Mendelian randomization analysis rules out disylipidaemia as colorectal cancer cause

[EN] Colorectal cancer (CRC) has been associated with both genetic and environmental risk factors such as diet1,2, alcohol3, smoking4, physical activity5 and metabolic syndrome6,7. Metabolic syndrome is a cluster of important cardiovascular risk factors: high fasting glucose, abdominal obesity, high...

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Autores: Ibáñez Sanz, Gemma, Díez Villanueva, Anna, Riera Ponsati, Marina, Fernández Villa, Tania, Fernández Navarro, Pablo, Bustamante, Mariona, Llorca, Javier, Amiano, Pilar, Ascunce, Nieves, Fernández Tardón, Guillermo, Salcedo Bellido, Inmaculada, Salas, Dolores, Capelo Álvarez, Rocío, Crous Bou, Marta, Ortega Valín, Luis, Pérez Gómez, Beatriz, Castaño Vinyals, Gemma, Palazuelos, Camilo, Altzibar, Jone M., Ardanaz, Eva, Tardón, Adonina, Jiménez Moleón, José Juan, Olmos Juste, Valle, Aragonés, Nuria, Pollán, Marina, Kogevinas, Manolis, Moreno González, Víctor
Tipo de recurso: artículo
Estado:Versión publicada
Fecha de publicación:2019
País:España
Institución:Universidad de León
Repositorio:BULERIA. Repositorio Institucional de la Universidad de León
OAI Identifier:oai:buleria.unileon.es:10612/22317
Acceso en línea:https://www.nature.com/articles/s41598-019-49880-w
https://hdl.handle.net/10612/22317
Access Level:acceso abierto
Palabra clave:Medicina. Salud
colorectal cancer
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spelling Mendelian randomization analysis rules out disylipidaemia as colorectal cancer causeIbáñez Sanz, GemmaDíez Villanueva, AnnaRiera Ponsati, MarinaFernández Villa, TaniaFernández Navarro, PabloBustamante, MarionaLlorca, JavierAmiano, PilarAscunce, NievesFernández Tardón, GuillermoSalcedo Bellido, InmaculadaSalas, DoloresCapelo Álvarez, RocíoCrous Bou, MartaOrtega Valín, LuisPérez Gómez, BeatrizCastaño Vinyals, GemmaPalazuelos, CamiloAltzibar, Jone M.Ardanaz, EvaTardón, AdoninaJiménez Moleón, José JuanOlmos Juste, ValleAragonés, NuriaPollán, MarinaKogevinas, ManolisMoreno González, VíctorMedicina. Saludcolorectal cancer[EN] Colorectal cancer (CRC) has been associated with both genetic and environmental risk factors such as diet1,2, alcohol3, smoking4, physical activity5 and metabolic syndrome6,7. Metabolic syndrome is a cluster of important cardiovascular risk factors: high fasting glucose, abdominal obesity, high triglycerides (TG), reduced high density lipoprotein cholesterol (HDL) and high blood pressure. As one of the components, dyslipidemia has been thought to have an important role in inflammatory pathways, oxidative stress and insulin resistance, which could contribute to the pathogenesis of cancer. However, findings from prospective studies that have examined the association between serum dyslipidemia (TG, HDL, low density lipoprotein cholesterol (LDL) or total cholesterol (TC)) and colorectal neoplasia have been inconsistent6,8,9,10,11. It is unknown whether lipids and lipoproteins cause cancer or are intermediate or correlated factors within carcinogenic pathways. The Mendelian randomization approach can be used to establish a causal relationship between dyslipidemia and CRC. Mendelian randomization studies use the distribution of alleles in the population to simulate