Prognostic value of X-chromosome inactivation in symptomatic female carriers of dystrophinopathy

Background: Between 8% and 22% of female carriers of DMD mutations exhibit clinical symptoms of variable severity. Development of symptoms in DMD mutation carriers without chromosomal rearrangements has been attributed to skewed X-chromosome inactivation (XCI) favouring predominant expression of the...

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Autores: Juan-Mateu, Jonàs, Rodríguez, María José, Nascimento, Andrés, Jiménez-Mallebrera, Cecilia, González Quereda, Lidia|||0000-0003-0715-6414, Rivas, Eloy, Paradas, Carmen|||0000-0002-6917-2236, Madruga, Marcos, Sánchez-Ayaso, Pedro, Jou, Cristina|||0000-0002-2223-7880, González Mera, Laura, Munell Casadesus, Francina|||0000-0001-8580-2819, Roig Quilis, Manuel|||0000-0001-8415-9972, Rabasa, M., Hernández-Lain, Aurelio, Diaz-Manera, Jordi|||0000-0003-2941-7988, Gallardo, Eduard|||0000-0002-3942-3436, Pascual Calvet, Jordi, Verdura, Edgard|||0000-0003-3856-2060, Colomer, Jaume, Baiget Bastús, Montserrat, Olivé i Plana, Montserrat|||0000-0001-5727-0165, Gallano, Pia|||0000-0001-8104-2197
Tipo de documento: artigo
Data de publicação:2012
País:España
Recursos:Universitat Autònoma de Barcelona
Repositório:Dipòsit Digital de Documents de la UAB
Idioma:inglês
OAI Identifier:oai:ddd.uab.cat:304924
Acesso em linha:https://ddd.uab.cat/record/304924
https://dx.doi.org/urn:doi:10.1186/1750-1172-7-82
Access Level:Acceso aberto
Palavra-chave:Becker muscular dystrophy
DMD
Duchenne muscular dystrophy
Dystrophin
Symptomatic carrier
X-chromosome inactivation
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spelling Prognostic value of X-chromosome inactivation in symptomatic female carriers of dystrophinopathyJuan-Mateu, JonàsRodríguez, María JoséNascimento, AndrésJiménez-Mallebrera, CeciliaGonzález Quereda, Lidia|||0000-0003-0715-6414Rivas, EloyParadas, Carmen|||0000-0002-6917-2236Madruga, MarcosSánchez-Ayaso, PedroJou, Cristina|||0000-0002-2223-7880González Mera, LauraMunell Casadesus, Francina|||0000-0001-8580-2819Roig Quilis, Manuel|||0000-0001-8415-9972Rabasa, M.Hernández-Lain, AurelioDiaz-Manera, Jordi|||0000-0003-2941-7988Gallardo, Eduard|||0000-0002-3942-3436Pascual Calvet, JordiVerdura, Edgard|||0000-0003-3856-2060Colomer, JaumeBaiget Bastús, MontserratOlivé i Plana, Montserrat|||0000-0001-5727-0165Gallano, Pia|||0000-0001-8104-2197Becker muscular dystrophyDMDDuchenne muscular dystrophyDystrophinSymptomatic carrierX-chromosome inactivationBackground: Between 8% and 22% of female carriers of DMD mutations exhibit clinical symptoms of variable severity. Development of symptoms in DMD mutation carriers without chromosomal rearrangements has been attributed to skewed X-chromosome inactivation (XCI) favouring predominant expression of the DMD mutant allele. However the prognostic use of XCI analysis is controversial. We aimed to evaluate the correlation between X-chromosome inactivation and development of clinical symptoms in a series of symptomatic female carriers of dystrophinopathy. Methods. We reviewed the clinical, pathological and genetic features of twenty-four symptomatic carriers covering a wide spectrum of clinical phenotypes. DMD gene analysis was performed using MLPA and whole gene sequencing in blood DNA and muscle cDNA. Blood and muscle DNA was used for X-chromosome inactivation (XCI) analysis thought the AR methylation assay in symptomatic carriers and their female relatives, asymptomatic carriers as well as non-carrier females. Results: Symptomatic carriers exhibited 49.2% more skewed XCI profiles than asymptomatic carriers. The extent of XCI skewing in blood tended to increase in line with the severity of muscle symptoms. Skewed XCI patterns were found in at least one first-degree female relative in 78.6% of symptomatic carrier families. No mutations altering XCI in the XIST gene promoter were found. Conclusions: Skewed XCI is in many cases familial inherited. The extent