The SGLT2 Inhibitor Empagliflozin Ameliorates the Inflammatory Profile in Type 2 Diabetic Patients and Promotes an Antioxidant Response in Leukocytes

[EN]Sodium-glucose co-transporter 2 inhibitors (iSGLT2) have been linked to a considerable reduction in cardiovascular risk in patients with type 2 diabetes (T2D), but the precise molecular mechanisms are still elusive. We aimed to evaluate the effects of the iSGLT2 empagliflozin on systemic inflamm...

ver descrição completa

Detalhes bibliográficos
Autores: Iannantuoni, Francesca, M. de Marañon, Aranzazu, Díaz Morales, Noelia, Falcon, Rosa, Bañuls, Celia, Abad-Jimenez, Zaida, Victor, Victor M., Hernandez-Mijares, Antonio, Rovira-Llopis, Susana
Formato: artículo
Estado:Versión publicada
Fecha de publicación:2019
País:España
Recursos:Universidad de Salamanca (USAL)
Repositorio:GREDOS. Repositorio Institucional de la Universidad de Salamanca
OAI Identifier:oai:gredos.usal.es:10366/169708
Acesso em linha:http://hdl.handle.net/10366/169708
Access Level:acceso abierto
Palavra-chave:Empagliflozina (Inhibidor de SGLT2)
Diabetes tipo 2
Inflamación
Estrés oxidativo
Leucocitos
Respuesta antioxidante
Empagliflozin (SGLT2 inhibitor)
Type 2 diabetes
Inflammation
Oxidative stress
Leukocytes
Antioxidant response
Diabetes Mellitus, Type 2
Oxidative Stress
3209 Farmacología
2403 Bioquímica
2411 Fisiología Humana
2411.04 Fisiología Endocrina
2407 Biología Celular
diabetes mellitus tipo II
estrés oxidativo
inflamación
leucocitos
Descrição
Resumo:[EN]Sodium-glucose co-transporter 2 inhibitors (iSGLT2) have been linked to a considerable reduction in cardiovascular risk in patients with type 2 diabetes (T2D), but the precise molecular mechanisms are still elusive. We aimed to evaluate the effects of the iSGLT2 empagliflozin on systemic inflammation and its potential antioxidant properties. This is an observational, prospective follow-up study of a cohort of fifteen patients with T2D who received 10 mg/day of empagliflozin according to standard clinical care. Measures at baseline, 12 and 24 weeks were taken. Metabolic and anthropometric parameters were evaluated. Production of mitochondrial superoxide, glutathione content, and glutathione s-reductase and catalase mRNA levels were measured in leukocytes. Serum levels of myeloperoxidase, hs-CRP and IL-10 were determined. In addition to decreased body weight and reduced glucose and HbA1c levels, we observed a reduction in superoxide production in leukocytes of diabetic patients and increased glutathione content, prominently after 24 weeks of empagliflozin treatment. Leukocyte expression of glutathione s-reductase and catalase, and serum levels of IL-10 were enhanced at 24 weeks of empagliflozin treatment. Concomitantly, reduced hs-CRP and myeloperoxidase levels were seen. This study provides evidence of the antioxidant and anti-inflammatory properties of empagliflozin treatment in humans, which may contribute to its beneficial cardiovascular effects.