Multiple drug regimen-refractory rosai–dorfman–destombes disease mimicking relapsing polychondritis successfully treated with cobimetinib

Rosai-Dorfman-Destombes disease (RDD) or sinus histiocytosis with massive lymphadenopathy is a rare non-Langerhans cell histiocytosis of unknown cause. The disease often manifests as bilateral painless cervical lymphadenopathy associated with systemic symptoms such as fever and weight loss. Extranod...

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Detalles Bibliográficos
Autores: López-Aldabe, Kima, Escrihuela-Vidal, Francesc, Tuells, Manel, Llobera-Ris, Clàudia, Bauer Alonso, Andrea, CORTES-ROMERA, MONTSERRAT, Gràcia-Sànchez, Laura, Tormo-Ratera, Marian, Juanola Roura, Xavier, Penin-Mosquera, Rosa Maria, Corbella Virós, Xavier, Solanich, Xavier
Tipo de recurso: artículo
Fecha de publicación:2022
País:España
Institución:Varias* (Consorci de Biblioteques Universitáries de Catalunya, Centre de Serveis Científics i Acadèmics de Catalunya)
Repositorio:Recercat. Dipósit de la Recerca de Catalunya
OAI Identifier:oai:recercat.cat:20.500.12328/3439
Acceso en línea:http://hdl.handle.net/20.500.12328/3439
https://dx.doi.org/10.12890/2022_003076
Access Level:acceso abierto
Palabra clave:Malaltia de Rosai-Dorfman-Destombes
Malaltia de Rosai-Dorfman
Policondritis recurrent
Enfermedad de Rosai-Dorfman-Destombes
Enfermedad de Rosai-Dorfman
Policondritis recurrente
Rosai-Dorfman-Destombes disease
Rosai-Dorfman disease
Recurrent polychondritis
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Descripción
Sumario:Rosai-Dorfman-Destombes disease (RDD) or sinus histiocytosis with massive lymphadenopathy is a rare non-Langerhans cell histiocytosis of unknown cause. The disease often manifests as bilateral painless cervical lymphadenopathy associated with systemic symptoms such as fever and weight loss. Extranodal disease is also frequent and can involve any organ, mostly skin, nasal cavity, bone, and retro-orbital tissue. Swelling of cartilaginous tissues such as ear helix or laryngeal structures may mimic the entity known as relapsing polychondritis. Although spontaneous remission is the most expected evolution, some cases require systemic treatment with prednisone, methotrexate or cytotoxic agents, with variable success rates. In this respect, since somatic variants in genes involved in the mitogen-activated protein kinase (MAPK) pathway have been observed to play a pathogenic role in RDD, the use of therapies targeting these pathogenic variants seems to be a reasonable strategy. Here we present a case of RDD with extensive extranodal involvement that showed a rapid and complete response to the MEK inhibitor cobimetinib.