Identification of shared risk loci and pathways for bipolar disorder and schizophrenia

Bipolar disorder (BD) is a highly heritable neuropsychiatric disease characterized by recurrent episodes of mania and depression. BD shows substantial clinical and genetic overlap with other psychiatric disorders, in particular schizophrenia (SCZ). The genes underlying this etiological overlap remai...

Descripción completa

Detalles Bibliográficos
Autores: Forstner, Andreas J., Hecker, Julian, Hofmann, Andrea, Reinbold, Celine S., Mühleisen, Thomas W., Leber, Markus, Strohmaier, Jana, Degenhardt, Franziska, Treutlein, Jens, Mattheisen, Manuel, Schumacher, Johannes, Streit, Fabian, Meier, Sandra, Herms, Stefan, Hoffmann, Per, Lacour, André, Witt, Stephanie H., Maaser, Anna, Reif, Andreas, Müller-Myhsok, Bertram, Lucae, Susanne, Maier, Wolfgang, Schwarz, Markus, Vedder, Helmut, Kammerer-Ciernioch, Jutta, Pfennig, Andrea, Bauer, Michael, Hautzinger, Martin, Moebus, Susanne, Schenk, Lorena M., Fischer, Sascha B., Sivalingam, Sugirthan, Czerski, Piotr M., Hauser, Joanna, Lissowska, Jolanta, Szeszenia-Dabrowska, Neonila, Brennan, Paul, McKay, James D., Wright, Adam, Mitchell, Philip B., Fullerton, Janice M., Schofield, Peter R., Montgomery, Grant W., Medland, Sarah E., Gordon, Scott D., Martin, Nicholas G., Krasnov, Valery, Chuchalin, Alexander, Babadjanova, Gulja, Pantelejeva, Galina, Abramova, Lilia I., Tiganov, Alexander S., Polonikov, Alexey, Khusnutdinova, Elza, Alda, Martin, Cruceanu, Cristiana, Rouleau, Guy A., Turecki, Gustavo, Laprise, Catherine, Rivas, Fabio, Mayoral, Fermin, Kogevinas, Manolis, Grigoroiu-Serbanescu, Maria, Becker, Tim, Schulze, Thomas G., Rietschel, Manolis, Cichon, Sven, Fier, Heide, Nöthen, Markus M.
Tipo de recurso: artículo
Estado:Versión publicada
Fecha de publicación:2017
País:España
Institución:Universidad de Barcelona
Repositorio:Dipòsit Digital de la UB
OAI Identifier:oai:diposit.ub.edu:2445/107944
Acceso en línea:https://hdl.handle.net/2445/107944
Access Level:acceso abierto
Palabra clave:Trastorn bipolar
Esquizofrènia
Manic-depressive illness
Schizophrenia
id ES_ad6f1e82e17e3fe150d85ca9cbb2e1c3
oai_identifier_str oai:diposit.ub.edu:2445/107944
network_acronym_str ES
network_name_str España
repository_id_str
spelling Identification of shared risk loci and pathways for bipolar disorder and schizophreniaForstner, Andreas J.Hecker, JulianHofmann, AndreaReinbold, Celine S.Mühleisen, Thomas W.Leber, MarkusStrohmaier, JanaDegenhardt, FranziskaTreutlein, JensMattheisen, ManuelSchumacher, JohannesStreit, FabianMeier, SandraHerms, StefanHoffmann, PerLacour, AndréWitt, Stephanie H.Maaser, AnnaReif, AndreasMüller-Myhsok, BertramLucae, SusanneMaier, WolfgangSchwarz, MarkusVedder, HelmutKammerer-Ciernioch, JuttaPfennig, AndreaBauer, MichaelHautzinger, MartinMoebus, SusanneSchenk, Lorena M.Fischer, Sascha B.Sivalingam, SugirthanCzerski, Piotr M.Hauser, JoannaLissowska, JolantaSzeszenia-Dabrowska, NeonilaBrennan, PaulMcKay, James D.Wright, AdamMitchell, Philip B.Fullerton, Janice M.Schofield, Peter R.Montgomery, Grant W.Medland, Sarah E.Gordon, Scott D.Martin, Nicholas G.Krasnov, ValeryChuchalin, AlexanderBabadjanova, GuljaPantelejeva, GalinaAbramova, Lilia I.Tiganov, Alexander S.Polonikov, AlexeyKhusnutdinova, ElzaAlda, MartinCruceanu, CristianaRouleau, Guy A.Turecki, GustavoLaprise, CatherineRivas, FabioMayoral, FerminKogevinas, ManolisGrigoroiu-Serbanescu, MariaBecker, TimSchulze, Thomas G.Rietschel, ManolisCichon, SvenFier, HeideNöthen, Markus M.Trastorn bipolarEsquizofrèniaManic-depressive illnessSchizophreniaBipolar disorder (BD) is a highly heritable neuropsychiatric disease characterized