Association study of human leukocyte antigen variants and idiopathic pulmonary fibrosis

Introduction Idiopathic pulmonary fibrosis (IPF) is a chronic interstitial pneumonia marked by progressive lung fibrosis and a poor prognosis. Recent studies have highlighted the potential role of infection in the pathogenesis of IPF, and a prior association of the HLA-DQB1 gene with idiopathic fibr...

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Autores: Guillén Guió, Beatriz, Paynton, Megan L., Allen, Richard J., Chin, Daniel P. W., Donoghue, Lauren J., Stockwell, Amy, Leavy, Olivia C., Hernández Beeftink, Tamara, Reynolds, Carl, Cullinan, Paul, Martínez, Fernando, Booth, Helen L., Fahy, William A., Hall, Ian P., Hart, Simon P., Hill, Mike R., Hirani, Nik, Hubbard, Richard B., Mcanulty, Robin J., Millar, Ann B., Navaratnam, Vidya, Oballa, Eunice, Parfrey, Helen, Saini, Gauri, Sayers, Ian, Tobin, Martin D., Whyte, Moira K. B., Adegunsoye, Ayodeji, Kaminski, Naftali, Ma, Shwu-Fan, Strek, Mary E., Zhang, Yingze, Fingerlin, Tasha E., Molina Molina, María, Neighbors, Margaret, Sheng, X. Rebecca, Oldham, Justin M., Maher, Toby M., Molyneaux, Philip L., Flores, Carlos, Noth, Imre, Schwartz, David A., Yaspan, Brian L., Jenkins, R. Gisli, Wain, Louise V., Hollox, Edward J.
Tipo de recurso: artículo
Estado:Versión publicada
Fecha de publicación:2023
País:España
Institución:Universidad de Barcelona
Repositorio:Dipòsit Digital de la UB
OAI Identifier:oai:diposit.ub.edu:2445/212227
Acceso en línea:https://hdl.handle.net/2445/212227
Access Level:acceso abierto
Palabra clave:Fibrosi pulmonar
Antígens CD
Pulmonary fibrosis
CD antigens
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spelling Association study of human leukocyte antigen variants and idiopathic pulmonary fibrosisGuillén Guió, BeatrizPaynton, Megan L.Allen, Richard J.Chin, Daniel P. W.Donoghue, Lauren J.Stockwell, AmyLeavy, Olivia C.Hernández Beeftink, TamaraReynolds, CarlCullinan, PaulMartínez, FernandoBooth, Helen L.Fahy, William A.Hall, Ian P.Hart, Simon P.Hill, Mike R.Hirani, NikHubbard, Richard B.Mcanulty, Robin J.Millar, Ann B.Navaratnam, VidyaOballa, EuniceParfrey, HelenSaini, GauriSayers, IanTobin, Martin D.Whyte, Moira K. B.Adegunsoye, AyodejiKaminski, NaftaliMa, Shwu-FanStrek, Mary E.Zhang, YingzeFingerlin, Tasha E.Molina Molina, MaríaNeighbors, MargaretSheng, X. RebeccaOldham, Justin M.Maher, Toby M.Molyneaux, Philip L.Flores, CarlosNoth, ImreSchwartz, David A.Yaspan, Brian L.Jenkins, R. GisliWain, Louise V.Hollox, Edward J.Fibrosi pulmonarAntígens CDPulmonary fibrosisCD antigensIntroduction Idiopathic pulmonary fibrosis (IPF) is a chronic interstitial pneumonia marked by progressive lung fibrosis and a poor prognosis. Recent studies have highlighted the potential role of infection in the pathogenesis of IPF, and a prior association of the HLA-DQB1 gene with idiopathic fibrotic interstitial pneumonia (including IPF) has been reported. Owing to the important role that the human leukocyte antigen (HLA) region plays in the immune response, here we evaluated if HLA genetic variation was associated specifically with IPF risk. Methods We performed a meta-analysis of associations of the HLA region with IPF risk in individuals of European ancestry from seven independent case-control studies of IPF (comprising 5159 cases and 27 459 controls, including a prior study of fibrotic interstitial pneumonia). Single nucleotide polymorphisms, classical HLA alleles and amino acids were analysed and signals meeting a region-wide association threshold of p<4.5x10-4 and a posterior probability of replication >90% were considered significant. We sought to replicate the previously reported HLA-DQB1 association in the subset of studies independent of the original