randomized assignment to lower or higher lipids. A previous Mendelian randomization analysis assessing the causality of dyslipidemia and CRC has been published before11. It reported an association between a genetic score for TC and the risk of CRC (OR per unit SD increase = 1.46; 95% CI: 1.20–1.79) but no significant association was found for LDL, HDL or TG. While the association of TC was strong, the interpretation of the results is not straightforward, since TC is the sum of LDL and HDL cholesterol, fractions that have been reported to have opposite effects regarding CRC and may explain the controversial findings of studies that have analyzed the association between high levels of serum TC and CRC risk8,9,12. A causal effect of lipids regarding CRC risk should have a clear mechanistic interpretation. Being TC essentially the sum of HLD and LDL, genetic instruments for TC are correlated either with LDL, HDL or both, which violates the pleiotropy assumption of Mendelian randomization studies.SINature ResearchMedicina PreventivaFacultad de Ciencias de la Salud2019info:eu-repo/semantics/articleinfo:eu-repo/semantics/publishedVersionhttps://www.nature.com/articles/s41598-019-49880-whttps://hdl.handle.net/10612/22317reponame:BULERIA. Repositorio Institucional de la Universidad de Leóninstname:Universidad de LeónIngléshttp://creativecommons.org/licenses/by-nc-nd/4.0/info:eu-repo/semantics/openAccessoai:buleria.unileon.es:10612/223172026-06-24T12:43:27Z
dc.title.none.fl_str_mv Mendelian randomization analysis rules out disylipidaemia as colorectal cancer cause
title Mendelian randomization analysis rules out disylipidaemia as colorectal cancer cause
spellingShingle Mendelian randomization analysis rules out disylipidaemia as colorectal cancer cause
Ibáñez Sanz, Gemma
Medicina. Salud
colorectal cancer
title_short Mendelian randomization analysis rules out disylipidaemia as colorectal cancer cause
title_full Mendelian randomization analysis rules out disylipidaemia as colorectal cancer cause
title_fullStr Mendelian randomization analysis rules out disylipidaemia as colorectal cancer cause
title_full_unstemmed Mendelian randomization analysis rules out disylipidaemia as colorectal cancer cause
title_sort Mendelian randomization analysis rules out disylipidaemia as colorectal cancer cause
dc.creator.none.fl_str_mv Ibáñez Sanz, Gemma
Díez Villanueva, Anna
Riera Ponsati, Marina
Fernández Villa, Tania
Fernández Navarro, Pablo
Bustamante, Mariona
Llorca, Javier
Amiano, Pilar
Ascunce, Nieves
Fernández Tardón, Guillermo
Salcedo Bellido, Inmaculada
Salas, Dolores
Capelo Álvarez, Rocío
Crous Bou, Marta
Ortega Valín, Luis
Pérez Gómez, Beatriz
Castaño Vinyals, Gemma
Palazuelos, Camilo
Altzibar, Jone M.
Ardanaz, Eva
Tardón, Adonina
Jiménez Moleón, José Juan
Olmos Juste, Valle
Aragonés, Nuria
Pollán, Marina
Kogevinas, Manolis
Moreno González, Víctor
author Ibáñez Sanz, Gemma
author_facet Ibáñez Sanz, Gemma
Díez Villanueva, Anna
Riera Ponsati, Marina
Fernández Villa, Tania
Fernández Navarro, Pablo
Bustamante, Mariona
Llorca, Javier
Amiano, Pilar
Ascunce, Nieves
Fernández Tardón, Guillermo
Salcedo Bellido, Inmaculada
Salas, Dolores
Capelo Álvarez, Rocío
Crous Bou, Marta
Ortega Valín, Luis
Pérez Gómez, Beatriz
Castaño Vinyals, Gemma
Palazuelos, Camilo
Altzibar, Jone M.