by recurrent episodes of mania and depression. BD shows substantial clinical and genetic overlap with other psychiatric disorders, in particular schizophrenia (SCZ). The genes underlying this etiological overlap remain largely unknown. A recent SCZ genome wide association study (GWAS) by the Psychiatric Genomics Consortium identified 128 independent genome-wide significant single nucleotide polymorphisms (SNPs). The present study investigated whether these SCZ-associated SNPs also contribute to BD development through the performance of association testing in a large BD GWAS dataset (9747 patients, 14278 controls). After re-imputation and correction for sample overlap, 22 of 107 investigated SCZ SNPs showed nominal association with BD. The number of shared SCZ-BD SNPs was significantly higher than expected (p = 1.46x10-8). This provides further evidence that SCZ-associated loci contribute to the development of BD. Two SNPs remained significant after Bonferroni correction. The most strongly associated SNP was located near TRANK1, which is a reported genome-wide significant risk gene for BD. Pathway analyses for all shared SCZ-BD SNPs revealed 25 nominally enriched gene-sets, which showed partial overlap in terms of the underlying genes. The enriched gene-sets included calcium- and glutamate signaling, neuropathic pain signaling in dorsal horn neurons, and calmodulin binding. The present data provide further insights into shared risk loci and disease-associated pathways for BD and SCZ. This may suggest new research directions for the treatment and prevention of these two major psychiatric disorders.Public Library of Science (PLoS)2017info:eu-repo/semantics/articleinfo:eu-repo/semantics/publishedVersionapplication/pdfhttps://hdl.handle.net/2445/107944Articles publicats en revistes (ISGlobal)reponame:Dipòsit Digital de la UBinstname:Universidad de BarcelonaInglésReproducció del document publicat a: http://dx.doi.org/10.1371/journal.pone.0171595PLoS One, 2017, vol. 12, num. 2, p. e0171595http://dx.doi.org/10.1371/journal.pone.0171595cc by (c) Forstner et al., 2017http://creativecommons.org/licenses/by/3.0/es/info:eu-repo/semantics/openAccessoai:diposit.ub.edu:2445/1079442026-05-27T06:46:51Z
dc.title.none.fl_str_mv Identification of shared risk loci and pathways for bipolar disorder and schizophrenia
title Identification of shared risk loci and pathways for bipolar disorder and schizophrenia
spellingShingle Identification of shared risk loci and pathways for bipolar disorder and schizophrenia
Forstner, Andreas J.
Trastorn bipolar
Esquizofrènia
Manic-depressive illness
Schizophrenia
title_short Identification of shared risk loci and pathways for bipolar disorder and schizophrenia
title_full Identification of shared risk loci and pathways for bipolar disorder and schizophrenia
title_fullStr Identification of shared risk loci and pathways for bipolar disorder and schizophrenia
title_full_unstemmed Identification of shared risk loci and pathways for bipolar disorder and schizophrenia
title_sort Identification of shared risk loci and pathways for bipolar disorder and schizophrenia
dc.creator.none.fl_str_mv Forstner, Andreas J.
Hecker, Julian
Hofmann, Andrea
Reinbold, Celine S.
Mühleisen, Thomas W.
Leber, Markus
Strohmaier, Jana
Degenhardt, Franziska
Treutlein, Jens
Mattheisen, Manuel
Schumacher, Johannes
Streit, Fabian
Meier, Sandra
Herms, Stefan
Hoffmann, Per
Lacour, André
Witt, Stephanie H.
Maaser, Anna
Reif, Andreas
Müller-Myhsok, Bertram
Lucae, Susanne
Maier, Wolfgang
Schwarz, Markus
Vedder, Helmut
Kammerer-Ciernioch, Jutta
Pfennig, Andrea
Bauer, Michael
Hautzinger, Martin
Moebus, Susanne
Schenk, Lorena M.
