Role of organic cation transporter 3 (OCT3) in the response of hepatocellular carcinoma to tyrosine kinase inhibitors

Impaired function of organic cation transporter 1 (OCT1) in hepatocellular carcinoma (HCC) has been associated with unsatisfactory response to sorafenib. However, some patients lacking OCT1 at the plasma membrane (PM) of HCC cells still respond to sorafenib, suggesting that another transporter may c...

Descripción completa

Detalles Bibliográficos
Autores: Herráez Aguilar, Elisa, Al Abdulla, Ruba, Soto Muñiz, Meraris, Briz Sánchez, Oscar, Bettinger, Dominik, Bantel, Heike, Carmen Martínez, Sofía del, Serrano García, María Ángeles, Geier, Andreas, García Marín, José Juan, Rodríguez Macías, Rocío Isabel
Tipo de recurso: artículo
Estado:Versión publicada
Fecha de publicación:2023
País:España
Institución:Universidad de Salamanca (USAL)
Repositorio:GREDOS. Repositorio Institucional de la Universidad de Salamanca
OAI Identifier:oai:gredos.usal.es:10366/155300
Acceso en línea:http://hdl.handle.net/10366/155300
Access Level:acceso abierto
Palabra clave:Hepatocarcinoma
Chemoresistance
Biomarker
Membrane Transport Proteins
Liver Neoplasms
Carcinoma
Humans
3209 Farmacología
neoplasias hepáticas
humanos
carcinoma
proteínas de transporte de membrana
id ES_708a6c46d84c97cf157ce1c2ca24ca71
oai_identifier_str oai:gredos.usal.es:10366/155300
network_acronym_str ES
network_name_str España
repository_id_str
spelling Role of organic cation transporter 3 (OCT3) in the response of hepatocellular carcinoma to tyrosine kinase inhibitorsHerráez Aguilar, ElisaAl Abdulla, RubaSoto Muñiz, MerarisBriz Sánchez, OscarBettinger, DominikBantel, HeikeCarmen Martínez, Sofía delSerrano García, María ÁngelesGeier, AndreasGarcía Marín, José JuanRodríguez Macías, Rocío IsabelHepatocarcinomaChemoresistanceBiomarkerMembrane Transport ProteinsLiver NeoplasmsCarcinomaHumans3209 Farmacologíaneoplasias hepáticashumanoscarcinomaproteínas de transporte de membranaImpaired function of organic cation transporter 1 (OCT1) in hepatocellular carcinoma (HCC) has been associated with unsatisfactory response to sorafenib. However, some patients lacking OCT1 at the plasma membrane (PM) of HCC cells still respond to sorafenib, suggesting that another transporter may contribute to take up this drug. The aim of this study was to investigate whether OCT3 could contribute to the uptake of sorafenib and other tyrosine kinase inhibitors (TKIs) and whether OCT3 determination can predict HCC response to sorafenib. Cells overexpressing OCT3 were used to determine the ability of this carrier to transport sorafenib. Immunostaining of OCT3 was performed in HCC samples obtained in the TRANSFER study. Considering the intensity of staining and the number of OCT3-positive cells, tumors were classified as having absent, weak, moderate, or strong OCT3 expression and were also categorized according to the presence or absence of PM staining. Functional in vitro studies revealed that OCT3 is also able to mediate sorafenib uptake. Other TKIs, such as regorafenib, lenvatinib, and cabozantinib can also interact with this transporter. In silico studies suggested that the expression of OCT3 is better preserved in HCC than that of OCT1. In HCC samples, OCT3 was expressed at the PM of cancer cells, and its presence, detected in 26% of tumors, was associated with better outcomes in patients treated with sorafenib. In conclusion, analysis by immunohistochemistry of OCT3 in the PM of tumor cells may help to predict the response of HCC patients to sorafenib and potentially to other TKIs.Fondo de Investigaciones Sanitarias, Instituto de Salud Carlos III Junta de Castilla y Leon202420242023info:eu-repo/semantics/articleinfo:eu-repo/semantics/publishedVersionapplication/pdfhttp://hdl.handle.net/10366/155300reponame:GREDOS. Repositorio Institucional de la Universidad de Salamancainstname:Universidad de Salamanca (USAL)InglésPI19/00819PI20/00189PI22/00526SA074P20GRS 2322/A/21info:eu-repo/semantics/openAccessoai:gredos.usal.es:10366/1553002026-06-07T06:28:51Z
dc.title.none.fl_str_mv Role of organic cation transporter 3 (OCT3) in the response of hepatocellular carcinoma to tyrosine kinase inhibitors
title Role of organic cation transporter 3 (OCT3) in the response of hepatocellular carcinoma to tyrosine kinase inhibitors
spellingShingle Role of organic cation transporter 3 (OCT3) in the response of hepatocellular carcinoma to tyrosine kinase inhibitors
Herráez Aguilar, Elisa
Hepatocarcinoma
Chemoresistance
Biomarker
Membrane Transport Proteins
Liver Neoplasms
Carcinoma
Humans
3209 Farmacología
neoplasias hepáticas
humanos
