Kv1.3 and Kv1.5 channels in leukocytes

Voltage dependent potassium channels are a group of plasma membrane ion channels with a key role in the immune system as the predominant ion channels controlling the resting membrane potential and tuning intracellular Ca2+ signaling in lymphocytes, monocytes, macrophages, and dendritic cells. Leukoc...

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Detalles Bibliográficos
Autor: Vallejo Gracia, Albert
Tipo de recurso: tesis doctoral
Estado:Versión publicada
Fecha de publicación:2016
País:España
Institución:CBUC, CESCA
Repositorio:TDR. Tesis Doctorales en Red
OAI Identifier:oai:www.tdx.cat:10803/397797
Acceso en línea:http://hdl.handle.net/10803/397797
Access Level:acceso abierto
Palabra clave:Leucòcits
Leucocitos
Leucocytes
Càncer
Cáncer
Cancer
Fisiologia cel·lular
Fisiología celular
Cell physiology
Canals de potassi
Canales de potasio
Potassium channels
Ciències Experimentals i Matemàtiques
577
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spelling Kv1.3 and Kv1.5 channels in leukocytesVallejo Gracia, AlbertLeucòcitsLeucocitosLeucocytesCàncerCáncerCancerFisiologia cel·lularFisiología celularCell physiologyCanals de potassiCanales de potasioPotassium channelsCiències Experimentals i Matemàtiques577Voltage dependent potassium channels are a group of plasma membrane ion channels with a key role in the immune system as the predominant ion channels controlling the resting membrane potential and tuning intracellular Ca2+ signaling in lymphocytes, monocytes, macrophages, and dendritic cells. Leukocytes present a limited Kv repertoire, including Kv1.3 and Kv1.5 channel isoforms. Kv1.3 is expressed in the immune system, and the blockade of this channel is associated with selective inhibition of T cell activation and proliferation. A functional Kv channel is an oligomeric complex composed of pore-forming and ancillary subunits. The KCNE gene family (KCNE1-5) is a novel group of modulatory Kv channel elements expressed in several tissues including leukocytes. KCNE peptides are small single spanning membrane proteins known to modulate Kv channels trafficking and biophysical properties. The hypothesis of the present PhD thesis entitled “Kv1.3 and Kv1.5 channels in leukocytes” was that changes in the channelosome composition by modulating the heterooligomeric combinations of the Kv1.3 channelosome control physiological and neoplastic cell growth as well as leukocyte responses. Evidence suggests that Kv channels are involved in cell differentiation and cell cycle control (because non-specific drugs, such as 4-AP and TEA, impaire proliferation), and they are also known to be remodeled during carcinogenesis. Thus, we elucidated the role of Kv1.3 and Kv1.5 channels in cell growth and their relationship with cancer, in models such as B lymphocytes and lymphomas (non-Hodgkin lymphomas), pancreatic ductal adenocarcinoma (PDAC) and glioblastomas. In spite of its significance, the mechanisms that regulate Kv1.3 and its role in the T cell activation are not well known. To that end, we analyzed the expression of KCNEs ancillary subunits upon different states of activation and proliferation of leukocytes (macrophages, T and B lymphocytes). In addition, recent data from our laboratory demonstrate that KCNE4, acting as a dominant negative ancillary subunit, physically interacts with Kv1.3 inhibiting K+ currents and retaining the channel intracellularly. Therefore, we studied the Kv1.3 modulation by the auxiliary subunit KCNE4 in leukocytes.Universitat de BarcelonaFelipe Campo, AntonioUniversitat de Barcelona. Departament de Bioquímica i Biologia Molecular (Biologia)201620172016info:eu-repo/semantics/doctoralThesisinfo:eu-repo/semantics/publishedVersion142 p.application/pdfapplication/pdfhttp://hdl.handle.net/10803/397797TDX (Tesis Doctorals en Xarxa)reponame:TDR. Tesis Doctorales en Redinstname:CBUC, CESCAInglésADVERTIMENT. L'accés als continguts d'aquesta tesi doctoral i la seva utilització ha de respectar els drets de la persona autora. Pot ser utilitzada per a consulta o estudi personal, així com en activitats o materials d'investigació i docència en els termes establerts a l'art. 32 del Text Refós de la Llei de Propietat Intel·lectual (RDL 1/1996). Per altres utilitzacions es requereix l'autorització prèvia i expressa de la persona autora. En qualsevol cas, en la utilització dels seus continguts caldrà indicar de forma clara el nom i cognoms de la persona autora i el títol de la tesi doctoral. No s'autoritza la seva reproducció o altres formes d'explotació efectuades amb finalitats de lucre ni la seva comunicació pública des d'un lloc aliè al servei TDX. Tampoc s'autoritza la presentació del seu contingut en una finestra o marc aliè a TDX (framing). Aquesta reserva de drets afecta tant als continguts de la tesi com als seus resums i índexs.info:eu-repo/semantics/openAccessoai:www.tdx.cat:10803/3977972026-06-14T12:46:07Z
