Potential Role of Circulating PD-L1+ Leukocytes as a Predictor of Response to Anti-PD-(L)1 Therapy in NSCLC Patients

PD-(L)1 inhibitors are part of the treatment strategy for non-small cell lung cancer (NSCLC) although its efficacy is limited to certain patients. Our study aimed to identify patients who might benefit from anti-PD-(L)1 inhibitors by analyzing the PD-L1 expression on circulating leukocytes and its e...

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Detalles Bibliográficos
Autores: Anguera, Georgia|||0000-0002-6060-1110, Mulet Gual, Maria|||0000-0003-3639-3291, Zamora, Carlos|||0000-0003-2962-678X, Osuna Gómez, Rubén|||0000-0003-2875-4405, Barba Joaquin, Andrés|||0000-0003-2500-0253, Sullivan, Ivana|||0000-0002-0434-3436, Serra, Jorgina|||0000-0002-2363-7149, Cantó, Elisabet|||0000-0002-1782-1855, Vidal, Silvia|||0000-0002-3909-6682, Majem Tarruella, Margarita|||0000-0002-9919-7485
Tipo de recurso: artículo
Fecha de publicación:2024
País:España
Institución:Universitat Autònoma de Barcelona
Repositorio:Dipòsit Digital de Documents de la UAB
Idioma:inglés
OAI Identifier:oai:ddd.uab.cat:293371
Acceso en línea:https://ddd.uab.cat/record/293371
https://dx.doi.org/urn:doi:10.3390/biomedicines12050958
Access Level:acceso abierto
Palabra clave:NSCLC
PD-L1+ CD14+ cells
PD-L1+ neutrophils
PDL-1+ platelets
Immunotherapy
Descripción
Sumario:PD-(L)1 inhibitors are part of the treatment strategy for non-small cell lung cancer (NSCLC) although its efficacy is limited to certain patients. Our study aimed to identify patients who might benefit from anti-PD-(L)1 inhibitors by analyzing the PD-L1 expression on circulating leukocytes and its evolution during treatment. One hundred thirteen NSCLC patients, according to their radiological response after 10-12 weeks of treatment, were classified into responders, stable, and progressive disease. Percentages of circulating PD-L1 leukocytes, PD-L1 platelets (PLTs), and leukocyte-PLT complexes were assessed using flow cytometry, and plasma concentrations of soluble immunomodulatory factors were quantified by ELISA. Responders exhibited significantly higher pre-treatment percentages of PD-L1 neutrophils, PD-L1 CD14 cells, and PD-L1 PLTs than progressors. The percentages of these populations decreased in responders post-treatment, contrasting with stables and progressors. PLTs notably contributed to PD-L1 expression in CD14 cells and neutrophils. Plasma cytokine analysis revealed baseline differences only in IL-17 concentration among groups, whereas network analyses highlighted distinct association patterns between plasma molecules and PD-L1 leukocytes after 10-12 weeks of treatment. Our findings suggest that pre-treatment assessment of circulating PD-L1 neutrophils, PD-L1 CD14 cells, and PD-L1 PLTs may be helpful in identifying NSCLC patients who are potential candidates for anti-PD-(L)1 therapy.