The release of catecholamines to the cytosol and the exocytosis of secretory vesicles triggered by IP3 in chromaffin cells

The aim of the present study was to investigate the secretory responses elicited by inositol 1,4,5-trisphosphate (IP3) and their regulation by Ca2+ from different sources. Fura-2, carbon fiber amperometry, and plasma membrane capacitance recordings were performed in mouse chromaffin cells to evaluat...

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Autores: Sanz Lázaro, Sara, Jiménez Pompa, Amanda, Hernández Vivanco, Alicia, Carmona Hidalgo, Beatriz, García Magro, Nuria, Pérez Álvarez, Alberto, Caba González, José Carlos, Rueda Ruzafa, Lola, Albillos Martínez, María Almudena
Tipo de recurso: artículo
Fecha de publicación:2025
País:España
Institución:Universidad Autónoma de Madrid
Repositorio:Biblos-e Archivo. Repositorio Institucional de la UAM
Idioma:inglés
OAI Identifier:oai:repositorio.uam.es:10486/731800
Acceso en línea:https://hdl.handle.net/10486/731800
https://dx.doi.org/10.1152/ajpcell.00328.2025
Access Level:acceso abierto
Palabra clave:amperometry
calcium
capacitance
catecholamine
IP3
Medicina
Farmacia
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spelling The release of catecholamines to the cytosol and the exocytosis of secretory vesicles triggered by IP3 in chromaffin cellsSanz Lázaro, SaraJiménez Pompa, AmandaHernández Vivanco, AliciaCarmona Hidalgo, BeatrizGarcía Magro, NuriaPérez Álvarez, AlbertoCaba González, José CarlosRueda Ruzafa, LolaAlbillos Martínez, María AlmudenaamperometrycalciumcapacitancecatecholamineIP3MedicinaFarmaciaThe aim of the present study was to investigate the secretory responses elicited by inositol 1,4,5-trisphosphate (IP3) and their regulation by Ca2+ from different sources. Fura-2, carbon fiber amperometry, and plasma membrane capacitance recordings were performed in mouse chromaffin cells to evaluate cytosolic Ca2+ changes, catecholamine release, and exocytosis, respectively. Amperometric recordings revealed that IP3 triggered the continuous release of catecholamines to the cytosol with a plateau shape, either applied independently or in combination with the V-ATPase blocker bafilomycin A1, without exhibiting additive effects, which suggests that V-ATPase blockade might be a potential mechanism of action. The catecholamine release elicited by IP3 can take place in the absence of cytosolic Ca2+; however, it may be also regulated by it through a bell-shaped mechanism, with the contribution of Ca2+ stored in intracellular organelles. Furthermore, plasma membrane capacitance recordings showed that IP3 could also elicit exocytosis of secretory vesicles with the participation of intracellular organelle Ca2+ stores. This exocytosis could be regulated by vesicular or cytosolic Ca2+, as shown in experiments with bafilomycin A1 or the Ca2+ chelator BAPTA-AM, respectively, and by kaempferol, an activator of the mitochondrial Ca2+ uniporter, suggesting that mitochondria may exert physiologically this Ca2+ regulatory mechanism. Therefore, in the IP3-mediated secretion, Ca2+ from different sources control the different steps of catecholamine release from the secretory vesicle to the cytosol and then finally to the extracellular spaceThis work was supported by grants from the Spanish Ministry of Science and Innovation to A.A. (grant numbers BFU2008-01382/BFI, BFU2011-27690, and BFU2015-69092)ElsevierDepartamento de FarmacologíaFacultad de MedicinaGobierno de EspañaAgencia Estatal de Investigación20252025-12-08research articlehttp://purl.org/coar/resource_type/c_2df8fbb1VoRhttp://purl.org/coar/version/c_970fb48d4fbd8a85info:eu-repo/semantics/articleapplication/pdfhttps://hdl.handle.net/10486/731800https://dx.doi.org/10.1152/ajpcell.00328.202541196165reponame:Biblos-e Archivo. Repositorio Institucional de la UAMinstname:Universidad Autónoma de MadridInglésengopen accesshttp://purl.org/coar/access_right/c_abf2Attribution-NonCommercial-NoDerivatives 4.0 Internationalhttp://creativecommons.org/licenses/by-nc-nd/4.0/info:eu-repo/semantics/openAccessoai:repositorio.uam.es:10486/7318002026-06-23T12:46:27Z
dc.title.none.fl_str_mv The release of catecholamines to the cytosol and the exocytosis of secretory vesicles triggered by IP3 in chromaffin cells
title The release of catecholamines to the cytosol and the exocytosis of secretory vesicles triggered by IP3 in chromaffin cells
spellingShingle The release of catecholamines to the cytosol and the exocytosis of secretory vesicles triggered by IP3 in chromaffin cells
Sanz Lázaro, Sara
amperometry
calcium
capacitance
catecholamine
IP3
Medicina
Farmacia
title_short The release of catecholamines to the cytosol and the exocytosis of secretory vesicles triggered by IP3 in chromaffin cells