dc.title.none.fl_str_mv Kv1.3 and Kv1.5 channels in leukocytes
title Kv1.3 and Kv1.5 channels in leukocytes
spellingShingle Kv1.3 and Kv1.5 channels in leukocytes
Vallejo Gracia, Albert
Leucòcits
Leucocitos
Leucocytes
Càncer
Cáncer
Cancer
Fisiologia cel·lular
Fisiología celular
Cell physiology
Canals de potassi
Canales de potasio
Potassium channels
Ciències Experimentals i Matemàtiques
577
title_short Kv1.3 and Kv1.5 channels in leukocytes
title_full Kv1.3 and Kv1.5 channels in leukocytes
title_fullStr Kv1.3 and Kv1.5 channels in leukocytes
title_full_unstemmed Kv1.3 and Kv1.5 channels in leukocytes
title_sort Kv1.3 and Kv1.5 channels in leukocytes
dc.creator.none.fl_str_mv Vallejo Gracia, Albert
author Vallejo Gracia, Albert
author_facet Vallejo Gracia, Albert
author_role author
dc.contributor.none.fl_str_mv Felipe Campo, Antonio
Universitat de Barcelona. Departament de Bioquímica i Biologia Molecular (Biologia)
dc.subject.none.fl_str_mv Leucòcits
Leucocitos
Leucocytes
Càncer
Cáncer
Cancer
Fisiologia cel·lular
Fisiología celular
Cell physiology
Canals de potassi
Canales de potasio
Potassium channels
Ciències Experimentals i Matemàtiques
577
topic Leucòcits
Leucocitos
Leucocytes
Càncer
Cáncer
Cancer
Fisiologia cel·lular
Fisiología celular
Cell physiology
Canals de potassi
Canales de potasio
Potassium channels
Ciències Experimentals i Matemàtiques
577
description Voltage dependent potassium channels are a group of plasma membrane ion channels with a key role in the immune system as the predominant ion channels controlling the resting membrane potential and tuning intracellular Ca2+ signaling in lymphocytes, monocytes, macrophages, and dendritic cells. Leukocytes present a limited Kv repertoire, including Kv1.3 and Kv1.5 channel isoforms. Kv1.3 is expressed in the immune system, and the blockade of this channel is associated with selective inhibition of T cell activation and proliferation. A functional Kv channel is an oligomeric complex composed of pore-forming and ancillary subunits. The KCNE gene family (KCNE1-5) is a novel group of modulatory Kv channel elements expressed in several tissues including leukocytes. KCNE peptides are small single spanning membrane proteins known to modulate Kv channels trafficking and biophysical properties. The hypothesis of the present PhD thesis entitled “Kv1.3 and Kv1.5 channels in leukocytes” was that changes in the channelosome composition by modulating the heterooligomeric combinations of the Kv1.3 channelosome control physiological and neoplastic cell growth as well as leukocyte responses. Evidence suggests that Kv channels are involved in cell differentiation and cell cycle control (because non-specific drugs, such as 4-AP and TEA, impaire proliferation), and they are also known to be remodeled during carcinogenesis. Thus, we elucidated the role of Kv1.3 and Kv1.5 channels in cell growth and their relationship with cancer, in models such as B lymphocytes and lymphomas (non-Hodgkin lymphomas), pancreatic ductal adenocarcinoma (PDAC) and glioblastomas. In spite of its significance, the mechanisms that regulate Kv1.3 and its role in the T cell activation are not well known. To that end, we analyzed the expression of KCNEs ancillary subunits upon different states of activation and proliferation of leukocytes (macrophages, T and B lymphocytes). In addition, recent data from our laboratory demonstrate that KCNE4, acting as a dominant negative ancillary subunit, physically interacts with Kv1.3 inhibiting K+ currents and retaining the channel intracellularly. Therefore, we studied the Kv1.3 modulation by the auxiliary subunit KCNE4 in leukocytes.
publishDate 2016
dc.date.none.fl_str_mv 2016
2016
2017
dc.type.none.fl_str_mv info:eu-repo/semantics/doctoralThesis
info:eu-repo/semantics/publishedVersion
format doctoralThesis
status_str publishedVersion
dc.identifier.none.fl_str_mv http://hdl.handle.net/10803/397797
url http://hdl.handle.net/10803/397797
dc.language.none.fl_str_mv Inglés
language_invalid_str_mv Inglés
dc.rights.none.fl_str_mv info:eu-repo/semantics/openAccess
eu_rights_str_mv openAccess
dc.format.none.fl_str_mv 142 p.
application/pdf
application/pdf
dc.publisher.none.fl_str_mv Universitat de Barcelona
publisher.none.fl_str_mv Universitat de Barcelona
dc.source.none.fl_str_mv TDX (Tesis Doctorals en Xarxa)
reponame:TDR. Tesis Doctorales en Red
instname:CBUC, CESCA
instname_str CBUC, CESCA
reponame_str TDR. Tesis Doctorales en Red
collection TDR. Tesis Doctorales en Red
repository.name.fl_str_mv
repository.mail.fl_str_mv
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score 15,300724