Recent advances in the targeting of epigenetic regulators in b-cell non-hodgkin lymphoma

In the last 10 years, major advances have been made in the diagnosis and development of selective therapies for several blood cancers, including B-cell non-Hodgkin lymphoma (B-NHL), a heterogeneous group of malignancies arising from the mature B lymphocyte compartment. However, most of these entitie...

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Autores: Ribeiro, Marcelo L.|||0000-0003-4529-7832, Reyes Garau, Diana|||0000-0002-6124-8953, Armengol, Marc Antoni|||0000-0001-5206-9139, Roué, Gaël|||0000-0003-0245-2257
Formato: artículo
Fecha de publicación:2019
País:España
Recursos:Universitat Autònoma de Barcelona
Repositorio:Dipòsit Digital de Documents de la UAB
Idioma:inglés
OAI Identifier:oai:ddd.uab.cat:223420
Acesso em linha:https://ddd.uab.cat/record/223420
https://dx.doi.org/urn:doi:10.3389/fgene.2019.00986
Access Level:acceso abierto
Palavra-chave:B-cell lymphoma
DNMT
EZH2
HDAC
PRMT inhibitor
BET bromodomain inhibitor (BETi)
Combination therapy
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spelling Recent advances in the targeting of epigenetic regulators in b-cell non-hodgkin lymphomaRibeiro, Marcelo L.|||0000-0003-4529-7832Reyes Garau, Diana|||0000-0002-6124-8953Armengol, Marc Antoni|||0000-0001-5206-9139Roué, Gaël|||0000-0003-0245-2257B-cell lymphomaDNMTEZH2HDACPRMT inhibitorBET bromodomain inhibitor (BETi)Combination therapyIn the last 10 years, major advances have been made in the diagnosis and development of selective therapies for several blood cancers, including B-cell non-Hodgkin lymphoma (B-NHL), a heterogeneous group of malignancies arising from the mature B lymphocyte compartment. However, most of these entities remain incurable and current treatments are associated with variable efficacy, several adverse events, and frequent relapses. Thus, new diagnostic paradigms and novel therapeutic options are required to improve the prognosis of patients with B-NHL. With the recent deciphering of the mutational landscapes of B-cell disorders by high-throughput sequencing, it came out that different epigenetic deregulations might drive and/or promote B lymphomagenesis. Consistently, over the last decade, numerous epigenetic drugs (or epidrugs) have emerged in the clinical management of B-NHL patients. In this review, we will present an overview of the most relevant epidrugs tested and/or used so far for the treatment of different subtypes of B-NHL, from first-generation epigenetic therapies like histone acetyl transferases (HDACs) or DNA-methyl transferases (DNMTs) inhibitors to new agents showing selectivity for proteins that are mutated, translocated, and/or overexpressed in these diseases, including EZH2, BET, and PRMT. We will dissect the mechanisms of action of these epigenetic inhibitors, as well as the molecular processes underlying their lack of efficacy in refractory patients. This review will also provide a summary of the latest strategies being employed in preclinical and clinical settings, and will point out the most promising lines of investigation in the field.Universitat Autònoma de Barcelona 22019-01-0120192019-01-01Article de revisióhttp://purl.org/coar/resource_type/c_dcae04bcVoRhttp://purl.org/coar/version/c_970fb48d4fbd8a85info:eu-repo/semantics/articleapplication/pdfhttps://ddd.uab.cat/record/223420https://dx.doi.org/urn:doi:10.3389/fgene.2019.00986reponame:Dipòsit Digital de Documents de la UABinstname:Universitat Autònoma de BarcelonaInglésengInstituto de Salud Carlos III https://doi.org/10.13039/501100004587 PI18-01383Instituto de Salud Carlos III https://doi.org/10.13039/501100004587 PI15-00102open accesshttp://purl.org/coar/access_right/c_abf2Aquest document està subjecte a una llicència d'ús Creative Commons. Es permet la reproducció total o parcial, la distribució, la comunicació pública de l'obra i la creació d'obres derivades, fins i tot amb finalitats comercials, sempre i quan es reconegui l'autoria de l'obra original.https://creativecommons.org/licenses/by/4.0/info:eu-repo/semantics/openAccessoai:ddd.uab.cat:2234202026-06-06T12:50:31Z
dc.title.none.fl_str_mv Recent advances in the targeting of epigenetic regulators in b-cell non-hodgkin lymphoma
title Recent advances in the targeting of epigenetic regulators in b-cell non-hodgkin lymphoma
spellingShingle Recent advances in the targeting of epigenetic regulators in b-cell non-hodgkin lymphoma
Ribeiro, Marcelo L.|||0000-0003-4529-7832
B-cell lymphoma
DNMT
EZH2
HDAC
PRMT inhibitor
BET bromodomain inhibitor (BETi)
Combination therapy