Fischer, Sascha B.
Sivalingam, Sugirthan
Czerski, Piotr M.
Hauser, Joanna
Lissowska, Jolanta
Szeszenia-Dabrowska, Neonila
Brennan, Paul
McKay, James D.
Wright, Adam
Mitchell, Philip B.
Fullerton, Janice M.
Schofield, Peter R.
Montgomery, Grant W.
Medland, Sarah E.
Gordon, Scott D.
Martin, Nicholas G.
Krasnov, Valery
Chuchalin, Alexander
Babadjanova, Gulja
Pantelejeva, Galina
Abramova, Lilia I.
Tiganov, Alexander S.
Polonikov, Alexey
Khusnutdinova, Elza
Alda, Martin
Cruceanu, Cristiana
Rouleau, Guy A.
Turecki, Gustavo
Laprise, Catherine
Rivas, Fabio
Mayoral, Fermin
Kogevinas, Manolis
Grigoroiu-Serbanescu, Maria
Becker, Tim
Schulze, Thomas G.
Rietschel, Manolis
Cichon, Sven
Fier, Heide
Nöthen, Markus M.
author Forstner, Andreas J.
author_facet Forstner, Andreas J.
Hecker, Julian
Hofmann, Andrea
Reinbold, Celine S.
Mühleisen, Thomas W.
Leber, Markus
Strohmaier, Jana
Degenhardt, Franziska
Treutlein, Jens
Mattheisen, Manuel
Schumacher, Johannes
Streit, Fabian
Meier, Sandra
Herms, Stefan
Hoffmann, Per
Lacour, André
Witt, Stephanie H.
Maaser, Anna
Reif, Andreas
Müller-Myhsok, Bertram
Lucae, Susanne
Maier, Wolfgang
Schwarz, Markus
Vedder, Helmut
Kammerer-Ciernioch, Jutta
Pfennig, Andrea
Bauer, Michael
Hautzinger, Martin
Moebus, Susanne
Schenk, Lorena M.
Fischer, Sascha B.
Sivalingam, Sugirthan
Czerski, Piotr M.
Hauser, Joanna
Lissowska, Jolanta
Szeszenia-Dabrowska, Neonila
Brennan, Paul
McKay, James D.
Wright, Adam
Mitchell, Philip B.
Fullerton, Janice M.
Schofield, Peter R.
Montgomery, Grant W.
Medland, Sarah E.
Gordon, Scott D.
Martin, Nicholas G.
Krasnov, Valery
Chuchalin, Alexander
Babadjanova, Gulja
Pantelejeva, Galina
Abramova, Lilia I.
Tiganov, Alexander S.
Polonikov, Alexey
Khusnutdinova, Elza
Alda, Martin
Cruceanu, Cristiana
Rouleau, Guy A.
Turecki, Gustavo
Laprise, Catherine
Rivas, Fabio
Mayoral, Fermin
Kogevinas, Manolis
Grigoroiu-Serbanescu, Maria
Becker, Tim
Schulze, Thomas G.
Rietschel, Manolis
Cichon, Sven
Fier, Heide
Nöthen, Markus M.
author_role author
author2 Hecker, Julian
Hofmann, Andrea
Reinbold, Celine S.
Mühleisen, Thomas W.
Leber, Markus
Strohmaier, Jana
Degenhardt, Franziska
Treutlein, Jens
Mattheisen, Manuel
Schumacher, Johannes
Streit, Fabian
Meier, Sandra
Herms, Stefan
Hoffmann, Per
Lacour, André
Witt, Stephanie H.
Maaser, Anna
Reif, Andreas
Müller-Myhsok, Bertram
Lucae, Susanne
Maier, Wolfgang
Schwarz, Markus
Vedder, Helmut
Kammerer-Ciernioch, Jutta
Pfennig, Andrea
Bauer, Michael
Hautzinger, Martin
Moebus, Susanne
Schenk, Lorena M.
Fischer, Sascha B.
Sivalingam, Sugirthan
Czerski, Piotr M.
Hauser, Joanna
Lissowska, Jolanta
Szeszenia-Dabrowska, Neonila
Brennan, Paul
McKay, James D.