report. Results The meta-analysis of all seven studies identified four significant independent single nucleotide polymorphisms associated with IPF risk. However, none met the posterior probability for replication criterion. The HLA-DQB1 association was not replicated in the independent IPF studies. Conclusion Variation in the HLA region was not consistently associated with risk in studies of IPF. However, this does not preclude the possibility that other genomic regions linked to the immune response may be involved in the aetiology of IPF.European Respiratory Society (ERS)2023info:eu-repo/semantics/articleinfo:eu-repo/semantics/publishedVersionapplication/pdfhttps://hdl.handle.net/2445/212227Articles publicats en revistes (Institut d'lnvestigació Biomèdica de Bellvitge (IDIBELL))reponame:Dipòsit Digital de la UBinstname:Universidad de BarcelonaInglésReproducció del document publicat a: https://doi.org/10.1183/23120541.00553-2023ERJ Open Research, 2023, vol. 10, num. 1https://doi.org/10.1183/23120541.00553-2023cc by (c) Guillén Guió, Beatriz et al, 2023http://creativecommons.org/licenses/by/3.0/es/info:eu-repo/semantics/openAccessoai:diposit.ub.edu:2445/2122272026-05-27T06:46:51Z
dc.title.none.fl_str_mv Association study of human leukocyte antigen variants and idiopathic pulmonary fibrosis
title Association study of human leukocyte antigen variants and idiopathic pulmonary fibrosis
spellingShingle Association study of human leukocyte antigen variants and idiopathic pulmonary fibrosis
Guillén Guió, Beatriz
Fibrosi pulmonar
Antígens CD
Pulmonary fibrosis
CD antigens
title_short Association study of human leukocyte antigen variants and idiopathic pulmonary fibrosis
title_full Association study of human leukocyte antigen variants and idiopathic pulmonary fibrosis
title_fullStr Association study of human leukocyte antigen variants and idiopathic pulmonary fibrosis
title_full_unstemmed Association study of human leukocyte antigen variants and idiopathic pulmonary fibrosis
title_sort Association study of human leukocyte antigen variants and idiopathic pulmonary fibrosis
dc.creator.none.fl_str_mv Guillén Guió, Beatriz
Paynton, Megan L.
Allen, Richard J.
Chin, Daniel P. W.
Donoghue, Lauren J.
Stockwell, Amy
Leavy, Olivia C.
Hernández Beeftink, Tamara
Reynolds, Carl
Cullinan, Paul
Martínez, Fernando
Booth, Helen L.
Fahy, William A.
Hall, Ian P.
Hart, Simon P.
Hill, Mike R.
Hirani, Nik
Hubbard, Richard B.
Mcanulty, Robin J.
Millar, Ann B.
Navaratnam, Vidya
Oballa, Eunice
Parfrey, Helen
Saini, Gauri
Sayers, Ian
Tobin, Martin D.
Whyte, Moira K. B.
Adegunsoye, Ayodeji
Kaminski, Naftali
Ma, Shwu-Fan
Strek, Mary E.
Zhang, Yingze
Fingerlin, Tasha E.
Molina Molina, María
Neighbors, Margaret
Sheng, X. Rebecca
Oldham, Justin M.
Maher, Toby M.
Molyneaux, Philip L.
Flores, Carlos
Noth, Imre
Schwartz, David A.
Yaspan, Brian L.
Jenkins, R. Gisli
Wain, Louise V.
Hollox, Edward J.
author Guillén Guió, Beatriz
author_facet Guillén Guió, Beatriz
Paynton, Megan L.
Allen, Richard J.
Chin, Daniel P. W.
Donoghue, Lauren J.
Stockwell, Amy
Leavy, Olivia C.
Hernández Beeftink, Tamara
Reynolds, Carl
Cullinan, Paul
Martínez, Fernando
Booth, Helen L.
Fahy, William A.
Hall, Ian P.
Hart, Simon P.
Hill, Mike R.
Hirani, Nik
Hubbard, Richard B.
Mcanulty, Robin J.
Millar, Ann B.
Navaratnam, Vidya
Oballa, Eunice
Parfrey, Helen
Saini, Gauri
Sayers, Ian
Tobin, Martin D.
Whyte, Moira K. B.
Adegunsoye, Ayodeji
Kaminski, Naftali
Ma, Shwu-Fan
Strek, Mary E.
Zhang, Yingze
Fingerlin, Tasha E.
Molina Molina, María
Neighbors, Margaret
Sheng, X. Rebecca
Oldham, Justin M.
Maher, Toby M.
Molyneaux, Philip L.
Flores, Carlos
Noth, Imre
Schwartz, David A.
Yaspan, Brian L.
Jenkins, R. Gisli
Wain, Louise V.
Hollox, Edward J.
author_role author
author2 Paynton, Megan L.