title_short Recent advances in the targeting of epigenetic regulators in b-cell non-hodgkin lymphoma
title_full Recent advances in the targeting of epigenetic regulators in b-cell non-hodgkin lymphoma
title_fullStr Recent advances in the targeting of epigenetic regulators in b-cell non-hodgkin lymphoma
title_full_unstemmed Recent advances in the targeting of epigenetic regulators in b-cell non-hodgkin lymphoma
title_sort Recent advances in the targeting of epigenetic regulators in b-cell non-hodgkin lymphoma
dc.creator.none.fl_str_mv Ribeiro, Marcelo L.|||0000-0003-4529-7832
Reyes Garau, Diana|||0000-0002-6124-8953
Armengol, Marc Antoni|||0000-0001-5206-9139
Roué, Gaël|||0000-0003-0245-2257
author Ribeiro, Marcelo L.|||0000-0003-4529-7832
author_facet Ribeiro, Marcelo L.|||0000-0003-4529-7832
Reyes Garau, Diana|||0000-0002-6124-8953
Armengol, Marc Antoni|||0000-0001-5206-9139
Roué, Gaël|||0000-0003-0245-2257
author_role author
author2 Reyes Garau, Diana|||0000-0002-6124-8953
Armengol, Marc Antoni|||0000-0001-5206-9139
Roué, Gaël|||0000-0003-0245-2257
author2_role author
author
author
dc.contributor.none.fl_str_mv Universitat Autònoma de Barcelona
dc.subject.none.fl_str_mv B-cell lymphoma
DNMT
EZH2
HDAC
PRMT inhibitor
BET bromodomain inhibitor (BETi)
Combination therapy
topic B-cell lymphoma
DNMT
EZH2
HDAC
PRMT inhibitor
BET bromodomain inhibitor (BETi)
Combination therapy
description In the last 10 years, major advances have been made in the diagnosis and development of selective therapies for several blood cancers, including B-cell non-Hodgkin lymphoma (B-NHL), a heterogeneous group of malignancies arising from the mature B lymphocyte compartment. However, most of these entities remain incurable and current treatments are associated with variable efficacy, several adverse events, and frequent relapses. Thus, new diagnostic paradigms and novel therapeutic options are required to improve the prognosis of patients with B-NHL. With the recent deciphering of the mutational landscapes of B-cell disorders by high-throughput sequencing, it came out that different epigenetic deregulations might drive and/or promote B lymphomagenesis. Consistently, over the last decade, numerous epigenetic drugs (or epidrugs) have emerged in the clinical management of B-NHL patients. In this review, we will present an overview of the most relevant epidrugs tested and/or used so far for the treatment of different subtypes of B-NHL, from first-generation epigenetic therapies like histone acetyl transferases (HDACs) or DNA-methyl transferases (DNMTs) inhibitors to new agents showing selectivity for proteins that are mutated, translocated, and/or overexpressed in these diseases, including EZH2, BET, and PRMT. We will dissect the mechanisms of action of these epigenetic inhibitors, as well as the molecular processes underlying their lack of efficacy in refractory patients. This review will also provide a summary of the latest strategies being employed in preclinical and clinical settings, and will point out the most promising lines of investigation in the field.
publishDate 2019
dc.date.none.fl_str_mv 2
2019-01-01
2019
2019-01-01
dc.type.none.fl_str_mv Article de revisió
http://purl.org/coar/resource_type/c_dcae04bc
VoR
http://purl.org/coar/version/c_970fb48d4fbd8a85
dc.type.openaire.fl_str_mv info:eu-repo/semantics/article
format article
dc.identifier.none.fl_str_mv https://ddd.uab.cat/record/223420
https://dx.doi.org/urn:doi:10.3389/fgene.2019.00986
url https://ddd.uab.cat/record/223420
https://dx.doi.org/urn:doi:10.3389/fgene.2019.00986
dc.language.none.fl_str_mv Inglés
eng
language_invalid_str_mv Inglés
language eng
dc.relation.none.fl_str_mv Instituto de Salud Carlos III https://doi.org/10.13039/501100004587 PI18-01383
Instituto de Salud Carlos III https://doi.org/10.13039/501100004587 PI15-00102
dc.rights.none.fl_str_mv open access
http://purl.org/coar/access_right/c_abf2
https://creativecommons.org/licenses/by/4.0/
dc.rights.openaire.fl_str_mv info:eu-repo/semantics/openAccess
rights_invalid_str_mv open access
http://purl.org/coar/access_right/c_abf2
https://creativecommons.org/licenses/by/4.0/
eu_rights_str_mv openAccess
dc.format.none.fl_str_mv application/pdf
dc.source.none.fl_str_mv reponame:Dipòsit Digital de Documents de la UAB
instname:Universitat Autònoma de Barcelona
instname_str Universitat Autònoma de Barcelona
reponame_str Dipòsit Digital de Documents de la UAB
collection Dipòsit Digital de Documents de la UAB
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repository.mail.fl_str_mv
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