GTPases of the Rho subfamily are required for Brucella abortus internalization in nonprofessional phagocytes: direct activation of Cdc42

Members of the genus Brucella are intracellular alpha-Proteobacteria responsible for brucellosis, a chronic disease of humans and animals. Little is known about Brucella virulence mechanisms, but the abilities of these bacteria to invade and to survive within cells are decisive factors for causing d...

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Autores: Guzman-Verri, C. (Caterina)|||/items/8608bab4-b31a-4c53-b441-c2a0b6d93554, Chaves-Olarte, E. (Esteban)|||/items/dafc39e3-e04f-4c8b-bf17-70eb2e1d1aaf, Eichel-Streiber, C. (Christoph) von|||/items/f1ba8f3a-1d3b-46da-bdab-4b2dabaeca9c, López-Goñi, I. (Ignacio)|||/items/228678b8-8965-4c7c-aa6f-493277285d88, Thelestam, M. (Monica)|||/items/2b87cb07-b831-401b-bce7-b9dd85000976, Arvidson, S. (Staffan)|||/items/4b8ac5bd-5811-4dbb-9d1e-540e54c5214a, Gorvel, J.P. (Jean Pierre)|||/items/b754f278-d985-4792-9319-e9fc71f1cb11, Moreno, E. (Edgardo)|||/items/11186877-6440-423f-a77a-c63ec45c54b0
Tipo de recurso: artículo
Fecha de publicación:2001
País:España
Institución:Universidad de Navarra
Repositorio:Dadun. Depósito Académico Digital de la Universidad de Navarra
Idioma:inglés
OAI Identifier:oai:dadun.unav.edu:10171/66205
Acceso en línea:https://hdl.handle.net/10171/66205
Access Level:acceso abierto
Palabra clave:Brucellosi
Zoonosis
Brucella
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spelling GTPases of the Rho subfamily are required for Brucella abortus internalization in nonprofessional phagocytes: direct activation of Cdc42Guzman-Verri, C. (Caterina)|||/items/8608bab4-b31a-4c53-b441-c2a0b6d93554Chaves-Olarte, E. (Esteban)|||/items/dafc39e3-e04f-4c8b-bf17-70eb2e1d1aafEichel-Streiber, C. (Christoph) von|||/items/f1ba8f3a-1d3b-46da-bdab-4b2dabaeca9cLópez-Goñi, I. (Ignacio)|||/items/228678b8-8965-4c7c-aa6f-493277285d88Thelestam, M. (Monica)|||/items/2b87cb07-b831-401b-bce7-b9dd85000976Arvidson, S. (Staffan)|||/items/4b8ac5bd-5811-4dbb-9d1e-540e54c5214aGorvel, J.P. (Jean Pierre)|||/items/b754f278-d985-4792-9319-e9fc71f1cb11Moreno, E. (Edgardo)|||/items/11186877-6440-423f-a77a-c63ec45c54b0BrucellosiZoonosisBrucellaMembers of the genus Brucella are intracellular alpha-Proteobacteria responsible for brucellosis, a chronic disease of humans and animals. Little is known about Brucella virulence mechanisms, but the abilities of these bacteria to invade and to survive within cells are decisive factors for causing disease. Transmission electron and fluorescence microscopy of infected nonprofessional phagocytic HeLa cells revealed minor membrane changes accompanied by discrete recruitment of F-actin at the site of Brucella abortus entry. Cell uptake of B. abortus was negatively affected to various degrees by actin, actin-myosin, and microtubule chemical inhibitors. Modulators of MAPKs and protein-tyrosine kinases hampered Brucella cell internalization. Inactivation of Rho small GTPases using clostridial toxins TcdB-10463, TcdB-1470, TcsL-1522, and TcdA significantly reduced the uptake of B. abortus by HeLa cells. In contrast, cytotoxic necrotizing factor from Escherichia coli, known to activate Rho, Rac, and Cdc42 small GTPases, increased the internalization of both virulent and non-virulent B. abortus. Expression of dominant-positive Rho, Rac, and Cdc42 forms in HeLa cells promoted