Unraveling the key to the resistance of canids to prion diseases

One of the characteristics of prions is their ability to infect some species but not others and prion resistant species have been of special interest because of their potential in deciphering the determinants for susceptibility. Previously, we developed different in vitro and in vivo models to asses...

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Autores: Fernández-Borges, Natalia, Parra, Beatriz|||0000-0002-5371-7449, Vidal Barba, Enric|||0000-0002-4965-3286, Eraña, Hasier|||0000-0001-8776-4211, Sánchez-Martín, Manuel A.|||0000-0001-8370-1336, de Castro, Jorge|||0000-0002-2380-8956, Elezgarai, Saioa R.|||0000-0001-7054-9689, Pumarola i Batlle, Martí|||0000-0002-0935-7941, Mayoral, Tomás|||0000-0003-2644-1966, Castilla, Joaquín|||0000-0002-2216-1361
Tipo de recurso: artículo
Fecha de publicación:2017
País:España
Institución:Universitat Autònoma de Barcelona
Repositorio:Dipòsit Digital de Documents de la UAB
Idioma:inglés
OAI Identifier:oai:ddd.uab.cat:254029
Acceso en línea:https://ddd.uab.cat/record/254029
https://dx.doi.org/urn:doi:10.1371/journal.ppat.1006716
Access Level:acceso abierto
Palabra clave:Malalties priòniques en els animals
Gossos
Prion diseases
Prion
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spelling Unraveling the key to the resistance of canids to prion diseasesFernández-Borges, NataliaParra, Beatriz|||0000-0002-5371-7449Vidal Barba, Enric|||0000-0002-4965-3286Eraña, Hasier|||0000-0001-8776-4211Sánchez-Martín, Manuel A.|||0000-0001-8370-1336de Castro, Jorge|||0000-0002-2380-8956Elezgarai, Saioa R.|||0000-0001-7054-9689Pumarola i Batlle, Martí|||0000-0002-0935-7941Mayoral, Tomás|||0000-0003-2644-1966Castilla, Joaquín|||0000-0002-2216-1361Malalties priòniques en els animalsGossosPrion diseasesPrionOne of the characteristics of prions is their ability to infect some species but not others and prion resistant species have been of special interest because of their potential in deciphering the determinants for susceptibility. Previously, we developed different in vitro and in vivo models to assess the susceptibility of species that were erroneously considered resistant to prion infection, such as members of the Leporidae and Equidae families. Here we undertake in vitro and in vivo approaches to understand the unresolved low prion susceptibility of canids. Studies based on the amino acid sequence of the canine prion protein (PrP), together with a structural analysis in silico, identified unique key amino acids whose characteristics could orchestrate its high resistance to prion disease. Cell- and brain-based PMCA studies were performed highlighting the relevance of the D163 amino acid in proneness to protein misfolding. This was also investigated by the generation of a novel transgenic mouse model carrying this substitution and these mice showed complete resistance to disease despite intracerebral challenge with three different mouse prion strains (RML, 22L and 301C) known to cause disease in wild-type mice. These findings suggest that dog D163 amino acid is primarily, if not totally, responsible for the prion resistance of canids. Detection of individuals or whole species resistant to any infectious disease is vital to understand the determinants of susceptibility and to develop appropriate therapeutic and preventative strategies. Canids have long been considered resistant to prion infection given the absence of clinical disease despite exposure to the causal agent. Through extensive analysis of the canine prion protein we have detected a key amino acid that might be responsible for their universal resistance to prion disease. Using in vitro and in vivo models we demonstrated that the presence of this residue confers resistance to prion infection when introduced to susceptible animals, opening the way to develop a new therapeutic approach against these, at present, untreatable disorders. 22017-01-0120172017-01-01Articlehttp://purl.org/coar/resource_type/c_6501VoRhttp://purl.org/coar/version/c_970fb48d4fbd8a85info:eu-repo/semantics/articleapplication/pdfhttps://ddd.uab.cat/record/254029https://dx.doi.org/urn:doi:10.1371/journal.ppat.1006716reponame:Dipòsit Digital de Documents de la UABinstname:Universitat Autònoma de BarcelonaInglésengMinisterio de Economía y Competitividad https://doi.org/10.13039/501100003329 AGL2015-65046-C2-1-RMinisterio de Economía y Competitividad https://doi.org/10.13039/501100003329 PCIN-2013-065open accesshttp://purl.org/coar/access_right/c_abf2Aquest document està subjecte a una llicència d'ús Creative Commons. Es permet la reproducció total o parcial, la distribució, la comunicació pública de l'obra i la creació d'obres derivades, fins i tot amb finalitats comercials, sempre i quan es reconegui l'autoria de l'obra original.https://creativecommons.org/licenses/by/4.0/info:eu-repo/semantics/openAccessoai:ddd.uab.cat:2540292026-06-06T12:50:31Z
dc.title.none.fl_str_mv Unraveling the key to the resistance of canids to prion diseases
title Unraveling the key to the resistance of canids to prion diseases
spellingShingle Unraveling the key to the resistance of canids to prion diseases
Fernández-Borges, Natalia
Malalties priòniques en els animals
Gossos
Prion diseases
Prion
title_short Unraveling the key to the resistance of canids to prion diseases