of XCI skewing is related to phenotype severity. However, the assessment of XCI by means of the AR methylation assay has a poor prognostic value, probably because the methylation status of the AR gene in muscle may not reflect in all cases the methylation status of the DMD gene. © 2012 Juan-Mateu et al.; licensee BioMed Central Ltd.Universitat Autònoma de Barcelona 22012-01-0120122012-01-01Article de revisióhttp://purl.org/coar/resource_type/c_dcae04bcVoRhttp://purl.org/coar/version/c_970fb48d4fbd8a85info:eu-repo/semantics/articleapplication/pdfhttps://ddd.uab.cat/record/304924https://dx.doi.org/urn:doi:10.1186/1750-1172-7-82reponame:Dipòsit Digital de Documents de la UABinstname:Universitat Autònoma de BarcelonaInglésengopen accesshttp://purl.org/coar/access_right/c_abf2Aquest document està subjecte a una llicència d'ús Creative Commons. Es permet la reproducció total o parcial, la distribució, la comunicació pública de l'obra i la creació d'obres derivades, fins i tot amb finalitats comercials, sempre i quan es reconegui l'autoria de l'obra original.https://creativecommons.org/licenses/by/4.0/info:eu-repo/semantics/openAccessoai:ddd.uab.cat:3049242026-06-06T12:50:31Z
dc.title.none.fl_str_mv Prognostic value of X-chromosome inactivation in symptomatic female carriers of dystrophinopathy
title Prognostic value of X-chromosome inactivation in symptomatic female carriers of dystrophinopathy
spellingShingle Prognostic value of X-chromosome inactivation in symptomatic female carriers of dystrophinopathy
Juan-Mateu, Jonàs
Becker muscular dystrophy
DMD
Duchenne muscular dystrophy
Dystrophin
Symptomatic carrier
X-chromosome inactivation
title_short Prognostic value of X-chromosome inactivation in symptomatic female carriers of dystrophinopathy
title_full Prognostic value of X-chromosome inactivation in symptomatic female carriers of dystrophinopathy
title_fullStr Prognostic value of X-chromosome inactivation in symptomatic female carriers of dystrophinopathy
title_full_unstemmed Prognostic value of X-chromosome inactivation in symptomatic female carriers of dystrophinopathy
title_sort Prognostic value of X-chromosome inactivation in symptomatic female carriers of dystrophinopathy
dc.creator.none.fl_str_mv Juan-Mateu, Jonàs
Rodríguez, María José
Nascimento, Andrés
Jiménez-Mallebrera, Cecilia
González Quereda, Lidia|||0000-0003-0715-6414
Rivas, Eloy
Paradas, Carmen|||0000-0002-6917-2236
Madruga, Marcos
Sánchez-Ayaso, Pedro
Jou, Cristina|||0000-0002-2223-7880
González Mera, Laura
Munell Casadesus, Francina|||0000-0001-8580-2819
Roig Quilis, Manuel|||0000-0001-8415-9972
Rabasa, M.
Hernández-Lain, Aurelio
Diaz-Manera, Jordi|||0000-0003-2941-7988
Gallardo, Eduard|||0000-0002-3942-3436
Pascual Calvet, Jordi
Verdura, Edgard|||0000-0003-3856-2060
Colomer, Jaume
Baiget Bastús, Montserrat
Olivé i Plana, Montserrat|||0000-0001-5727-0165
Gallano, Pia|||0000-0001-8104-2197
author Juan-Mateu, Jonàs
author_facet Juan-Mateu, Jonàs
Rodríguez, María José
Nascimento, Andrés
Jiménez-Mallebrera, Cecilia
González Quereda, Lidia|||0000-0003-0715-6414
Rivas, Eloy
Paradas, Carmen|||0000-0002-6917-2236
Madruga, Marcos
Sánchez-Ayaso, Pedro
Jou, Cristina|||0000-0002-2223-7880
González Mera, Laura
Munell Casadesus, Francina|||0000-0001-8580-2819
Roig Quilis, Manuel|||0000-0001-8415-9972
Rabasa, M.
Hernández-Lain, Aurelio
Diaz-Manera, Jordi|||0000-0003-2941-7988
Gallardo, Eduard|||0000-0002-3942-3436
Pascual Calvet, Jordi
Verdura, Edgard|||0000-0003-3856-2060
Colomer, Jaume
Baiget Bastús, Montserrat
Olivé i Plana, Montserrat|||0000-0001-5727-0165
Gallano, Pia|||0000-0001-8104-2197
author_role author
author2 Rodríguez, María José
Nascimento, Andrés
Jiménez-Mallebrera, Cecilia
González Quereda, Lidia|||0000-0003-0715-6414
Rivas, Eloy
Paradas, Carmen|||0000-0002-6917-2236
Madruga, Marcos
Sánchez-Ayaso, Pedro
Jou, Cristina|||0000-0002-2223-7880
González Mera, Laura
Munell Casadesus, Francina|||0000-0001-8580-2819
Roig Quilis, Manuel|||0000-0001-8415-9972
Rabasa, M.
