Annexin A6 modulates TBC1D15/Rab7/StARD3 axis to control endosomal cholesterol export in NPC1 cells

Cholesterol accumulation in late endosomes is a prevailing phenotype of Niemann-Pick type C1 (NPC1) mutant cells. Likewise, annexin A6 (AnxA6) overexpression induces a phenotype reminiscent of NPC1 mutant cells. Here, we demonstrate that this cellular cholesterol imbalance is due to AnxA6 promoting...

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Detalles Bibliográficos
Autores: Meneses-Salas E., García-Melero A., Kanerva K., Blanco-Muñoz P., Morales-Paytuvi F., Bonjoch J., Casas J., Egert A., Beevi S.S., Jose J., Llorente-Cortés V., Rye K.-A., Heeren J., Lu A., Pol A., Tebar F., Ikonen E., Grewal T., Enrich C., Rentero C.
Tipo de recurso: artículo
Estado:Versión publicada
Fecha de publicación:2020
País:España
Institución:Institut d’Investigació Biomèdica Sant Pau (IIB Sant Pau)
Repositorio:r-IIB SANT PAU. Repositorio Institucional de Producción Científica del Instituto de Investigación Biomédica Sant Pau
OAI Identifier:oai:iibsantpau.fundanetsuite.com:p10191
Acceso en línea:https://iibsantpau.fundanetsuite.com/Publicaciones/ProdCientif/PublicacionFrw.aspx?id=10191
https://www.scopus.com/inward/record.uri?eid=2-s2.0-85074662041&doi=10.1007%2fs00018-019-03330-y&partnerID=40&md5=dfad68f2649986defcf6cd87e0310ef6
Access Level:acceso abierto
Palabra clave:calphobindin II
cholesterol
cholesterol acyltransferase
fat droplet
Rab7 protein
carrier protein
guanosine triphosphatase activating protein
membrane protein
Rab protein
small interfering RNA
STARD3 protein, human
TBC1D15 protein, human
animal cell
animal tissue
AnxA6 gene
Article
cell interaction
cellular distribution
controlled study
electron microscopy
endosome
enzyme activation
gene
gene function
gene inactivation
lipid storage
mouse
Niemann-Pick disease cell line
nonhuman
protein depletion
Rab7 gene
StARD3 gene
TBC1D15 gene
animal
cell membrane
CHO cell line
Cricetulus
endoplasmic reticulum
genetics
human
metabolism
Niemann Pick disease
pathology
protein domain
protein transport
Animals
Annexin A6
Carrier Proteins
Cell Membrane
CHO Cells
Cholesterol
Endoplasmic Reticulum
Endosomes
GTPase-Activating Proteins
Humans
Membrane Proteins
Niemann-Pick Disease, Type C
Protein D
Descripción
Sumario:Cholesterol accumulation in late endosomes is a prevailing phenotype of Niemann-Pick type C1 (NPC1) mutant cells. Likewise, annexin A6 (AnxA6) overexpression induces a phenotype reminiscent of NPC1 mutant cells. Here, we demonstrate that this cellular cholesterol imbalance is due to AnxA6 promoting Rab7 inactivation via TBC1D15, a Rab7-GAP. In NPC1 mutant cells, AnxA6 depletion and eventual Rab7 activation was associated with peripheral distribution and increased mobility of late endosomes. This was accompanied by an enhanced lipid accumulation in lipid droplets in an acyl-CoA:cholesterol acyltransferase (ACAT)-dependent manner. Moreover, in AnxA6-deficient NPC1 mutant cells, Rab7-mediated rescue of late endosome-cholesterol export required the StAR-related lipid transfer domain-3 (StARD3) protein. Electron microscopy revealed a significant increase of membrane contact sites (MCS) between late endosomes and ER in NPC1 mutant cells lacking AnxA6, suggesting late endosome-cholesterol transfer to the ER via Rab7 and StARD3-dependent MCS formation. This study identifies AnxA6 as a novel gatekeeper that controls cellular distribution of late endosome-cholesterol via regulation of a Rab7-GAP and MCS formation. © 2019, The Author(s).