Mercè, 1964-
Tipo de recurso: artículo
Estado:Versión enviada para evaluación y publicación
Fecha de publicación:2021
País:España
Institución:Varias* (Consorci de Biblioteques Universitáries de Catalunya, Centre de Serveis Científics i Acadèmics de Catalunya)
Repositorio:Recercat. Dipósit de la Recerca de Catalunya
OAI Identifier:oai:recercat.cat:2445/178883
Acceso en línea:https://hdl.handle.net/2445/178883
Access Level:acceso abierto
Palabra clave:Malaltia d'Alzheimer
Malalties neurodegeneratives
Envelliment
Alzheimer's disease
Neurodegenerative Diseases
Aging
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spelling A bicyclic α-iminophosphonate improves cognitive decline in 5xFAD murine model of neurodegenerationEscolano Mirón, CarmenAbás Prades, SòniaRodríguez-Arévalo, SergioBagan Polonio, AndreaGriñán Ferré, ChristianVasilopoulou, FoteiniBrocos-Mosquera, IriaMuguruza, CarolinaCallado, Luis F.Pérez, BelénPérez Lozano, PilarBrea, JoséLoza, María IsabelHernández-Hernández, ElenaGarcía-Sevilla, Jesús AGarcía-Fuster, M. JuliaRadan, MilicaDjikic, TeodoraNikolic, KatarinaDíaz, C.Pérez, J.Ramos Guerra, CristianVicente, FilipaMolins i Grau, EliesPallàs i Llibería, Mercè, 1964-Malaltia d'AlzheimerMalalties neurodegenerativesEnvellimentAlzheimer's diseaseNeurodegenerative DiseasesAgingI2 receptors (I2-IR) are widely distributed in the central nervous system. I2-IR ligands are associated with a neuroprotective effect but, as I2-IR structure remains unknown, the discovery of better and more selective ligands is necessary to understand the pharmacological and molecular implications of I2-IR. Recently, we described a new imidazoline-structure family which showed high affinity and selectivity for I2-IR. In vivo studies in mice indicated a neuroprotective role and revealed beneficial effects in behaviour and cognition with a murine model of neurodegeneration, senescence-accelerated prone mouse (SAMP8). Herein, we report a novel non-imidazoline-structure of bicyclic α-iminophosphonates family with high affinities for I2-IR. In vivo studies in 5X-FAD mice (a transgenic representative model of AD) and SAMP8 mice (a model of neurodegeneration linked to aging) showed an improvement in behaviour and cognition, a reduction of AD hallmarks and of neuroinflammation markers for the mice treated with the lead compound B06. After evaluating several pathways associated with neurodegeneration, we demonstrated that CaN pathway plays a critical role on the neuroprotective effects of I2-IR ligands on SAMP8 mice model. To rule out warnings of the novel family, we calculated DMPK and physicochemical properties for the novel bicyclic α-iminophosphonates. As well, we carried out drug metabolism, safety studies and in vivo pharmacokinetics for lead compound B06. In summary, we present a novel family of I2-IR ligands, its effectiveness in in vivo models and the possible neuroprotective molecular mechanism mediated by them. This highlights that the modulation of I2-IR by bicyclic α-iminophosphonates may open a new therapeutic venue for unmet neurodegenerative conditions.The Federation of American Society of Experimental Biology2021202120212021info:eu-repo/semantics/articleinfo:eu-repo/semantics/submittedVersionapplication/pdfhttps://hdl.handle.net/2445/178883Articles publicats en revistes (Farmacologia, Toxicologia i Química Terapèutica)reponame:Recercat. Dipósit de la Recerca de Catalunyainstname:Varias* (Consorci de Biblioteques Universitáries de Catalunya, Centre de Serveis Científics i Acadèmics de Catalunya)InglésVersió preprint del document publicat a: https://doi.org/10.1096/fasebj.2021.35.S1.04974The FASEB Journal , 2021, vol. 35, num. S1https://doi.org/10.1096/fasebj.2021.35.S1.04974(c) The Federation of American Society of Experimental Biology, 2021info:eu-repo/semantics/openAccessoai:recercat.cat:2445/1788832026-05-29T05:05:01Z
dc.title.none.fl_str_mv A bicyclic α-iminophosphonate improves cognitive decline in 5xFAD murine model of neurodegeneration
title A bicyclic α-iminophosphonate improves cognitive decline in 5xFAD murine model of neurodegeneration
spellingShingle A bicyclic α-iminophosphonate improves cognitive decline in 5xFAD murine model of neurodegeneration
Escolano Mirón, Carmen
Malaltia d'Alzheimer
Malalties neurodegeneratives
Envelliment
Alzheimer's disease
Neurodegenerative Diseases
Aging
title_short A bicyclic α-iminophosphonate improves cognitive decline in 5xFAD murine model of neurodegeneration
title_full A bicyclic α-iminophosphonate improves cognitive decline in 5xFAD murine model of neurodegeneration
title_fullStr A bicyclic α-iminophosphonate improves cognitive decline in 5xFAD murine model of neurodegeneration
title_full_unstemmed A bicyclic α-iminophosphonate improves cognitive decline in 5xFAD murine model of neurodegeneration
title_sort A bicyclic α-iminophosphonate improves cognitive decline in 5xFAD murine model of neurodegeneration
dc.creator.none.fl_str_mv Escolano Mirón, Carmen
Abás Prades, Sònia
Rodríguez-Arévalo, Sergio
Bagan Polonio, Andrea
Griñán Ferré, Christian
Vasilopoulou, Foteini
Brocos-Mosquera, Iria
Muguruza, Carolina
Callado, Luis F.