title_full Unraveling the key to the resistance of canids to prion diseases
title_fullStr Unraveling the key to the resistance of canids to prion diseases
title_full_unstemmed Unraveling the key to the resistance of canids to prion diseases
title_sort Unraveling the key to the resistance of canids to prion diseases
dc.creator.none.fl_str_mv Fernández-Borges, Natalia
Parra, Beatriz|||0000-0002-5371-7449
Vidal Barba, Enric|||0000-0002-4965-3286
Eraña, Hasier|||0000-0001-8776-4211
Sánchez-Martín, Manuel A.|||0000-0001-8370-1336
de Castro, Jorge|||0000-0002-2380-8956
Elezgarai, Saioa R.|||0000-0001-7054-9689
Pumarola i Batlle, Martí|||0000-0002-0935-7941
Mayoral, Tomás|||0000-0003-2644-1966
Castilla, Joaquín|||0000-0002-2216-1361
author Fernández-Borges, Natalia
author_facet Fernández-Borges, Natalia
Parra, Beatriz|||0000-0002-5371-7449
Vidal Barba, Enric|||0000-0002-4965-3286
Eraña, Hasier|||0000-0001-8776-4211
Sánchez-Martín, Manuel A.|||0000-0001-8370-1336
de Castro, Jorge|||0000-0002-2380-8956
Elezgarai, Saioa R.|||0000-0001-7054-9689
Pumarola i Batlle, Martí|||0000-0002-0935-7941
Mayoral, Tomás|||0000-0003-2644-1966
Castilla, Joaquín|||0000-0002-2216-1361
author_role author
author2 Parra, Beatriz|||0000-0002-5371-7449
Vidal Barba, Enric|||0000-0002-4965-3286
Eraña, Hasier|||0000-0001-8776-4211
Sánchez-Martín, Manuel A.|||0000-0001-8370-1336
de Castro, Jorge|||0000-0002-2380-8956
Elezgarai, Saioa R.|||0000-0001-7054-9689
Pumarola i Batlle, Martí|||0000-0002-0935-7941
Mayoral, Tomás|||0000-0003-2644-1966
Castilla, Joaquín|||0000-0002-2216-1361
author2_role author
author
author
author
author
author
author
author
author
dc.subject.none.fl_str_mv Malalties priòniques en els animals
Gossos
Prion diseases
Prion
topic Malalties priòniques en els animals
Gossos
Prion diseases
Prion
description One of the characteristics of prions is their ability to infect some species but not others and prion resistant species have been of special interest because of their potential in deciphering the determinants for susceptibility. Previously, we developed different in vitro and in vivo models to assess the susceptibility of species that were erroneously considered resistant to prion infection, such as members of the Leporidae and Equidae families. Here we undertake in vitro and in vivo approaches to understand the unresolved low prion susceptibility of canids. Studies based on the amino acid sequence of the canine prion protein (PrP), together with a structural analysis in silico, identified unique key amino acids whose characteristics could orchestrate its high resistance to prion disease. Cell- and brain-based PMCA studies were performed highlighting the relevance of the D163 amino acid in proneness to protein misfolding. This was also investigated by the generation of a novel transgenic mouse model carrying this substitution and these mice showed complete resistance to disease despite intracerebral challenge with three different mouse prion strains (RML, 22L and 301C) known to cause disease in wild-type mice. These findings suggest that dog D163 amino acid is primarily, if not totally, responsible for the prion resistance of canids. Detection of individuals or whole species resistant to any infectious disease is vital to understand the determinants of susceptibility and to develop appropriate therapeutic and preventative strategies. Canids have long been considered resistant to prion infection given the absence of clinical disease despite exposure to the causal agent. Through extensive analysis of the canine prion protein we have detected a key amino acid that might be responsible for their universal resistance to prion disease. Using in vitro and in vivo models we demonstrated that the presence of this residue confers resistance to prion infection when introduced to susceptible animals, opening the way to develop a new therapeutic approach against these, at present, untreatable disorders.
publishDate 2017
dc.date.none.fl_str_mv 2
2017-01-01
2017
2017-01-01
dc.type.none.fl_str_mv Article
http://purl.org/coar/resource_type/c_6501
VoR
http://purl.org/coar/version/c_970fb48d4fbd8a85
dc.type.openaire.fl_str_mv info:eu-repo/semantics/article
format article
dc.identifier.none.fl_str_mv https://ddd.uab.cat/record/254029
https://dx.doi.org/urn:doi:10.1371/journal.ppat.1006716
url https://ddd.uab.cat/record/254029
https://dx.doi.org/urn:doi:10.1371/journal.ppat.1006716
dc.language.none.fl_str_mv Inglés
eng
language_invalid_str_mv Inglés
language eng
dc.relation.none.fl_str_mv Ministerio de Economía y Competitividad https://doi.org/10.13039/501100003329 AGL2015-65046-C2-1-R
Ministerio de Economía y Competitividad https://doi.org/10.13039/501100003329 PCIN-2013-065
dc.rights.none.fl_str_mv open access
http://purl.org/coar/access_right/c_abf2
https://creativecommons.org/licenses/by/4.0/
dc.rights.openaire.fl_str_mv info:eu-repo/semantics/openAccess
rights_invalid_str_mv open access
http://purl.org/coar/access_right/c_abf2
https://creativecommons.org/licenses/by/4.0/
eu_rights_str_mv openAccess
dc.format.none.fl_str_mv application/pdf
dc.source.none.fl_str_mv reponame:Dipòsit Digital de Documents de la UAB
instname:Universitat Autònoma de Barcelona
instname_str Universitat Autònoma de Barcelona
reponame_str Dipòsit Digital de Documents de la UAB
collection Dipòsit Digital de Documents de la UAB
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