Aptamer-peptide conjugates as a new strategy to modulate human α-thrombin binding affinity

Aptamers are single-stranded RNA or DNA molecules that specifically recognize their targets and have proven valuable for functionalizing sensitive biosensors. α-thrombin is a trypsin-like serine proteinase which plays a crucial role in haemostasis and thrombosis. An abnormal activity or overexpressi...

Descripción completa

Detalles Bibliográficos
Autores: Aviñó, Ana|||0000-0003-3047-738X, Jorge, Andreia F., Huertas, César S.|||0000-0002-3100-4034, Cova, Tania F. G. G., Pais, Alberto, Lechuga, Laura M.|||0000-0001-5187-5358, Eritja, Ramon|||0000-0001-5383-9334, Fabrega, Carme|||0000-0003-3816-7634
Tipo de recurso: artículo
Fecha de publicación:2019
País:España
Institución:Universitat Autònoma de Barcelona
Repositorio:Dipòsit Digital de Documents de la UAB
Idioma:inglés
OAI Identifier:oai:ddd.uab.cat:214183
Acceso en línea:https://ddd.uab.cat/record/214183
https://dx.doi.org/urn:doi:10.1016/j.bbagen.2019.06.014
Access Level:acceso abierto
Palabra clave:Thrombin
Biosensor
Binding affinity
Inhibition
Aptamer-peptide conjugate
Descripción
Sumario:Aptamers are single-stranded RNA or DNA molecules that specifically recognize their targets and have proven valuable for functionalizing sensitive biosensors. α-thrombin is a trypsin-like serine proteinase which plays a crucial role in haemostasis and thrombosis. An abnormal activity or overexpression of this protein is associated with a variety of diseases. A great deal of attention was devoted to the construction of high-throughput biosensors for accurately detect thrombin for the early diagnosis and treatment of related diseases. Herein, we propose a new approach to modulate the interaction between α-thrombin and the aptamer TBA. To this end, TBA was chemically conjugated to two peptide sequences (TBA-GFIE-Ac and TBA-GEIF-Ac) corresponding to a short fragment of the acidic region of the human factor V, which is known to interact directly with exosite I. Surface Plasmon Resonance (SPR) results showed enhanced analytical performances of thrombin with TBA-GEIF-Ac than with TBA wild-type, reaching a limit of detection as low as 44.9 pM. Electrophoresis mobility shift assay (EMSA) corroborated the SPR results. Molecular dynamics (MD) simulations support experimental evidences and provided further insight into thrombin/TBA-peptide interaction. Our findings demonstrate that the combination of TBA with key interacting peptides offers good opportunities to produce sensitive devices for thrombin detection and potential candidates to block thrombin activity.