Pérez, Belén
Pérez Lozano, Pilar
Brea, José
Loza, María Isabel
Hernández-Hernández, Elena
García-Sevilla, Jesús A
García-Fuster, M. Julia
Radan, Milica
Djikic, Teodora
Nikolic, Katarina
Díaz, C.
Pérez, J.
Ramos Guerra, Cristian
Vicente, Filipa
Molins i Grau, Elies
Pallàs i Llibería, Mercè, 1964-
author Escolano Mirón, Carmen
author_facet Escolano Mirón, Carmen
Abás Prades, Sònia
Rodríguez-Arévalo, Sergio
Bagan Polonio, Andrea
Griñán Ferré, Christian
Vasilopoulou, Foteini
Brocos-Mosquera, Iria
Muguruza, Carolina
Callado, Luis F.
Pérez, Belén
Pérez Lozano, Pilar
Brea, José
Loza, María Isabel
Hernández-Hernández, Elena
García-Sevilla, Jesús A
García-Fuster, M. Julia
Radan, Milica
Djikic, Teodora
Nikolic, Katarina
Díaz, C.
Pérez, J.
Ramos Guerra, Cristian
Vicente, Filipa
Molins i Grau, Elies
Pallàs i Llibería, Mercè, 1964-
author_role author
author2 Abás Prades, Sònia
Rodríguez-Arévalo, Sergio
Bagan Polonio, Andrea
Griñán Ferré, Christian
Vasilopoulou, Foteini
Brocos-Mosquera, Iria
Muguruza, Carolina
Callado, Luis F.
Pérez, Belén
Pérez Lozano, Pilar
Brea, José
Loza, María Isabel
Hernández-Hernández, Elena
García-Sevilla, Jesús A
García-Fuster, M. Julia
Radan, Milica
Djikic, Teodora
Nikolic, Katarina
Díaz, C.
Pérez, J.
Ramos Guerra, Cristian
Vicente, Filipa
Molins i Grau, Elies
Pallàs i Llibería, Mercè, 1964-
author2_role author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
dc.subject.none.fl_str_mv Malaltia d'Alzheimer
Malalties neurodegeneratives
Envelliment
Alzheimer's disease
Neurodegenerative Diseases
Aging
topic Malaltia d'Alzheimer
Malalties neurodegeneratives
Envelliment
Alzheimer's disease
Neurodegenerative Diseases
Aging
description I2 receptors (I2-IR) are widely distributed in the central nervous system. I2-IR ligands are associated with a neuroprotective effect but, as I2-IR structure remains unknown, the discovery of better and more selective ligands is necessary to understand the pharmacological and molecular implications of I2-IR. Recently, we described a new imidazoline-structure family which showed high affinity and selectivity for I2-IR. In vivo studies in mice indicated a neuroprotective role and revealed beneficial effects in behaviour and cognition with a murine model of neurodegeneration, senescence-accelerated prone mouse (SAMP8). Herein, we report a novel non-imidazoline-structure of bicyclic α-iminophosphonates family with high affinities for I2-IR. In vivo studies in 5X-FAD mice (a transgenic representative model of AD) and SAMP8 mice (a model of neurodegeneration linked to aging) showed an improvement in behaviour and cognition, a reduction of AD hallmarks and of neuroinflammation markers for the mice treated with the lead compound B06. After evaluating several pathways associated with neurodegeneration, we demonstrated that CaN pathway plays a critical role on the neuroprotective effects of I2-IR ligands on SAMP8 mice model. To rule out warnings of the novel family, we calculated DMPK and physicochemical properties for the novel bicyclic α-iminophosphonates. As well, we carried out drug metabolism, safety studies and in vivo pharmacokinetics for lead compound B06. In summary, we present a novel family of I2-IR ligands, its effectiveness in in vivo models and the possible neuroprotective molecular mechanism mediated by them. This highlights that the modulation of I2-IR by bicyclic α-iminophosphonates may open a new therapeutic venue for unmet neurodegenerative conditions.
publishDate 2021
dc.date.none.fl_str_mv 2021
2021
2021
2021
dc.type.none.fl_str_mv info:eu-repo/semantics/article
info:eu-repo/semantics/submittedVersion
format article
status_str submittedVersion
dc.identifier.none.fl_str_mv https://hdl.handle.net/2445/178883
url https://hdl.handle.net/2445/178883
dc.language.none.fl_str_mv Inglés
language_invalid_str_mv Inglés
dc.relation.none.fl_str_mv Versió preprint del document publicat a: https://doi.org/10.1096/fasebj.2021.35.S1.04974
The FASEB Journal , 2021, vol. 35, num. S1
https://doi.org/10.1096/fasebj.2021.35.S1.04974
dc.rights.none.fl_str_mv (c) The Federation of American Society of Experimental Biology, 2021
info:eu-repo/semantics/openAccess
rights_invalid_str_mv (c) The Federation of American Society of Experimental Biology, 2021
eu_rights_str_mv openAccess
dc.format.none.fl_str_mv application/pdf
dc.publisher.none.fl_str_mv The Federation of American Society of Experimental Biology
publisher.none.fl_str_mv The Federation of American Society of Experimental Biology
dc.source.none.fl_str_mv Articles publicats en revistes (Farmacologia, Toxicologia i Química Terapèutica)
reponame:Recercat. Dipósit de la Recerca de Catalunya
instname:Varias* (Consorci de Biblioteques Universitáries de Catalunya, Centre de Serveis Científics i Acadèmics de Catalunya)
instname_str Varias* (Consorci de Biblioteques Universitáries de Catalunya, Centre de Serveis Científics i Acadèmics de Catalunya)
reponame_str Recercat. Dipósit de la Recerca de Catalunya
collection Recercat. Dipósit de la Recerca de Catalunya
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repository.mail.fl_str_mv
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