carcinoma
proteínas de transporte de membrana
title_short Role of organic cation transporter 3 (OCT3) in the response of hepatocellular carcinoma to tyrosine kinase inhibitors
title_full Role of organic cation transporter 3 (OCT3) in the response of hepatocellular carcinoma to tyrosine kinase inhibitors
title_fullStr Role of organic cation transporter 3 (OCT3) in the response of hepatocellular carcinoma to tyrosine kinase inhibitors
title_full_unstemmed Role of organic cation transporter 3 (OCT3) in the response of hepatocellular carcinoma to tyrosine kinase inhibitors
title_sort Role of organic cation transporter 3 (OCT3) in the response of hepatocellular carcinoma to tyrosine kinase inhibitors
dc.creator.none.fl_str_mv Herráez Aguilar, Elisa
Al Abdulla, Ruba
Soto Muñiz, Meraris
Briz Sánchez, Oscar
Bettinger, Dominik
Bantel, Heike
Carmen Martínez, Sofía del
Serrano García, María Ángeles
Geier, Andreas
García Marín, José Juan
Rodríguez Macías, Rocío Isabel
author Herráez Aguilar, Elisa
author_facet Herráez Aguilar, Elisa
Al Abdulla, Ruba
Soto Muñiz, Meraris
Briz Sánchez, Oscar
Bettinger, Dominik
Bantel, Heike
Carmen Martínez, Sofía del
Serrano García, María Ángeles
Geier, Andreas
García Marín, José Juan
Rodríguez Macías, Rocío Isabel
author_role author
author2 Al Abdulla, Ruba
Soto Muñiz, Meraris
Briz Sánchez, Oscar
Bettinger, Dominik
Bantel, Heike
Carmen Martínez, Sofía del
Serrano García, María Ángeles
Geier, Andreas
García Marín, José Juan
Rodríguez Macías, Rocío Isabel
author2_role author
author
author
author
author
author
author
author
author
author
dc.subject.none.fl_str_mv Hepatocarcinoma
Chemoresistance
Biomarker
Membrane Transport Proteins
Liver Neoplasms
Carcinoma
Humans
3209 Farmacología
neoplasias hepáticas
humanos
carcinoma
proteínas de transporte de membrana
topic Hepatocarcinoma
Chemoresistance
Biomarker
Membrane Transport Proteins
Liver Neoplasms
Carcinoma
Humans
3209 Farmacología
neoplasias hepáticas
humanos
carcinoma
proteínas de transporte de membrana
description Impaired function of organic cation transporter 1 (OCT1) in hepatocellular carcinoma (HCC) has been associated with unsatisfactory response to sorafenib. However, some patients lacking OCT1 at the plasma membrane (PM) of HCC cells still respond to sorafenib, suggesting that another transporter may contribute to take up this drug. The aim of this study was to investigate whether OCT3 could contribute to the uptake of sorafenib and other tyrosine kinase inhibitors (TKIs) and whether OCT3 determination can predict HCC response to sorafenib. Cells overexpressing OCT3 were used to determine the ability of this carrier to transport sorafenib. Immunostaining of OCT3 was performed in HCC samples obtained in the TRANSFER study. Considering the intensity of staining and the number of OCT3-positive cells, tumors were classified as having absent, weak, moderate, or strong OCT3 expression and were also categorized according to the presence or absence of PM staining. Functional in vitro studies revealed that OCT3 is also able to mediate sorafenib uptake. Other TKIs, such as regorafenib, lenvatinib, and cabozantinib can also interact with this transporter. In silico studies suggested that the expression of OCT3 is better preserved in HCC than that of OCT1. In HCC samples, OCT3 was expressed at the PM of cancer cells, and its presence, detected in 26% of tumors, was associated with better outcomes in patients treated with sorafenib. In conclusion, analysis by immunohistochemistry of OCT3 in the PM of tumor cells may help to predict the response of HCC patients to sorafenib and potentially to other TKIs.
publishDate 2023
dc.date.none.fl_str_mv 2023
2024
2024
dc.type.none.fl_str_mv info:eu-repo/semantics/article
info:eu-repo/semantics/publishedVersion
format article
status_str publishedVersion
dc.identifier.none.fl_str_mv http://hdl.handle.net/10366/155300
url http://hdl.handle.net/10366/155300
dc.language.none.fl_str_mv Inglés
language_invalid_str_mv Inglés
dc.relation.none.fl_str_mv PI19/00819
PI20/00189
PI22/00526
SA074P20
GRS 2322/A/21
dc.rights.none.fl_str_mv info:eu-repo/semantics/openAccess
eu_rights_str_mv openAccess
dc.format.none.fl_str_mv application/pdf
dc.source.none.fl_str_mv reponame:GREDOS. Repositorio Institucional de la Universidad de Salamanca
instname:Universidad de Salamanca (USAL)
instname_str Universidad de Salamanca (USAL)
reponame_str GREDOS. Repositorio Institucional de la Universidad de Salamanca
collection GREDOS. Repositorio Institucional de la Universidad de Salamanca
repository.name.fl_str_mv
repository.mail.fl_str_mv
_version_ 1869410588037218304
score 15.301603