Cancer risk in patients with rheumatoid arthritis receiving biologic and targeted synthetic disease modifying antirheumatic drugs: results from the BIOBADASER III registry.

OBJECTIVES: To assess the risk of cancer in patients with rheumatoid arthritis (RA) treated with biologic and targeted synthetic disease-modifying antirheumatic drugs (b/tsDMARDs). METHODS: We analyzed RA patients from the BIOBADASER III registry, a multicentre national registry of b/tsDMARDs, inclu...

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Detalhes bibliográficos
Autores: Molina-Collada J, Otero-Varela L, Vela P, Manrique S, Erausquin C, Campos C, Manero J, Calvo J, Expósito L, García González J, Ruiz Montesinos MD, Rabadán E, Varela AM, Vilamajo IR, Colazo Burlato M, Plasencia C, Sánchez-Alonso F, Castrejón I
Formato: artículo
Estado:Versión publicada
Fecha de publicación:2026
País:España
Recursos:Instituto de Investigación Biomédica y Sanitaria de Alicante (ISABIAL)
Repositorio:r-ISABIAL. Repositorio Institucional de Producción Científica del Instituto de Investigación Biomédica y Sanitaria de Alicante
OAI Identifier:oai:isabial.fundanetsuite.com:p11462
Acesso em linha:https://isabial.portalinvestigacion.com/publicaciones11462
https://doi.org/10.1093/rheumatology/keaf556
Access Level:acceso abierto
Palavra-chave:DMARDs
biologics
cancer
epidemiology
registries
rheumatoid arthritis
targeted therapies
Descrição
Resumo:OBJECTIVES: To assess the risk of cancer in patients with rheumatoid arthritis (RA) treated with biologic and targeted synthetic disease-modifying antirheumatic drugs (b/tsDMARDs). METHODS: We analyzed RA patients from the BIOBADASER III registry, a multicentre national registry of b/tsDMARDs, included from 2000 to 2023. Patients with a history of cancer were excluded. Incidence rates (IRs) were analysed, and adjusted Cox regression models were used to estimate hazard ratios (HRs) for all cancers excluding non-melanoma skin cancer (NMSC), as well as for NMSC. Tumor necrosis factor inhibitors (TNFi) served as the reference for comparison. RESULTS: Among 4,635 RA patients (mean age 55.5 years; 79% female; median follow-up 3.6 years), 187 incident cancers were identified. For all cancers excluding NMSC, adjusted HRs (95% CI) compared with TNFi were: 1.2 (0.8-1.6) for IL6i, 0.9 (0.5-1.4) for CD20i, 1.2 (0.8-1.8) for JAKi, and 1.1 (0.8-1.6) for CTLA4-A. For NMSC, adjusted HRs (95% CI) were 0.6 (0.2-1.5) for IL6i, 0.6 (0.2-1.8) for CD20i, 0.7 (0.2-2) for JAKi, and 1.1 (0.5-2.6) for CTLA4-A. No increased cancer risk was observed when adjusting for cardiovascular risk or treatment line, for either cancer type. CONCLUSIONS: In this large, real-world cohort of RA patients, we found no increased cancer risk associated with any bDMARDs or tsDMARDs compared with TNFi.