Cyclin-Dependent Kinase Inhibitor p21 Controls Adult Neural Stem Cell Expansion by Regulating Sox2 Gene Expression

In the adult brain, continual neurogenesis of olfactory neurons is sustained by the existence of neural stem cells (NSCs) in the subependymal niche. Elimination of the cyclin-dependent kinase inhibitor 1A (p21) leads to premature exhaustion of the subependymal NSC pool, suggesting a relationship bet...

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Detalles Bibliográficos
Autores: Marques-Torrejon, MA, Porlan, E, Banito, A, Gomez Ibarlucea, Esther, Lopez-Contreras, AJ, Fernandez-Capetillo, O, Vidal Figueroa, Anxo, Gil, J, Torres, J, Farinas, I
Tipo de recurso: artículo
Fecha de publicación:2013
País:España
Institución:Servizo Galego de Saúde (SERGAS)
Repositorio:RUNA. Repositorio da Consellería de Sanidade e Sergas
OAI Identifier:oai:runa.sergas.gal:20.500.11940/3984
Acceso en línea:http://hdl.handle.net/20.500.11940/3984
Access Level:acceso abierto
Palabra clave:Adult Stem Cells
Animals
Cells, Cultured
Chromatin Immunoprecipitation
Cyclin-Dependent Kinase Inhibitor p21
Immunoblotting
Immunohistochemistry
Mice
Mice, Mutant Strains
Neural Stem Cells
Protein Binding
Reverse Transcriptase Polymerase Chain Reaction
SOXB1 Transcription Factors
Descripción
Sumario:In the adult brain, continual neurogenesis of olfactory neurons is sustained by the existence of neural stem cells (NSCs) in the subependymal niche. Elimination of the cyclin-dependent kinase inhibitor 1A (p21) leads to premature exhaustion of the subependymal NSC pool, suggesting a relationship between cell cycle control and long-term self-renewal, but the molecular mechanisms underlying NSC maintenance by p21 remain unexplored. Here we identify a function of p21 in the direct regulation of the expression of pluripotency factor Sox2, a key regulator of the specification and maintenance of neural progenitors. We observe that p21 directly binds a Sox2 enhancer and negatively regulates Sox2 expression in NSCs. Augmented levels of Sox2 in p21 null cells induce replicative stress and a DNA damage response that leads to cell growth arrest mediated by increased levels of p19(Arf) and p53. Our results show a regulation of NSC expansion driven by a p21/Sox2/p53 axis.