the uptake of B. abortus, whereas expression of dominant-negative forms of these GTPases in HeLa cells hampered Brucella uptake. Cdc42 was activated upon cell contact by virulent B. abortus, but not by a noninvasive isogenic strain, as proven by affinity precipitation of active Rho, Rac, and Cdc42. The polyphasic approach used to discern the molecular events leading to Brucella internalization provides new alternatives for exploring the complexity of the signals required by intracellular pathogens for cell invasion.Dadun. Depósito Académico Digital Universidad de Navarra20232023-05-1020012001-01-0120012001-01-01journal articlehttp://purl.org/coar/resource_type/c_6501info:eu-repo/semantics/articleapplication/pdfhttps://hdl.handle.net/10171/66205reponame:Dadun. Depósito Académico Digital de la Universidad de Navarrainstname:Universidad de NavarraInglésengopen accesshttp://purl.org/coar/access_right/c_abf2info:eu-repo/semantics/openAccessoai:dadun.unav.edu:10171/662052026-06-21T12:47:57Z
dc.title.none.fl_str_mv GTPases of the Rho subfamily are required for Brucella abortus internalization in nonprofessional phagocytes: direct activation of Cdc42
title GTPases of the Rho subfamily are required for Brucella abortus internalization in nonprofessional phagocytes: direct activation of Cdc42
spellingShingle GTPases of the Rho subfamily are required for Brucella abortus internalization in nonprofessional phagocytes: direct activation of Cdc42
Guzman-Verri, C. (Caterina)|||/items/8608bab4-b31a-4c53-b441-c2a0b6d93554
Brucellosi
Zoonosis
Brucella
title_short GTPases of the Rho subfamily are required for Brucella abortus internalization in nonprofessional phagocytes: direct activation of Cdc42
title_full GTPases of the Rho subfamily are required for Brucella abortus internalization in nonprofessional phagocytes: direct activation of Cdc42
title_fullStr GTPases of the Rho subfamily are required for Brucella abortus internalization in nonprofessional phagocytes: direct activation of Cdc42
title_full_unstemmed GTPases of the Rho subfamily are required for Brucella abortus internalization in nonprofessional phagocytes: direct activation of Cdc42
title_sort GTPases of the Rho subfamily are required for Brucella abortus internalization in nonprofessional phagocytes: direct activation of Cdc42
dc.creator.none.fl_str_mv Guzman-Verri, C. (Caterina)|||/items/8608bab4-b31a-4c53-b441-c2a0b6d93554
Chaves-Olarte, E. (Esteban)|||/items/dafc39e3-e04f-4c8b-bf17-70eb2e1d1aaf
Eichel-Streiber, C. (Christoph) von|||/items/f1ba8f3a-1d3b-46da-bdab-4b2dabaeca9c
López-Goñi, I. (Ignacio)|||/items/228678b8-8965-4c7c-aa6f-493277285d88
Thelestam, M. (Monica)|||/items/2b87cb07-b831-401b-bce7-b9dd85000976
Arvidson, S. (Staffan)|||/items/4b8ac5bd-5811-4dbb-9d1e-540e54c5214a
Gorvel, J.P. (Jean Pierre)|||/items/b754f278-d985-4792-9319-e9fc71f1cb11
Moreno, E. (Edgardo)|||/items/11186877-6440-423f-a77a-c63ec45c54b0
author Guzman-Verri, C. (Caterina)|||/items/8608bab4-b31a-4c53-b441-c2a0b6d93554
author_facet Guzman-Verri, C. (Caterina)|||/items/8608bab4-b31a-4c53-b441-c2a0b6d93554
Chaves-Olarte, E. (Esteban)|||/items/dafc39e3-e04f-4c8b-bf17-70eb2e1d1aaf
Eichel-Streiber, C. (Christoph) von|||/items/f1ba8f3a-1d3b-46da-bdab-4b2dabaeca9c
López-Goñi, I. (Ignacio)|||/items/228678b8-8965-4c7c-aa6f-493277285d88
Thelestam, M. (Monica)|||/items/2b87cb07-b831-401b-bce7-b9dd85000976
Arvidson, S. (Staffan)|||/items/4b8ac5bd-5811-4dbb-9d1e-540e54c5214a