Ardanaz, Eva
Tardón, Adonina
Jiménez Moleón, José Juan
Olmos Juste, Valle
Aragonés, Nuria
Pollán, Marina
Kogevinas, Manolis
Moreno González, Víctor
author_role author
author2 Díez Villanueva, Anna
Riera Ponsati, Marina
Fernández Villa, Tania
Fernández Navarro, Pablo
Bustamante, Mariona
Llorca, Javier
Amiano, Pilar
Ascunce, Nieves
Fernández Tardón, Guillermo
Salcedo Bellido, Inmaculada
Salas, Dolores
Capelo Álvarez, Rocío
Crous Bou, Marta
Ortega Valín, Luis
Pérez Gómez, Beatriz
Castaño Vinyals, Gemma
Palazuelos, Camilo
Altzibar, Jone M.
Ardanaz, Eva
Tardón, Adonina
Jiménez Moleón, José Juan
Olmos Juste, Valle
Aragonés, Nuria
Pollán, Marina
Kogevinas, Manolis
Moreno González, Víctor
author2_role author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
dc.contributor.none.fl_str_mv Medicina Preventiva
Facultad de Ciencias de la Salud
dc.subject.none.fl_str_mv Medicina. Salud
colorectal cancer
topic Medicina. Salud
colorectal cancer
description [EN] Colorectal cancer (CRC) has been associated with both genetic and environmental risk factors such as diet1,2, alcohol3, smoking4, physical activity5 and metabolic syndrome6,7. Metabolic syndrome is a cluster of important cardiovascular risk factors: high fasting glucose, abdominal obesity, high triglycerides (TG), reduced high density lipoprotein cholesterol (HDL) and high blood pressure. As one of the components, dyslipidemia has been thought to have an important role in inflammatory pathways, oxidative stress and insulin resistance, which could contribute to the pathogenesis of cancer. However, findings from prospective studies that have examined the association between serum dyslipidemia (TG, HDL, low density lipoprotein cholesterol (LDL) or total cholesterol (TC)) and colorectal neoplasia have been inconsistent6,8,9,10,11. It is unknown whether lipids and lipoproteins cause cancer or are intermediate or correlated factors within carcinogenic pathways. The Mendelian randomization approach can be used to establish a causal relationship between dyslipidemia and CRC. Mendelian randomization studies use the distribution of alleles in the population to simulate randomized assignment to lower or higher lipids. A previous Mendelian randomization analysis assessing the causality of dyslipidemia and CRC has been published before11. It reported an association between a genetic score for TC and the risk of CRC (OR per unit SD increase = 1.46; 95% CI: 1.20–1.79) but no significant association was found for LDL, HDL or TG. While the association of TC was strong, the interpretation of the results is not straightforward, since TC is the sum of LDL and HDL cholesterol, fractions that have been reported to have opposite effects regarding CRC and may explain the controversial findings of studies that have analyzed the association between high levels of serum TC and CRC risk8,9,12. A causal effect of lipids regarding CRC risk should have a clear mechanistic interpretation. Being TC essentially the sum of HLD and LDL, genetic instruments for TC are correlated either with LDL, HDL or both, which violates the pleiotropy assumption of Mendelian randomization studies.
publishDate 2019
dc.date.none.fl_str_mv 2019
dc.type.none.fl_str_mv info:eu-repo/semantics/article
info:eu-repo/semantics/publishedVersion
format article
status_str publishedVersion
dc.identifier.none.fl_str_mv https://www.nature.com/articles/s41598-019-49880-w
https://hdl.handle.net/10612/22317
url https://www.nature.com/articles/s41598-019-49880-w
https://hdl.handle.net/10612/22317
dc.language.none.fl_str_mv Inglés
language_invalid_str_mv Inglés
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info:eu-repo/semantics/openAccess
rights_invalid_str_mv http://creativecommons.org/licenses/by-nc-nd/4.0/
eu_rights_str_mv openAccess
dc.publisher.none.fl_str_mv Nature Research
publisher.none.fl_str_mv Nature Research
dc.source.none.fl_str_mv reponame:BULERIA. Repositorio Institucional de la Universidad de León
instname:Universidad de León
instname_str Universidad de León
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