Modifications in the distribution of met-enkephalin in the cat spinal cord after administration of clonidine. An immunocytochemical study

We have studied the modifications in the distribution of methionine-enkephalin in the cat spinal cord after intravenous or intrathecal administration of clonidine by using an immunocytochemical technique. In animals not treated with the substance, a very high density of immunoreactive fibers was fou...

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Autores: Vázquez Tapioles, José, Coveñas Rodríguez, Rafael, Muñoz Sáez, Miguel, García, J. A., Victoria, A.
Tipo de recurso: artículo
Estado:Versión publicada
Fecha de publicación:1998
País:España
Institución:Universidad de Sevilla (US)
Repositorio:idUS. Depósito de Investigación de la Universidad de Sevilla
OAI Identifier:oai:idus.us.es:11441/58809
Acceso en línea:http://hdl.handle.net/11441/58809
Access Level:acceso abierto
Palabra clave:Met-cnkephalin
Clonidine
Spinal cord
Immunocytochemistry
Cat
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spelling Modifications in the distribution of met-enkephalin in the cat spinal cord after administration of clonidine. An immunocytochemical studyVázquez Tapioles, JoséCoveñas Rodríguez, RafaelMuñoz Sáez, MiguelGarcía, J. A.Victoria, A.Met-cnkephalinClonidineSpinal cordImmunocytochemistryCatWe have studied the modifications in the distribution of methionine-enkephalin in the cat spinal cord after intravenous or intrathecal administration of clonidine by using an immunocytochemical technique. In animals not treated with the substance, a very high density of immunoreactive fibers was found in layers I and 11; a high density in the dorso-lateral funiculus and in the reticular formation; a moderate density in layers 111, IV and V; and a low density in layer VI. However, after intravenous or intrathecal administration of clonidine a decrease in fibers containing met-enkephalin was observed in layers I and I1 (high or moderate density), the dorso-lateral funiculus, and the reticular formation (moderate or low density), and in layers IV and V (low or very low density). In all cases, the decrease in the immunoreactivity was more marked when clonidine was administered intrathecally. Our results suggest that clonidine induces the release of metenkephalin in the spinal cord. They further suggest that the opioid peptide released could be involved in the control of nociceptive transmission by inhibiting the release of neurotransmitters (e.g., substance P). In summary, our study shows that clonidine could be involved in antinociceptive mechanisms in the cat spinal cord.Universidad de Murcia. Departamento de Biología Celular e HistologíaMedicinaBiología Celular1998info:eu-repo/semantics/articleinfo:eu-repo/semantics/publishedVersionapplication/pdfapplication/pdfhttp://hdl.handle.net/11441/58809reponame:idUS. Depósito de Investigación de la Universidad de Sevillainstname:Universidad de Sevilla (US)InglésHistology and Histopathology, 13 (4), 955-959.info:eu-repo/semantics/openAccessoai:idus.us.es:11441/588092026-06-17T12:51:07Z
dc.title.none.fl_str_mv Modifications in the distribution of met-enkephalin in the cat spinal cord after administration of clonidine. An immunocytochemical study
title Modifications in the distribution of met-enkephalin in the cat spinal cord after administration of clonidine. An immunocytochemical study
spellingShingle Modifications in the distribution of met-enkephalin in the cat spinal cord after administration of clonidine. An immunocytochemical study
Vázquez Tapioles, José
Met-cnkephalin
Clonidine
Spinal cord
Immunocytochemistry
Cat
title_short Modifications in the distribution of met-enkephalin in the cat spinal cord after administration of clonidine. An immunocytochemical study
title_full Modifications in the distribution of met-enkephalin in the cat spinal cord after administration of clonidine. An immunocytochemical study
title_fullStr Modifications in the distribution of met-enkephalin in the cat spinal cord after administration of clonidine. An immunocytochemical study
title_full_unstemmed Modifications in the distribution of met-enkephalin in the cat spinal cord after administration of clonidine. An immunocytochemical study
title_sort Modifications in the distribution of met-enkephalin in the cat spinal cord after administration of clonidine. An immunocytochemical study
dc.creator.none.fl_str_mv Vázquez Tapioles, José
Coveñas Rodríguez, Rafael
Muñoz Sáez, Miguel
García, J. A.
Victoria, A.
author Vázquez Tapioles, José
author_facet Vázquez Tapioles, José
Coveñas Rodríguez, Rafael
Muñoz Sáez, Miguel
García, J. A.
Victoria, A.
author_role author
author2 Coveñas Rodríguez, Rafael
Muñoz Sáez, Miguel
García, J. A.
Victoria, A.
author2_role author
author
author
author
dc.contributor.none.fl_str_mv Medicina
Biología Celular
dc.subject.none.fl_str_mv Met-cnkephalin
Clonidine
Spinal cord
Immunocytochemistry
Cat
topic Met-cnkephalin
Clonidine
Spinal cord
Immunocytochemistry
Cat
description We have studied the modifications in the distribution of methionine-enkephalin in the cat spinal cord after intravenous or intrathecal administration of clonidine by using an immunocytochemical technique. In animals not treated with the substance, a very high density of immunoreactive fibers was found in layers I and 11; a high density in the dorso-lateral funiculus and in the reticular formation; a moderate density in layers 111, IV and V; and a low density in layer VI. However, after intravenous or intrathecal administration of clonidine a decrease in fibers containing met-enkephalin was observed in layers I and I1 (high or moderate density), the dorso-lateral funiculus, and the reticular formation (moderate or low density), and in layers IV and V (low or very low density). In all cases, the decrease in the immunoreactivity was more marked when clonidine was administered intrathecally. Our results suggest that clonidine induces the release of metenkephalin in the spinal cord. They further suggest that the opioid peptide released could be involved in the control of nociceptive transmission by inhibiting the release of neurotransmitters (e.g., substance P). In summary, our study shows that clonidine could be involved in antinociceptive mechanisms in the cat spinal cord.
publishDate 1998
dc.date.none.fl_str_mv 1998
dc.type.none.fl_str_mv info:eu-repo/semantics/article
info:eu-repo/semantics/publishedVersion
format article
status_str publishedVersion
dc.identifier.none.fl_str_mv http://hdl.handle.net/11441/58809
url http://hdl.handle.net/11441/58809
dc.language.none.fl_str_mv Inglés
language_invalid_str_mv Inglés
dc.relation.none.fl_str_mv Histology and Histopathology, 13 (4), 955-959.
dc.rights.none.fl_str_mv info:eu-repo/semantics/openAccess
eu_rights_str_mv openAccess
dc.format.none.fl_str_mv application/pdf
application/pdf
dc.publisher.none.fl_str_mv Universidad de Murcia. Departamento de Biología Celular e Histología
publisher.none.fl_str_mv Universidad de Murcia. Departamento de Biología Celular e Histología
dc.source.none.fl_str_mv reponame:idUS. Depósito de Investigación de la Universidad de Sevilla
instname:Universidad de Sevilla (US)
instname_str Universidad de Sevilla (US)
reponame_str idUS. Depósito de Investigación de la Universidad de Sevilla
collection idUS. Depósito de Investigación de la Universidad de Sevilla
repository.name.fl_str_mv
repository.mail.fl_str_mv
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