Gorvel, J.P. (Jean Pierre)|||/items/b754f278-d985-4792-9319-e9fc71f1cb11
Moreno, E. (Edgardo)|||/items/11186877-6440-423f-a77a-c63ec45c54b0
author_role author
author2 Chaves-Olarte, E. (Esteban)|||/items/dafc39e3-e04f-4c8b-bf17-70eb2e1d1aaf
Eichel-Streiber, C. (Christoph) von|||/items/f1ba8f3a-1d3b-46da-bdab-4b2dabaeca9c
López-Goñi, I. (Ignacio)|||/items/228678b8-8965-4c7c-aa6f-493277285d88
Thelestam, M. (Monica)|||/items/2b87cb07-b831-401b-bce7-b9dd85000976
Arvidson, S. (Staffan)|||/items/4b8ac5bd-5811-4dbb-9d1e-540e54c5214a
Gorvel, J.P. (Jean Pierre)|||/items/b754f278-d985-4792-9319-e9fc71f1cb11
Moreno, E. (Edgardo)|||/items/11186877-6440-423f-a77a-c63ec45c54b0
author2_role author
author
author
author
author
author
author
dc.contributor.none.fl_str_mv Dadun. Depósito Académico Digital Universidad de Navarra
dc.subject.none.fl_str_mv Brucellosi
Zoonosis
Brucella
topic Brucellosi
Zoonosis
Brucella
description Members of the genus Brucella are intracellular alpha-Proteobacteria responsible for brucellosis, a chronic disease of humans and animals. Little is known about Brucella virulence mechanisms, but the abilities of these bacteria to invade and to survive within cells are decisive factors for causing disease. Transmission electron and fluorescence microscopy of infected nonprofessional phagocytic HeLa cells revealed minor membrane changes accompanied by discrete recruitment of F-actin at the site of Brucella abortus entry. Cell uptake of B. abortus was negatively affected to various degrees by actin, actin-myosin, and microtubule chemical inhibitors. Modulators of MAPKs and protein-tyrosine kinases hampered Brucella cell internalization. Inactivation of Rho small GTPases using clostridial toxins TcdB-10463, TcdB-1470, TcsL-1522, and TcdA significantly reduced the uptake of B. abortus by HeLa cells. In contrast, cytotoxic necrotizing factor from Escherichia coli, known to activate Rho, Rac, and Cdc42 small GTPases, increased the internalization of both virulent and non-virulent B. abortus. Expression of dominant-positive Rho, Rac, and Cdc42 forms in HeLa cells promoted the uptake of B. abortus, whereas expression of dominant-negative forms of these GTPases in HeLa cells hampered Brucella uptake. Cdc42 was activated upon cell contact by virulent B. abortus, but not by a noninvasive isogenic strain, as proven by affinity precipitation of active Rho, Rac, and Cdc42. The polyphasic approach used to discern the molecular events leading to Brucella internalization provides new alternatives for exploring the complexity of the signals required by intracellular pathogens for cell invasion.
publishDate 2001
dc.date.none.fl_str_mv 2001
2001-01-01
2001
2001-01-01
2023
2023-05-10
dc.type.none.fl_str_mv journal article
http://purl.org/coar/resource_type/c_6501
dc.type.openaire.fl_str_mv info:eu-repo/semantics/article
format article
dc.identifier.none.fl_str_mv https://hdl.handle.net/10171/66205
url https://hdl.handle.net/10171/66205
dc.language.none.fl_str_mv Inglés
eng
language_invalid_str_mv Inglés
language eng
dc.rights.none.fl_str_mv open access
http://purl.org/coar/access_right/c_abf2
dc.rights.openaire.fl_str_mv info:eu-repo/semantics/openAccess
rights_invalid_str_mv open access
http://purl.org/coar/access_right/c_abf2
eu_rights_str_mv openAccess
dc.format.none.fl_str_mv application/pdf
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