Hernández-Lain, Aurelio
Diaz-Manera, Jordi|||0000-0003-2941-7988
Gallardo, Eduard|||0000-0002-3942-3436
Pascual Calvet, Jordi
Verdura, Edgard|||0000-0003-3856-2060
Colomer, Jaume
Baiget Bastús, Montserrat
Olivé i Plana, Montserrat|||0000-0001-5727-0165
Gallano, Pia|||0000-0001-8104-2197
author2_role author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
author
dc.contributor.none.fl_str_mv Universitat Autònoma de Barcelona
dc.subject.none.fl_str_mv Becker muscular dystrophy
DMD
Duchenne muscular dystrophy
Dystrophin
Symptomatic carrier
X-chromosome inactivation
topic Becker muscular dystrophy
DMD
Duchenne muscular dystrophy
Dystrophin
Symptomatic carrier
X-chromosome inactivation
description Background: Between 8% and 22% of female carriers of DMD mutations exhibit clinical symptoms of variable severity. Development of symptoms in DMD mutation carriers without chromosomal rearrangements has been attributed to skewed X-chromosome inactivation (XCI) favouring predominant expression of the DMD mutant allele. However the prognostic use of XCI analysis is controversial. We aimed to evaluate the correlation between X-chromosome inactivation and development of clinical symptoms in a series of symptomatic female carriers of dystrophinopathy. Methods. We reviewed the clinical, pathological and genetic features of twenty-four symptomatic carriers covering a wide spectrum of clinical phenotypes. DMD gene analysis was performed using MLPA and whole gene sequencing in blood DNA and muscle cDNA. Blood and muscle DNA was used for X-chromosome inactivation (XCI) analysis thought the AR methylation assay in symptomatic carriers and their female relatives, asymptomatic carriers as well as non-carrier females. Results: Symptomatic carriers exhibited 49.2% more skewed XCI profiles than asymptomatic carriers. The extent of XCI skewing in blood tended to increase in line with the severity of muscle symptoms. Skewed XCI patterns were found in at least one first-degree female relative in 78.6% of symptomatic carrier families. No mutations altering XCI in the XIST gene promoter were found. Conclusions: Skewed XCI is in many cases familial inherited. The extent of XCI skewing is related to phenotype severity. However, the assessment of XCI by means of the AR methylation assay has a poor prognostic value, probably because the methylation status of the AR gene in muscle may not reflect in all cases the methylation status of the DMD gene. © 2012 Juan-Mateu et al.; licensee BioMed Central Ltd.
publishDate 2012
dc.date.none.fl_str_mv 2
2012-01-01
2012
2012-01-01
dc.type.none.fl_str_mv Article de revisió
http://purl.org/coar/resource_type/c_dcae04bc
VoR
http://purl.org/coar/version/c_970fb48d4fbd8a85
dc.type.openaire.fl_str_mv info:eu-repo/semantics/article
format article
dc.identifier.none.fl_str_mv https://ddd.uab.cat/record/304924
https://dx.doi.org/urn:doi:10.1186/1750-1172-7-82
url https://ddd.uab.cat/record/304924
https://dx.doi.org/urn:doi:10.1186/1750-1172-7-82
dc.language.none.fl_str_mv Inglés
eng
language_invalid_str_mv Inglés
language eng
dc.rights.none.fl_str_mv open access
http://purl.org/coar/access_right/c_abf2
https://creativecommons.org/licenses/by/4.0/
dc.rights.openaire.fl_str_mv info:eu-repo/semantics/openAccess
rights_invalid_str_mv open access
http://purl.org/coar/access_right/c_abf2
https://creativecommons.org/licenses/by/4.0/
eu_rights_str_mv openAccess
dc.format.none.fl_str_mv application/pdf
dc.source.none.fl_str_mv reponame:Dipòsit Digital de Documents de la UAB
instname:Universitat Autònoma de Barcelona
instname_str Universitat Autònoma de Barcelona
reponame_str Dipòsit Digital de Documents de la UAB
collection Dipòsit Digital de Documents de la UAB
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