Wright, Adam
Mitchell, Philip B.
Fullerton, Janice M.
Schofield, Peter R.
Montgomery, Grant W.
Medland, Sarah E.
Gordon, Scott D.
Martin, Nicholas G.
Krasnov, Valery
Chuchalin, Alexander
Babadjanova, Gulja
Pantelejeva, Galina
Abramova, Lilia I.
Tiganov, Alexander S.
Polonikov, Alexey
Khusnutdinova, Elza
Alda, Martin
Cruceanu, Cristiana
Rouleau, Guy A.
Turecki, Gustavo
Laprise, Catherine
Rivas, Fabio
Mayoral, Fermin
Kogevinas, Manolis
Grigoroiu-Serbanescu, Maria
Becker, Tim
Schulze, Thomas G.
Rietschel, Manolis
Cichon, Sven
Fier, Heide
Nöthen, Markus M.
author2_role author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
dc.subject.none.fl_str_mv Trastorn bipolar
Esquizofrènia
Manic-depressive illness
Schizophrenia
topic Trastorn bipolar
Esquizofrènia
Manic-depressive illness
Schizophrenia
description Bipolar disorder (BD) is a highly heritable neuropsychiatric disease characterized by recurrent episodes of mania and depression. BD shows substantial clinical and genetic overlap with other psychiatric disorders, in particular schizophrenia (SCZ). The genes underlying this etiological overlap remain largely unknown. A recent SCZ genome wide association study (GWAS) by the Psychiatric Genomics Consortium identified 128 independent genome-wide significant single nucleotide polymorphisms (SNPs). The present study investigated whether these SCZ-associated SNPs also contribute to BD development through the performance of association testing in a large BD GWAS dataset (9747 patients, 14278 controls). After re-imputation and correction for sample overlap, 22 of 107 investigated SCZ SNPs showed nominal association with BD. The number of shared SCZ-BD SNPs was significantly higher than expected (p = 1.46x10-8). This provides further evidence that SCZ-associated loci contribute to the development of BD. Two SNPs remained significant after Bonferroni correction. The most strongly associated SNP was located near TRANK1, which is a reported genome-wide significant risk gene for BD. Pathway analyses for all shared SCZ-BD SNPs revealed 25 nominally enriched gene-sets, which showed partial overlap in terms of the underlying genes. The enriched gene-sets included calcium- and glutamate signaling, neuropathic pain signaling in dorsal horn neurons, and calmodulin binding. The present data provide further insights into shared risk loci and disease-associated pathways for BD and SCZ. This may suggest new research directions for the treatment and prevention of these two major psychiatric disorders.
publishDate 2017
dc.date.none.fl_str_mv 2017
dc.type.none.fl_str_mv info:eu-repo/semantics/article
info:eu-repo/semantics/publishedVersion
format article
status_str publishedVersion
dc.identifier.none.fl_str_mv https://hdl.handle.net/2445/107944
url https://hdl.handle.net/2445/107944
dc.language.none.fl_str_mv Inglés
language_invalid_str_mv Inglés
dc.relation.none.fl_str_mv Reproducció del document publicat a: http://dx.doi.org/10.1371/journal.pone.0171595
PLoS One, 2017, vol. 12, num. 2, p. e0171595
http://dx.doi.org/10.1371/journal.pone.0171595
dc.rights.none.fl_str_mv cc by (c) Forstner et al., 2017
http://creativecommons.org/licenses/by/3.0/es/
info:eu-repo/semantics/openAccess
rights_invalid_str_mv cc by (c) Forstner et al., 2017
http://creativecommons.org/licenses/by/3.0/es/
eu_rights_str_mv openAccess
dc.format.none.fl_str_mv application/pdf
dc.publisher.none.fl_str_mv Public Library of Science (PLoS)
publisher.none.fl_str_mv Public Library of Science (PLoS)
dc.source.none.fl_str_mv Articles publicats en revistes (ISGlobal)
reponame:Dipòsit Digital de la UB
instname:Universidad de Barcelona
instname_str Universidad de Barcelona
reponame_str Dipòsit Digital de la UB
collection Dipòsit Digital de la UB
repository.name.fl_str_mv
repository.mail.fl_str_mv
_version_ 1869416439211884544
score 15,300719