title_full The release of catecholamines to the cytosol and the exocytosis of secretory vesicles triggered by IP3 in chromaffin cells
title_fullStr The release of catecholamines to the cytosol and the exocytosis of secretory vesicles triggered by IP3 in chromaffin cells
title_full_unstemmed The release of catecholamines to the cytosol and the exocytosis of secretory vesicles triggered by IP3 in chromaffin cells
title_sort The release of catecholamines to the cytosol and the exocytosis of secretory vesicles triggered by IP3 in chromaffin cells
dc.creator.none.fl_str_mv Sanz Lázaro, Sara
Jiménez Pompa, Amanda
Hernández Vivanco, Alicia
Carmona Hidalgo, Beatriz
García Magro, Nuria
Pérez Álvarez, Alberto
Caba González, José Carlos
Rueda Ruzafa, Lola
Albillos Martínez, María Almudena
author Sanz Lázaro, Sara
author_facet Sanz Lázaro, Sara
Jiménez Pompa, Amanda
Hernández Vivanco, Alicia
Carmona Hidalgo, Beatriz
García Magro, Nuria
Pérez Álvarez, Alberto
Caba González, José Carlos
Rueda Ruzafa, Lola
Albillos Martínez, María Almudena
author_role author
author2 Jiménez Pompa, Amanda
Hernández Vivanco, Alicia
Carmona Hidalgo, Beatriz
García Magro, Nuria
Pérez Álvarez, Alberto
Caba González, José Carlos
Rueda Ruzafa, Lola
Albillos Martínez, María Almudena
author2_role author
author
author
author
author
author
author
author
dc.contributor.none.fl_str_mv Departamento de Farmacología
Facultad de Medicina
Gobierno de España
Agencia Estatal de Investigación
dc.subject.none.fl_str_mv amperometry
calcium
capacitance
catecholamine
IP3
Medicina
Farmacia
topic amperometry
calcium
capacitance
catecholamine
IP3
Medicina
Farmacia
description The aim of the present study was to investigate the secretory responses elicited by inositol 1,4,5-trisphosphate (IP3) and their regulation by Ca2+ from different sources. Fura-2, carbon fiber amperometry, and plasma membrane capacitance recordings were performed in mouse chromaffin cells to evaluate cytosolic Ca2+ changes, catecholamine release, and exocytosis, respectively. Amperometric recordings revealed that IP3 triggered the continuous release of catecholamines to the cytosol with a plateau shape, either applied independently or in combination with the V-ATPase blocker bafilomycin A1, without exhibiting additive effects, which suggests that V-ATPase blockade might be a potential mechanism of action. The catecholamine release elicited by IP3 can take place in the absence of cytosolic Ca2+; however, it may be also regulated by it through a bell-shaped mechanism, with the contribution of Ca2+ stored in intracellular organelles. Furthermore, plasma membrane capacitance recordings showed that IP3 could also elicit exocytosis of secretory vesicles with the participation of intracellular organelle Ca2+ stores. This exocytosis could be regulated by vesicular or cytosolic Ca2+, as shown in experiments with bafilomycin A1 or the Ca2+ chelator BAPTA-AM, respectively, and by kaempferol, an activator of the mitochondrial Ca2+ uniporter, suggesting that mitochondria may exert physiologically this Ca2+ regulatory mechanism. Therefore, in the IP3-mediated secretion, Ca2+ from different sources control the different steps of catecholamine release from the secretory vesicle to the cytosol and then finally to the extracellular space
publishDate 2025
dc.date.none.fl_str_mv 2025
2025-12-08
dc.type.none.fl_str_mv research article
http://purl.org/coar/resource_type/c_2df8fbb1
VoR
http://purl.org/coar/version/c_970fb48d4fbd8a85
dc.type.openaire.fl_str_mv info:eu-repo/semantics/article
format article
dc.identifier.none.fl_str_mv https://hdl.handle.net/10486/731800
https://dx.doi.org/10.1152/ajpcell.00328.2025
41196165
url https://hdl.handle.net/10486/731800
https://dx.doi.org/10.1152/ajpcell.00328.2025
identifier_str_mv 41196165
dc.language.none.fl_str_mv Inglés
eng
language_invalid_str_mv Inglés
language eng
dc.rights.none.fl_str_mv open access
http://purl.org/coar/access_right/c_abf2
Attribution-NonCommercial-NoDerivatives 4.0 International
http://creativecommons.org/licenses/by-nc-nd/4.0/
dc.rights.openaire.fl_str_mv info:eu-repo/semantics/openAccess
rights_invalid_str_mv open access
http://purl.org/coar/access_right/c_abf2
Attribution-NonCommercial-NoDerivatives 4.0 International
http://creativecommons.org/licenses/by-nc-nd/4.0/
eu_rights_str_mv openAccess
dc.format.none.fl_str_mv application/pdf
dc.publisher.none.fl_str_mv Elsevier
publisher.none.fl_str_mv Elsevier
dc.source.none.fl_str_mv reponame:Biblos-e Archivo. Repositorio Institucional de la UAM
instname:Universidad Autónoma de Madrid
instname_str Universidad Autónoma de Madrid
reponame_str Biblos-e Archivo. Repositorio Institucional de la UAM
collection Biblos-e Archivo. Repositorio Institucional de la UAM
repository.name.fl_str_mv
repository.mail.fl_str_mv
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