Allen, Richard J.
Chin, Daniel P. W.
Donoghue, Lauren J.
Stockwell, Amy
Leavy, Olivia C.
Hernández Beeftink, Tamara
Reynolds, Carl
Cullinan, Paul
Martínez, Fernando
Booth, Helen L.
Fahy, William A.
Hall, Ian P.
Hart, Simon P.
Hill, Mike R.
Hirani, Nik
Hubbard, Richard B.
Mcanulty, Robin J.
Millar, Ann B.
Navaratnam, Vidya
Oballa, Eunice
Parfrey, Helen
Saini, Gauri
Sayers, Ian
Tobin, Martin D.
Whyte, Moira K. B.
Adegunsoye, Ayodeji
Kaminski, Naftali
Ma, Shwu-Fan
Strek, Mary E.
Zhang, Yingze
Fingerlin, Tasha E.
Molina Molina, María
Neighbors, Margaret
Sheng, X. Rebecca
Oldham, Justin M.
Maher, Toby M.
Molyneaux, Philip L.
Flores, Carlos
Noth, Imre
Schwartz, David A.
Yaspan, Brian L.
Jenkins, R. Gisli
Wain, Louise V.
Hollox, Edward J.
author2_role author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
dc.subject.none.fl_str_mv Fibrosi pulmonar
Antígens CD
Pulmonary fibrosis
CD antigens
topic Fibrosi pulmonar
Antígens CD
Pulmonary fibrosis
CD antigens
description Introduction Idiopathic pulmonary fibrosis (IPF) is a chronic interstitial pneumonia marked by progressive lung fibrosis and a poor prognosis. Recent studies have highlighted the potential role of infection in the pathogenesis of IPF, and a prior association of the HLA-DQB1 gene with idiopathic fibrotic interstitial pneumonia (including IPF) has been reported. Owing to the important role that the human leukocyte antigen (HLA) region plays in the immune response, here we evaluated if HLA genetic variation was associated specifically with IPF risk. Methods We performed a meta-analysis of associations of the HLA region with IPF risk in individuals of European ancestry from seven independent case-control studies of IPF (comprising 5159 cases and 27 459 controls, including a prior study of fibrotic interstitial pneumonia). Single nucleotide polymorphisms, classical HLA alleles and amino acids were analysed and signals meeting a region-wide association threshold of p<4.5x10-4 and a posterior probability of replication >90% were considered significant. We sought to replicate the previously reported HLA-DQB1 association in the subset of studies independent of the original report. Results The meta-analysis of all seven studies identified four significant independent single nucleotide polymorphisms associated with IPF risk. However, none met the posterior probability for replication criterion. The HLA-DQB1 association was not replicated in the independent IPF studies. Conclusion Variation in the HLA region was not consistently associated with risk in studies of IPF. However, this does not preclude the possibility that other genomic regions linked to the immune response may be involved in the aetiology of IPF.
publishDate 2023
dc.date.none.fl_str_mv 2023
dc.type.none.fl_str_mv info:eu-repo/semantics/article
info:eu-repo/semantics/publishedVersion
format article
status_str publishedVersion
dc.identifier.none.fl_str_mv https://hdl.handle.net/2445/212227
url https://hdl.handle.net/2445/212227
dc.language.none.fl_str_mv Inglés
language_invalid_str_mv Inglés
dc.relation.none.fl_str_mv Reproducció del document publicat a: https://doi.org/10.1183/23120541.00553-2023
ERJ Open Research, 2023, vol. 10, num. 1
https://doi.org/10.1183/23120541.00553-2023
dc.rights.none.fl_str_mv cc by (c) Guillén Guió, Beatriz et al, 2023
http://creativecommons.org/licenses/by/3.0/es/
info:eu-repo/semantics/openAccess
rights_invalid_str_mv cc by (c) Guillén Guió, Beatriz et al, 2023
http://creativecommons.org/licenses/by/3.0/es/
eu_rights_str_mv openAccess
dc.format.none.fl_str_mv application/pdf
dc.publisher.none.fl_str_mv European Respiratory Society (ERS)
publisher.none.fl_str_mv European Respiratory Society (ERS)
dc.source.none.fl_str_mv Articles publicats en revistes (Institut d'lnvestigació Biomèdica de Bellvitge (IDIBELL))
reponame:Dipòsit Digital de la UB
instname:Universidad de Barcelona
instname_str Universidad de Barcelona
reponame_str Dipòsit Digital de la UB
collection Dipòsit Digital de la UB
repository.name.fl_str_mv
repository.mail.fl_str_mv
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score 15,811543